Role of oxidative stress in ultrafine particle-induced exacerbation of allergic lung inflammation.
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Update in asthma 2009Role of oxidants in interstitial lung diseases: pneumoconioses, constrictive bronchiolitis, and chronic tropical pulmonary eosinophiliaVolatile organic compounds enhance allergic airway inflammation in an experimental mouse modelNanomaterials vs Ambient Ultrafine Particles: an Opportunity to Exchange Toxicology KnowledgeLABORATORY EVALUATION OF A MICROFLUIDIC ELECTROCHEMICAL SENSOR FOR AEROSOL OXIDATIVE LOADCarbon black and titanium dioxide nanoparticles induce distinct molecular mechanisms of toxicityA work group report on ultrafine particles (American Academy of Allergy, Asthma & Immunology): Why ambient ultrafine and engineered nanoparticles should receive special attention for possible adverse health outcomes in human subjectsAcute respiratory inflammation in children and black carbon in ambient air before and during the 2008 Beijing OlympicsSurface modifications of silica nanoparticles are crucial for their inert versus proinflammatory and immunomodulatory properties.Developmental exposure to concentrated ambient ultrafine particulate matter air pollution in mice results in persistent and sex-dependent behavioral neurotoxicity and glial activation.Effects of ultrafine particles-induced oxidative stress on Clara cells in allergic lung inflammation.Exposure to moderate air pollution during late pregnancy and cord blood cytokine secretion in healthy neonates.Domestic airborne black carbon levels and 8-isoprostane in exhaled breath condensate among children in New York CityEffects of ultrafine particles on the allergic inflammation in the lung of asthmatics: results of a double-blinded randomized cross-over clinical pilot study.Preventing carbon nanoparticle-induced lung inflammation reduces antigen-specific sensitization and subsequent allergic reactions in a mouse model.Exposure to Deepwater Horizon Crude Oil Burnoff Particulate Matter Induces Pulmonary Inflammation and Alters Adaptive Immune ResponseUltrafine particles affect the balance of endogenous pro- and anti-inflammatory lipid mediators in the lung: in-vitro and in-vivo studies.Developmental exposure to concentrated ambient particles and preference for immediate reward in miceExposure to urban PM1 in rats: development of bronchial inflammation and airway hyperresponsivenessImpact of experimental type 1 diabetes mellitus on systemic and coagulation vulnerability in mice acutely exposed to diesel exhaust particles.Nanoparticle exposure reactivates latent herpesvirus and restores a signature of acute infection.The compatible solute ectoine reduces the exacerbating effect of environmental model particles on the immune response of the airways.Pulmonary microRNA profiles identify involvement of Creb1 and Sec14l3 in bronchial epithelial changes in allergic asthma.Regulation of the arachidonic acid mobilization in macrophages by combustion-derived particles.Monitoring fine and ultrafine particles in the atmosphere of a Southeast Chinese city.Lung-derived mediators induce cytokine production in downstream organs via an NF-κB-dependent mechanism.Children exposure to indoor ultrafine particles in urban and rural school environments.Differential Effects of Surface-Functionalized Zirconium Oxide Nanoparticles on Alveolar Macrophages, Rat Lung, and a Mouse Allergy Model.Exposure to mycotoxins increases the allergic immune response in a murine asthma model.Pro-Inflammatory versus Immunomodulatory Effects of Silver Nanoparticles in the Lung: The Critical Role of Dose, Size and Surface Modification.Mechanistic insight into the impact of nanomaterials on asthma and allergic airway disease.Ultrafine particles generated from coloring with scented markers in the presence of ozone.Pulmonary toxicity in mice following exposure to cerium chloride.Specific Surface Modifications of Silica Nanoparticles Diminish Inflammasome Activation and In Vivo Expression of Selected Inflammatory Genes.Evaluation on exposures to particulate matter at a junior secondary school: a comprehensive study on health risks and effective inflammatory responses in Northwestern China.Ultrafine Particulate Matter Increases Cardiac Ischemia/Reperfusion Injury via Mitochondrial Permeability Transition Pore.Pollen-derived nonallergenic substances enhance Th2-induced IgE production in B cells.Early-life exposure to three size-fractionated ultrafine and fine atmospheric particulates in Beijing exacerbates asthma development in mature mice.Jet exhaust particles alter human dendritic cell maturation.Low Dose Carbon Black Nanoparticle Exposure Does Not Aggravate Allergic Airway Inflammation in Mice Irrespective of the Presence of Surface Polycyclic Aromatic Hydrocarbons.
P2860
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P2860
Role of oxidative stress in ultrafine particle-induced exacerbation of allergic lung inflammation.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
Role of oxidative stress in ul ...... of allergic lung inflammation.
@en
Role of oxidative stress in ul ...... of allergic lung inflammation.
@nl
type
label
Role of oxidative stress in ul ...... of allergic lung inflammation.
@en
Role of oxidative stress in ul ...... of allergic lung inflammation.
@nl
prefLabel
Role of oxidative stress in ul ...... of allergic lung inflammation.
@en
Role of oxidative stress in ul ...... of allergic lung inflammation.
@nl
P2093
P2860
P1476
Role of oxidative stress in ul ...... of allergic lung inflammation.
@en
P2093
Bernhard Kloo
Erwin Karg
Francesca Alessandrini
Heidrun Behrendt
Holger Schulz
Ingrid Beck-Speier
Ingrid Weichenmeier
Martin Mempel
Shinji Takenaka
Thilo Jakob
P2860
P304
P356
10.1164/RCCM.200807-1061OC
P407
P577
2009-03-05T00:00:00Z