Insulin-like growth factor-I is essential for embryonic bone development. - Wikidata - wikimedia - TriplyDB

Insulin-like growth factor-I is essential for embryonic bone development.

Insulin-like growth factor-I is essential for embryonic bone development.

http://www.wikidata.org/entity/Q46133833

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The Regulatory Role of Signaling Crosstalk in Hypertrophy of MSCs and Human Articular Chondrocytes Role and Mechanisms of Actions of Thyroid Hormone on the Skeletal Development IL-6 and IGF-1 Signaling Within and Between Muscle and Bone: How Important is the mTOR Pathway for Bone Metabolism?Cartilage-specific deletion of ephrin-B2 in mice results in early developmental defects and an osteoarthritis-like phenotype during aging in vivo.Role of IGF-I signaling in muscle bone interactions Cooperative regulation of chondrocyte differentiation by CCN2 and CCN3 shown by a comprehensive analysis of the CCN family proteins in cartilage Molecular cloning, bioinformatics analysis and expression of insulin-like growth factor 2 from Tianzhu white yak, Bos grunniens Disruption of insulin-like growth factor-I expression in type IIalphaI collagen-expressing cells reduces bone length and width in mice.The PI3K pathway regulates endochondral bone growth through control of hypertrophic chondrocyte differentiation.Insulin-like growth factor 1 physiology: lessons from mouse models.Conditional deletion of insulin-like growth factor-I in collagen type 1alpha2-expressing cells results in postnatal lethality and a dramatic reduction in bone accretion The role of FGF-2 and BMP-2 in regulation of gene induction, cell proliferation and mineralization Effects of laser acupuncture on longitudinal bone growth in adolescent rats.Bone mineral density of the spine in 11,898 Chinese infants and young children: a cross-sectional study Thyroid hormone-mediated growth and differentiation of growth plate chondrocytes involves IGF-1 modulation of beta-catenin signaling Treatment with N- and C-terminal peptides of parathyroid hormone-related protein partly compensate the skeletal abnormalities in IGF-I deficient mice.IGF-1 and bone: New discoveries from mouse models Insulin exerts direct, IGF-1 independent actions in growth plate chondrocytes.Selective protein depletion impairs bone growth and causes liver fatty infiltration in female rats: prevention by Spirulina alga.Prenatal ethanol exposure increases osteoarthritis susceptibility in female rat offspring by programming a low-functioning IGF-1 signaling pathway.Bone and skeletal muscle: Key players in mechanotransduction and potential overlapping mechanisms.IGF-1R signaling in chondrocytes modulates growth plate development by interacting with the PTHrP/Ihh pathway.The extracellular calcium-sensing receptor (CaSR) is a critical modulator of skeletal development.Regulation of skeletal growth and mineral acquisition by the GH/IGF-1 axis: Lessons from mouse models.Mesenchymal stem cells expressing insulin-like growth factor-I (MSCIGF) promote fracture healing and restore new bone formation in Irs1 knockout mice: analyses of MSCIGF autocrine and paracrine regenerative effects.IGF-1 Regulates Vertebral Bone Aging Through Sex-Specific and Time-Dependent Mechanisms.Liver kinase b1 is required for white adipose tissue growth and differentiation.Insulin-like growth factor-1 receptor in mature osteoblasts is required for periosteal bone formation induced by reloading Systemically delivered insulin-like growth factor-I enhances mesenchymal stem cell-dependent fracture healing.Differential effects of IGF-1 deficiency during the life span on structural and biomechanical properties in the tibia of aged mice.Abnormalities of IGF-I signaling in the pathogenesis of diseases of the bone, brain, and fetoplacental unit in humans.Human umbilical cord mesenchymal stromal cells exhibit immature nucleus pulposus cell phenotype in a laminin-rich pseudo-three-dimensional culture system.aP2-Cre-mediated inactivation of acetyl-CoA carboxylase 1 causes growth retardation and reduced lipid accumulation in adipose tissues.Multiple roles of the SO4(2-)/Cl-/OH- exchanger protein Slc26a2 in chondrocyte functions.Meckel's and condylar cartilages anomalies in achondroplasia result in defective development and growth of the mandible.Anabolic effects of IGF-1 signaling on the skeleton.Ephrin B2/EphB4 mediates the actions of IGF-I signaling in regulating endochondral bone formation.Autocrine and Paracrine Actions of IGF-I Signaling in Skeletal Development.Developmental mechanisms in articular cartilage degradation in osteoarthritis Signaling pathways regulating cartilage growth plate formation and activity.

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Insulin-like growth factor-I is essential for embryonic bone development.

http://www.wikidata.org/entity/Q46133833

description

2006 nî lūn-bûn

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2006年の論文

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年學術文章

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2006年學術文章

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name

Insulin-like growth factor-I is essential for embryonic bone development.

@en

Insulin-like growth factor-I is essential for embryonic bone development.

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type

label

Insulin-like growth factor-I is essential for embryonic bone development.

@en

Insulin-like growth factor-I is essential for embryonic bone development.

@nl

prefLabel

Insulin-like growth factor-I is essential for embryonic bone development.

@en

Insulin-like growth factor-I is essential for embryonic bone development.

@nl

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Bernard P Halloran

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Daniel D Bikle

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Hashem Z Elalieh

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Shigeki Nishida

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Steven B Doty

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Takeshi Sakata

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Wenhan Chang

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Yongmei Wang

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10.1210/EN.2006-0196

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2006-07-20T00:00:00Z

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