Novel gene therapy for rheumatoid arthritis by FADD gene transfer: induction of apoptosis of rheumatoid synoviocytes but not chondrocytes.
about
p53 in rheumatoid arthritis: friend or foe?ARG098, a novel anti-human Fas antibody, suppresses synovial hyperplasia and prevents cartilage destruction in a severe combined immunodeficient-HuRAg mouse model.Inhibition of Fas-associated death domain-containing protein (FADD) protects against myocardial ischemia/reperfusion injury in a heart failure mouse modelGene transfer as a future therapy for rheumatoid arthritis.Harnessing programmed cell death as a therapeutic strategy in rheumatic diseases.Fas (CD95)-related apoptosis and rheumatoid arthritis.The inflammatory role of phagocyte apoptotic pathways in rheumatic diseases.Fas-associated death domain protein and adenosine partnership: fad in RA.Actinomycin D renders cultured synovial fibroblasts susceptible to tumour necrosis factor related apoptosis-inducing ligand (TRAIL)-induced apoptosis.
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Novel gene therapy for rheumatoid arthritis by FADD gene transfer: induction of apoptosis of rheumatoid synoviocytes but not chondrocytes.
description
2000 nî lūn-bûn
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2000年の論文
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2000年学术文章
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name
Novel gene therapy for rheumat ...... viocytes but not chondrocytes.
@en
Novel gene therapy for rheumat ...... viocytes but not chondrocytes.
@nl
type
label
Novel gene therapy for rheumat ...... viocytes but not chondrocytes.
@en
Novel gene therapy for rheumat ...... viocytes but not chondrocytes.
@nl
prefLabel
Novel gene therapy for rheumat ...... viocytes but not chondrocytes.
@en
Novel gene therapy for rheumat ...... viocytes but not chondrocytes.
@nl
P2093
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P1476
Novel gene therapy for rheumat ...... viocytes but not chondrocytes.
@en
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Hasunuma T
Kobayashi T
Nishioka K
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10.1038/SJ.GT.3301127
P577
2000-03-01T00:00:00Z
P6179
1003104512