NADPH Oxidase-Related Pathophysiology in Experimental Models of Stroke.

about

NADPH Oxidase-Related Pathophysiology in Experimental Models of Stroke.

Item

http://www.wikidata.org/entity/Q45833237

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh

2017年學術文章

@zh-hant

name

NADPH Oxidase-Related Pathophysiology in Experimental Models of Stroke.

@en

NADPH Oxidase-Related Pathophysiology in Experimental Models of Stroke.

@nl

type

Item

label

NADPH Oxidase-Related Pathophysiology in Experimental Models of Stroke.

@en

NADPH Oxidase-Related Pathophysiology in Experimental Models of Stroke.

@nl

prefLabel

NADPH Oxidase-Related Pathophysiology in Experimental Models of Stroke.

@en

NADPH Oxidase-Related Pathophysiology in Experimental Models of Stroke.

@nl

P1476

NADPH Oxidase-Related Pathophysiology in Experimental Models of Stroke.

@en

P2093

Hiroshi Yao

http://www.w3.org/2001/XMLSchema#string

Takanari Kitazono

http://www.w3.org/2001/XMLSchema#string

Toru Nabika

http://www.w3.org/2001/XMLSchema#string

P275

Creative Commons Attribution

P2860

Biochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular system

Pericytes: developmental, physiological, and pathological perspectives, problems, and promises

Post-stroke inhibition of induced NADPH oxidase type 4 prevents oxidative stress and neurodegeneration

The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology

Induction of reproducible brain infarction by photochemically initiated thrombosis

Pivotal Advance: Eosinophilia in the MES rat strain is caused by a loss-of-function mutation in the gene for cytochrome b(-245), alpha polypeptide (Cyba)

Reversible middle cerebral artery occlusion without craniectomy in rats

Nox1 is involved in angiotensin II-mediated hypertension: a study in Nox1-deficient mice

Focal cerebral ischaemia in the rat: 1. Description of technique and early neuropathological consequences following middle cerebral artery occlusion

Reactive oxygen species activate the HIF-1alpha promoter via a functional NFkappaB site

P31

scholarly article

review article

P356

10.3390/IJMS18102123

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Tetsuro Ago

P5008

ScienceSource

P577

2017-10-11T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29019942

http://www.w3.org/2001/XMLSchema#string

P921

cerebrovascular disease

membrane proteins

biomedical investigative technique

Anzim

P932

5666805

http://www.w3.org/2001/XMLSchema#string