Eμ/miR-125b transgenic mice develop lethal B-cell malignancies. - Wikidata - wikimedia - TriplyDB

Eμ/miR-125b transgenic mice develop lethal B-cell malignancies.

Eμ/miR-125b transgenic mice develop lethal B-cell malignancies.

http://www.wikidata.org/entity/Q45853633

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Role of microRNAs in hepatocellular carcinoma: a clinical perspective MicroRNA regulation of the germinal center response Myelodysplastic syndromes are induced by histone methylation–altering ASXL1 mutations MicroRNAs: history, biogenesis, and their evolving role in animal development and disease Alterations of microRNA and microRNA-regulated messenger RNA expression in germinal center B-cell lymphomas determined by integrative sequencing analysis.miR-125b induces cellular senescence in malignant melanoma.Dual mechanisms by which miR-125b represses IRF4 to induce myeloid and B-cell leukemias.miR-125b targets erythropoietin and its receptor and their expression correlates with metastatic potential and ERBB2/HER2 expression.miR-125b can enhance skin tumor initiation and promote malignant progression by repressing differentiation and prolonging cell survival IFN-α regulates Blimp-1 expression via miR-23a and miR-125b in both monocytes-derived DC and pDC.Divergence of transcriptional landscape occurs early in B cell activation.MicroRNAs as therapeutic targets in human cancers Oncomir miR-125b regulates hematopoiesis by targeting the gene Lin28A.Dysfunction of the TP53 tumor suppressor gene in lymphoid malignancies.Complex oncogene dependence in microRNA-125a-induced myeloproliferative neoplasms.MicroRNA-125b transforms myeloid cell lines by repressing multiple mRNA.MAP3K11 is a tumor suppressor targeted by the oncomiR miR-125b in early B cells.The Yin and Yang of microRNAs: leukemia and immunity.Regulation of B-cell development and function by microRNAs.Attenuation of dexamethasone-induced cell death in multiple myeloma is mediated by miR-125b expression Understanding of leukemic stem cells and their clinical implications.miR-99a/100~125b tricistrons regulate hematopoietic stem and progenitor cell homeostasis by shifting the balance between TGFβ and Wnt signaling.MiR-125 in normal and malignant hematopoiesis.MicroRNAs in the p53 network: micromanagement of tumour suppression.MicroRNAs and lymphomagenesis: a functional review.MicroRNAs in mantle cell lymphoma.Targeted therapies through microRNAs: pulp or fiction?microRNAs as potential regulators of myeloid-derived suppressor cell expansion.Good guy or bad guy: the opposing roles of microRNA 125b in cancer The role of miRNAs in cancer: from pathogenesis to therapeutic implications.MicroRNA regulation of viral immunity, latency, and carcinogenesis of selected tumor viruses and HIV.Involvement of miR-125a in resistance to daunorubicin by inhibiting apoptosis in leukemia cell lines.Noncoding RNAs Regulating p53 and c-Myc Signaling.miR-125b promotes MLL-AF9-driven murine acute myeloid leukemia involving a VEGFA-mediated non-cell-intrinsic mechanism.Identification of the novel 3' UTR sequences of human IL-21 mRNA as potential targets of miRNAs.miR-125b predicts childhood acute lymphoblastic leukaemia poor response to BFM chemotherapy treatment.Expression analysis of microRNA-125 in patients with polycythemia vera and essential thrombocythemia and correlation with JAK2 allele burden and laboratory findings.Oxidative stress-induced p53 activity is enhanced by a redox-sensitive TP53INP1 SUMOylation.The down-regulation of miR-125b in chronic lymphocytic leukemias leads to metabolic adaptation of cells to a transformed state.MicroRNA-125b-1 accelerates a C-terminal mutant of C/EBPα (C/EBPα-C(m))-induced myeloid leukemia.

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Eμ/miR-125b transgenic mice develop lethal B-cell malignancies.

http://www.wikidata.org/entity/Q45853633

description

2011 nî lūn-bûn

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

@yue

2011年學術文章

@zh-hant

name

Eμ/miR-125b transgenic mice develop lethal B-cell malignancies.

@en

Eμ/miR-125b transgenic mice develop lethal B-cell malignancies.

@nl

type

label

Eμ/miR-125b transgenic mice develop lethal B-cell malignancies.

@en

Eμ/miR-125b transgenic mice develop lethal B-cell malignancies.

@nl

prefLabel

Eμ/miR-125b transgenic mice develop lethal B-cell malignancies.

@en

Eμ/miR-125b transgenic mice develop lethal B-cell malignancies.

@nl

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Eμ/miR-125b transgenic mice develop lethal B-cell malignancies

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C Haferlach

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H Aburatani

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H Nakakuma

http://www.w3.org/2001/XMLSchema#string

J Kitaura

http://www.w3.org/2001/XMLSchema#string

J Watanuki

http://www.w3.org/2001/XMLSchema#string

K Hatakeyama

http://www.w3.org/2001/XMLSchema#string

K Nishimura

http://www.w3.org/2001/XMLSchema#string

M Shimanuki

http://www.w3.org/2001/XMLSchema#string

M Takahashi

http://www.w3.org/2001/XMLSchema#string

M Taniwaki

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P2860

MicroRNAs enriched in hematopoietic stem cells differentially regulate long-term hematopoietic output

Tumor protein 53-induced nuclear protein 1 expression is repressed by miR-155, and its restoration inhibits pancreatic tumor development

The functions of animal microRNAs

MicroRNAs: genomics, biogenesis, mechanism, and function

Molecular and functional characterization of the stress-induced protein (SIP) gene and its two transcripts generated by alternative splicing. SIP induced by stress and promotes cell death

p53DINP1, a p53-inducible gene, regulates p53-dependent apoptosis

Pre-B cell proliferation and lymphoblastic leukemia/high-grade lymphoma in E(mu)-miR155 transgenic mice

Functional analysis of activating receptor LMIR4 as a counterpart of inhibitory receptor LMIR3.

Possible involvement of RasGRP4 in leukemogenesis.

Hsa-mir-125b-2 is highly expressed in childhood ETV6/RUNX1 (TEL/AML1) leukemias and confers survival advantage to growth inhibitory signals independent of p53

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