Oct1 loss of function induces a coordinate metabolic shift that opposes tumorigenicity.
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Phosphofructokinase 1 glycosylation regulates cell growth and metabolismTargeting Tumor Metabolism for Cancer Treatment: Is Pyruvate Dehydrogenase Kinases (PDKs) a Viable Anticancer Target?Imaging metabolic heterogeneity in cancerMitochondrial complex I inhibition triggers a mitophagy-dependent ROS increase leading to necroptosis and ferroptosis in melanoma cellsHydrogen peroxide stimulates nuclear import of the POU homeodomain protein Oct-1 and its repressive effect on the expression of Cdx-2.Enforcement of developmental lineage specificity by transcription factor Oct1.Dynamic regulation of Oct1 during mitosis by phosphorylation and ubiquitination.Mitochondrial metabolism directs stemness and differentiation in P19 embryonal carcinoma stem cellsOct1 is a switchable, bipotential stabilizer of repressed and inducible transcriptional states.Transcription factor Oct1 is a somatic and cancer stem cell determinant.Autophagy facilitates glycolysis during Ras-mediated oncogenic transformationZNF217, a candidate breast cancer oncogene amplified at 20q13, regulates expression of the ErbB3 receptor tyrosine kinase in breast cancer cellsOCT1 is a determinant of synbindin-related ERK signalling with independent prognostic significance in gastric cancerPeroxisome proliferator-activated receptor-gamma coactivator 1-alpha (PGC1alpha) is a metabolic regulator of intestinal epithelial cell fate.Glucose-based regulation of miR-451/AMPK signaling depends on the OCT1 transcription factor.Negative regulation of human U6 snRNA promoter by p38 kinase through Oct-1.Regulation of APC/C-Cdh1 and its function in neuronal survivalEnergy metabolism and energy-sensing pathways in mammalian embryonic and adult stem cell fate.Metabolic plasticity in stem cell homeostasis and differentiation.Elevated OCT1 participates in colon tumorigenesis and independently predicts poor prognoses of colorectal cancer patients.The Oct1 transcription factor and epithelial malignancies: Old protein learns new tricksMitochondrial respiratory uncoupling promotes keratinocyte differentiation and blocks skin carcinogenesisRegulation of Oct1/Pou2f1 transcription activity by O-GlcNAcylation.Oct transcription factors in development and stem cells: insights and mechanisms.The Role of p53 in Metabolic RegulationAntioxidant and bioenergetic coupling between neurons and astrocytes.Metabolic sensors and their interplay with cell signalling and transcription.Octamer-binding transcription factors: genomics and functions.Metabolic regulation of mesenchymal stem cell in expansion and therapeutic application.Different N-terminal isoforms of Oct-1 control expression of distinct sets of genes and their high levels in Namalwa Burkitt's lymphoma cells affect a wide range of cellular processes.The role of octamer binding transcription factors in glioblastoma multiforme.Electromagnetic Fields and Stem Cell Fate: When Physics Meets Biology.OCT-1 overexpression is associated with poor prognosis in patients with well-differentiated gastric cancer.Death-associated proliferation kinetic in normal and transformed cells.PGC1α promotes tumor growth by inducing gene expression programs supporting lipogenesis.Spatiotemporally Controlled Ablation of Klf5 Results in Dysregulated Epithelial Homeostasis in Adult Mouse Corneas.Response to energy depletion: miR-451/AMPK loop."Nutrient-sensing" and self-renewal: O-GlcNAc in a new role.BRCA1 through Its E3 Ligase Activity Regulates the Transcription Factor Oct1 and Carbohydrate Metabolism.Octamer transcription factor 1 mediates epithelial-mesenchymal transition in colorectal cancer
P2860
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P2860
Oct1 loss of function induces a coordinate metabolic shift that opposes tumorigenicity.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
Oct1 loss of function induces a coordinate metabolic shift that opposes tumorigenicity.
@en
Oct1 loss of function induces a coordinate metabolic shift that opposes tumorigenicity.
@nl
type
label
Oct1 loss of function induces a coordinate metabolic shift that opposes tumorigenicity.
@en
Oct1 loss of function induces a coordinate metabolic shift that opposes tumorigenicity.
@nl
prefLabel
Oct1 loss of function induces a coordinate metabolic shift that opposes tumorigenicity.
@en
Oct1 loss of function induces a coordinate metabolic shift that opposes tumorigenicity.
@nl
P2093
P356
P1433
P1476
Oct1 loss of function induces a coordinate metabolic shift that opposes tumorigenicity
@en
P2093
Arvind Shakya
Dean Tantin
James E Cox
Robert Cooksey
Victoria Wang
P2888
P304
P356
10.1038/NCB1840
P577
2009-02-15T00:00:00Z