Application of zinc-bis-(DL-hydrogensaspartate) does not reduce apoptotic cell death in myocardial infarction in the rat heart.

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Application of zinc-bis-(DL-hydrogensaspartate) does not reduce apoptotic cell death in myocardial infarction in the rat heart.

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2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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name

Application of zinc-bis-

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Application of zinc-bis-(DL-hy ...... l infarction in the rat heart.

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type

Item

label

Application of zinc-bis-

@nl

Application of zinc-bis-(DL-hy ...... l infarction in the rat heart.

@en

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Application of zinc-bis-

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Application of zinc-bis-(DL-hy ...... l infarction in the rat heart.

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P1476

Application of zinc-bis-(DL-hy ...... l infarction in the rat heart.

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P2093

Bernd Klosterhalfen

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Damian Domanski

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Erik Skobel

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Ernst R Schwarz

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Justin E Fuess

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Tanja Tussing

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P2860

Missing effects of zinc in a porcine model of recurrent endotoxemia

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Exogenous zinc protects cardiac cells from reperfusion injury by targeting mitochondrial permeability transition pore through inactivation of glycogen synthase kinase-3beta.

The physiological role of zinc as an antioxidant.

What is the required reperfusion period for assessment of myocardial infarct size using triphenyltetrazolium chloride staining in the rat?

Role of cellular zinc in programmed cell death: temporal relationship between zinc depletion, activation of caspases, and cleavage of Sp family transcription factors.

Apoptosis of endothelial cells precedes myocyte cell apoptosis in ischemia/reperfusion injury.

Cardioprotection by Carvedilol: antiapoptosis is independent of beta-adrenoceptor blockage in the rat heart.

Serum zinc level and coronary heart disease events in patients with type 2 diabetes.

Serum zinc and copper levels in ischemic cardiomyopathy.

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215-221

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scholarly article

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10.1177/1074248409340159

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P433

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P478

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P577

2009-07-15T00:00:00Z

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P698

19605571

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