Mechanisms of joint damage in an experimental model of hemophilic arthritis.
about
Rheumatological manifestations of haematological diseases.Short-term exposure of cartilage to blood results in chondrocyte apoptosis.The effect of secondary prophylaxis versus episodic treatment on the range of motion of target joints in patients with haemophiliaNeoangiogenesis contributes to the development of hemophilic synovitis.Osteoporosis in haemophilic patient, rehabilitative aspects.In vivo models of haemophilia - status on current knowledge of clinical phenotypes and therapeutic interventions.Repeated autologous intraarticular blood injections as an animal model for joint pain in haemophilic arthropathy.Exploration of the pathogenesis of haemophilic joint arthropathy: understanding implications for optimal clinical management.The detrimental effects of iron on the joint: a comparison between haemochromatosis and haemophilia.Guidelines for the management of acute joint bleeds and chronic synovitis in haemophilia: A United Kingdom Haemophilia Centre Doctors' Organisation (UKHCDO) guideline.Heat-shock proteins and their role in chondrocyte protection, an application for autologous transplantation.Longitudinal assessment of bone loss using quantitative ultrasound in a blood-induced arthritis rabbit model.Ammonium-Chloride-Potassium Lysing Buffer Treatment of Fully Differentiated Cells Increases Cell Purity and Resulting Neotissue Functional Properties.Proprioception and isometric muscular strength in haemophilic subjects.Haemophilic arthropathy resembles degenerative rather than inflammatory joint disease.Pilot study on 18 F-FDG PET/CT for detection of inflammatory changes in blood-induced knee arthropathy in a rabbit model.Diagnosis of haemophilic synovitis.Treatment of subchondral cysts in patients with haemophilia.Modified magnetic resonance imaging score compared with orthopaedic and radiological scores for the evaluation of haemophilic arthropathy.The risk of elective orthopaedic surgery for haemophilia patients: Japanese single-centre experience.Hemoglobin stimulates the expression of ADAMTS-5 and ADAMTS-9 by synovial cells: a possible cause of articular cartilage damage after intra-articular hemorrhage.
P2860
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P2860
Mechanisms of joint damage in an experimental model of hemophilic arthritis.
description
1988 nî lūn-bûn
@nan
1988年の論文
@ja
1988年学术文章
@wuu
1988年学术文章
@zh-cn
1988年学术文章
@zh-hans
1988年学术文章
@zh-my
1988年学术文章
@zh-sg
1988年學術文章
@yue
1988年學術文章
@zh
1988年學術文章
@zh-hant
name
Mechanisms of joint damage in an experimental model of hemophilic arthritis.
@en
Mechanisms of joint damage in an experimental model of hemophilic arthritis.
@nl
type
label
Mechanisms of joint damage in an experimental model of hemophilic arthritis.
@en
Mechanisms of joint damage in an experimental model of hemophilic arthritis.
@nl
prefLabel
Mechanisms of joint damage in an experimental model of hemophilic arthritis.
@en
Mechanisms of joint damage in an experimental model of hemophilic arthritis.
@nl
P2093
P2860
P356
P1476
Mechanisms of joint damage in an experimental model of hemophilic arthritis.
@en
P2093
P2860
P304
P356
10.1002/ART.1780310910
P577
1988-09-01T00:00:00Z