Postischemic inflammatory syndrome: a critical mechanism of progression in diabetic nephropathy.
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Use of Contrast-Enhanced Ultrasound to Study Relationship between Serum Uric Acid and Renal Microvascular Perfusion in Diabetic Kidney DiseaseAbrogation of lectin-like oxidized LDL receptor-1 attenuates acute myocardial ischemia-induced renal dysfunction by modulating systemic and local inflammation.High fat feeding promotes obesity and renal inflammation and protects against post cardiopulmonary bypass acute kidney injury in swineSystolic blood pressure and outcomes in stage 3-4 chronic kidney disease patients: evidence from a Taiwanese cohort.Hyperglycemia, p53, and mitochondrial pathway of apoptosis are involved in the susceptibility of diabetic models to ischemic acute kidney injury.Ischemic kidney injury and mechanisms of tissue repair.Cellular and molecular mechanisms of renal fibrosis.TNF-α mediates increased susceptibility to ischemic AKI in diabetes.Comprehensive genomic profiling in diabetic nephropathy reveals the predominance of proinflammatory pathways.Intravenous renal cell transplantation with SAA1-positive cells prevents the progression of chronic renal failure in rats with ischemic-diabetic nephropathy.Renoprotective approaches and strategies in acute kidney injuryLOX-1: a critical player in the genesis and progression of myocardial ischemia.Renal iron overload in rats with diabetic nephropathy.Glucose Homeostasis and Cardiovascular Alterations in Diabetes.Renal Tubular Cell-Derived Extracellular Vesicles Accelerate the Recovery of Established Renal Ischemia Reperfusion Injury.Thioredoxin-Interacting Protein Mediates NLRP3 Inflammasome Activation Involved in the Susceptibility to Ischemic Acute Kidney Injury in Diabetes.Intravenous cell therapy for acute renal failure with serum amyloid A protein-reprogrammed cells.Diagnostic value of quantitative contrast-enhanced ultrasound (CEUS) for early detection of renal hyperperfusion in diabetic kidney disease.Renal C3 complement component: feed forward to diabetic kidney disease.Treatment of the post-ischaemic inflammatory syndrome of diabetic nephropathy.Immune complexes and complexity: investigating mechanisms of renal disease.Human extracellular microvesicles from renal tubules reverse kidney ischemia-reperfusion injury in ratsRenal Ischemia-Reperfusion Injury in a Diabetic Monkey Model and Therapeutic Testing of Human Bone Marrow-Derived Mesenchymal Stem Cells
P2860
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P2860
Postischemic inflammatory syndrome: a critical mechanism of progression in diabetic nephropathy.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
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2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
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2009年學術文章
@zh-hant
name
Postischemic inflammatory synd ...... ssion in diabetic nephropathy.
@en
Postischemic inflammatory synd ...... ssion in diabetic nephropathy.
@nl
type
label
Postischemic inflammatory synd ...... ssion in diabetic nephropathy.
@en
Postischemic inflammatory synd ...... ssion in diabetic nephropathy.
@nl
prefLabel
Postischemic inflammatory synd ...... ssion in diabetic nephropathy.
@en
Postischemic inflammatory synd ...... ssion in diabetic nephropathy.
@nl
P2860
P1476
Postischemic inflammatory synd ...... ession in diabetic nephropathy
@en
P2093
Jesus H Dominguez
P2860
P304
P356
10.1152/AJPRENAL.00205.2009
P577
2009-08-05T00:00:00Z