β Adrenergic Receptor Kinase C-Terminal Peptide Gene-Therapy Improves β2-Adrenergic Receptor-Dependent Neoangiogenesis after Hindlimb Ischemia.
about
Myocardial pathology induced by aldosterone is dependent on non-canonical activities of G protein-coupled receptor kinases.Targeting β3-Adrenergic Receptors in the Heart: Selective Agonism and β-Blockade.GRK2 as negative modulator of NO bioavailability: Implications for cardiovascular disease.Sphingosine Kinases and Sphingosine 1-Phosphate Receptors: Signaling and Actions in the Cardiovascular System.GRK2 as a therapeutic target for heart failure.
P2860
β Adrenergic Receptor Kinase C-Terminal Peptide Gene-Therapy Improves β2-Adrenergic Receptor-Dependent Neoangiogenesis after Hindlimb Ischemia.
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
@zh-my
2015年学术文章
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2015年學術文章
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2015年學術文章
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2015年學術文章
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name
β Adrenergic Receptor Kinase C ...... nesis after Hindlimb Ischemia.
@en
β Adrenergic Receptor Kinase C ...... nesis after Hindlimb Ischemia.
@nl
type
label
β Adrenergic Receptor Kinase C ...... nesis after Hindlimb Ischemia.
@en
β Adrenergic Receptor Kinase C ...... nesis after Hindlimb Ischemia.
@nl
prefLabel
β Adrenergic Receptor Kinase C ...... nesis after Hindlimb Ischemia.
@en
β Adrenergic Receptor Kinase C ...... nesis after Hindlimb Ischemia.
@nl
P2093
P2860
P50
P356
P1476
β Adrenergic Receptor Kinase C ...... enesis after Hindlimb Ischemia
@en
P2093
Dario Leosco
Franco Rengo
Giuseppe Rengo
Giuseppina Gambino
Maria Loreta D'Amico
Nicola Ferrara
Walter J Koch
P2860
P304
P356
10.1124/JPET.115.228411
P407
P577
2015-11-24T00:00:00Z