about
Efficient transmission and characterization of Creutzfeldt-Jakob disease strains in bank voles.Health professions and risk of sporadic Creutzfeldt-Jakob disease, 1965 to 2010Cyclooxygenase-2, prostaglandin E2, and microglial activation in prion diseases[Creutzfeldt-Jakob disease and other human forms of transmissible spongiform encephalopathy in Italy: a mortality study carried out from different data sources].Advanced tests for early and accurate diagnosis of Creutzfeldt-Jakob disease.Scrapie infectivity is quickly cleared in tissues of orally-infected farmed fish.Human transmissible spongiform encephalopathies in eleven countries: diagnostic pattern across time, 1993-2002.Genomic and post-genomic analyses of human prion diseasesIdentification of the pathological prion protein allotypes in scrapie-infected heterozygous bank voles (Clethrionomys glareolus) by high-performance liquid chromatography-mass spectrometry.Quantitative profiling of the pathological prion protein allotypes in bank voles by liquid chromatography-mass spectrometry.Proteomic profiling of PrP27-30-enriched preparations extracted from the brain of hamsters with experimental scrapie.Creutzfeldt-Jakob disease mortality in Italy, 1982-1996.Diagnosis of Creutzfeldt-Jakob disease. Routine tonsil biopsy for diagnosis of new variant Creutzfeldt-Jakob disease is not justified.Iatrogenic Creutzfeldt-Jakob disease at the millennium.Ultra-high-pressure inactivation of prion infectivity in processed meat: a practical method to prevent human infection.A test for Creutzfeldt-Jakob disease using nasal brushings.A new variant of Creutzfeldt-Jakob disease in the UK.Ancestral origins and worldwide distribution of the PRNP 200K mutation causing familial Creutzfeldt-Jakob disease.Synthetic scrapie infectivity: interaction between recombinant PrP and scrapie brain-derived RNA.Age at Death of Creutzfeldt-Jakob disease in subsequent family generation carrying the E200K mutation of the prion protein gene.Molecular diagnostics of transmissible spongiform encephalopathies.Cerebrospinal fluid biomarkers in human genetic transmissible spongiform encephalopathiesA genome wide association study links glutamate receptor pathway to sporadic Creutzfeldt-Jakob disease risk.Prion (PrPres) allotypes profiling: a new perspectives from mass spectrometry.Assessing prion infectivity of human urine in sporadic Creutzfeldt-Jakob diseaseIatrogenic Creutzfeldt-Jakob disease, final assessment.Cerebrospinal fluid biomarker supported diagnosis of Creutzfeldt-Jakob disease and rapid dementias: a longitudinal multicentre study over 10 yearsSporadic Creutzfeldt-Jakob disease subtype-specific alterations of the brain proteome: impact on Rab3a recyclingCodon 200 mutation in a new family of Chilean origin with Creutzfeldt-Jakob disease.Methodological aspects of the validation of purification procedures of human/animal-derived products to remove unconventional slow viruses.Revisiting the Heidenhain Variant of Creutzfeldt-Jakob Disease: Evidence for Prion Type Variability Influencing Clinical Course and Laboratory FindingsIdentification of misfolded proteins in body fluids for the diagnosis of prion diseases.Cerebrospinal fluid real-time quaking-induced conversion is a robust and reliable test for sporadic creutzfeldt-jakob disease: An international studyUpdated clinical diagnostic criteria for sporadic Creutzfeldt-Jakob disease.Incidence and spectrum of sporadic Creutzfeldt-Jakob disease variants with mixed phenotype and co-occurrence of PrPSc types: an updated classificationInfluence of timing on CSF tests value for Creutzfeldt-Jakob disease diagnosis.Extended and direct evaluation of RT-QuIC assays for Creutzfeldt-Jakob disease diagnosisPrion-specific and surrogate CSF biomarkers in Creutzfeldt-Jakob disease: diagnostic accuracy in relation to molecular subtypes and analysis of neuropathological correlates of p-tau and Aβ42 levels.Need to improve clinical trials in rare neurodegenerative disorders.Sensitivity to biases of case-control studies on medical procedures, particularly surgery and blood transfusion, and risk of Creutzfeldt-Jakob disease.
P50
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P50
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Maurizio Pocchiari
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