Dissociation and protection of the neurovascular unit after thrombolysis and reperfusion in ischemic rat brain. - Wikidata - wikimedia - TriplyDB

Dissociation and protection of the neurovascular unit after thrombolysis and reperfusion in ischemic rat brain.

Dissociation and protection of the neurovascular unit after thrombolysis and reperfusion in ischemic rat brain.

http://www.wikidata.org/entity/Q46164241

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A Review of the Mechanisms of Blood-Brain Barrier Permeability by Tissue-Type Plasminogen Activator Treatment for Cerebral Ischemia Vascular protection to increase the safety of tissue plasminogen activator for stroke The efficacy of edaravone (radicut), a free radical scavenger, for cardiovascular disease Aging and the neurovascular unit Edaravone protects against methylglyoxal-induced barrier damage in human brain endothelial cells Phosphodiesterase-III inhibitor prevents hemorrhagic transformation induced by focal cerebral ischemia in mice treated with tPA Synergy of combined tPA-edaravone therapy in experimental thrombotic stroke Excess salt increases infarct size produced by photothrombotic distal middle cerebral artery occlusion in spontaneously hypertensive rats.Innate inflammatory responses in stroke: mechanisms and potential therapeutic targets.Plasmin-dependent modulation of the blood-brain barrier: a major consideration during tPA-induced thrombolysis?Stem cell therapy for cerebral ischemia: from basic science to clinical applications.Effect of edaravone on favorable outcome in patients with acute cerebral large vessel occlusion: subanalysis of RESCUE-Japan Registry.The contribution of L-arginine to the neurotoxicity of recombinant tissue plasminogen activator following cerebral ischemia: a review of rtPA neurotoxicity IMM-H004 prevents toxicity induced by delayed treatment of tPA in a rat model of focal cerebral ischemia involving PKA-and PI3K-dependent Akt activation.Early and progressive impairment of spinal blood flow-glucose metabolism coupling in motor neuron degeneration of ALS model mice.Frontal white matter hyperintensities, clasmatodendrosis and gliovascular abnormalities in ageing and post-stroke dementia Release of Matrix Metalloproteinases-2 and 9 by S-Nitrosylated Caveolin-1 Contributes to Degradation of Extracellular Matrix in tPA-Treated Hypoxic Endothelial Cells.The Relation of Urinary 8-OHdG, A Marker of Oxidative Stress to DNA, and Clinical Outcomes for Ischemic Stroke.The neurovascular unit and combination treatment strategies for stroke Targeting neutrophils in ischemic stroke: translational insights from experimental studies.Autologous Bone Marrow Mononuclear Cells Exert Broad Effects on Short- and Long-Term Biological and Functional Outcomes in Rodents with Intracerebral Hemorrhage.Does inhibiting Sur1 complement rt-PA in cerebral ischemia?Low dose tPA plus annexin A2 combination attenuates tPA delayed treatment-associated hemorrhage and improves recovery in rat embolic focal stroke Standards and pitfalls of focal ischemia models in spontaneously hypertensive rats: with a systematic review of recent articles Matrix metalloproteinases and blood-brain barrier disruption in acute ischemic stroke.Functional Recovery after Scutellarin Treatment in Transient Cerebral Ischemic Rats: A Pilot Study with (18) F-Fluorodeoxyglucose MicroPET.Alteplase treatment does not increase brain injury after mechanical middle cerebral artery occlusion in the rat.Hemorrhagic transformation after ischemic stroke in animals and humans Cerebral blood flow alteration in neuroprotection following cerebral ischaemia.Clinical predictors and management of hemorrhagic transformation.Perlecan domain V therapy for stroke: a beacon of hope?Edaravone improves functional and structural outcomes in animal models of focal cerebral ischemia: a systematic review.Clinical trials in acute ischemic stroke.Inflammatory responses in brain ischemia.Pericyte protection by edaravone after tissue plasminogen activator treatment in rat cerebral ischemia.Therapeutic Strategies to Attenuate Hemorrhagic Transformation After Tissue Plasminogen Activator Treatment for Acute Ischemic Stroke Vascular protection and restorative therapy in ischemic stroke.Thrombolysis with Low-Dose Tissue Plasminogen Activator 3-4.5 h After Acute Ischemic Stroke in Five Hospital Groups in Japan.Treatment with recombinant tissue plasminogen activator alters the microRNA expression profiles in mouse brain after acute ischemic stroke.Edaravone, a free radical scavenger, protects components of the neurovascular unit against oxidative stress in vitro.

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P2860

Dissociation and protection of the neurovascular unit after thrombolysis and reperfusion in ischemic rat brain.

http://www.wikidata.org/entity/Q46164241

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Dissociation and protection of ...... rfusion in ischemic rat brain.

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Journal of Cerebral Blood Flow & Metabolism

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Dissociation and protection of ...... rfusion in ischemic rat brain.

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Aiji Ohtsuka

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Hanzhe Zhang

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Jingwei Shang

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Kazunori Miyazaki

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Kentaro Deguchi

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Koji Abe

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Tatsushi Kamiya

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Toru Yamashita

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Toshiki Inaba

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Yasuo Katayama

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P2860

NXY-059 for acute ischemic stroke

Tissue plasminogen activator for acute ischemic stroke

Increased formation of reactive oxygen species after permanent and reversible middle cerebral artery occlusion in the rat.

Reactive oxygen radicals in signaling and damage in the ischemic brain.

Matrix metalloproteinases in neuroinflammation.

Cerebral microvessel responses to focal ischemia.

Rapid disruption of an astrocyte interaction with the extracellular matrix mediated by integrin alpha 6 beta 4 during focal cerebral ischemia/reperfusion.

Bradykinin- and thrombin-induced increases in endothelial permeability occur independently of phospholipase C but require protein kinase C activation.

Strong attenuation of ischemic and postischemic brain edema in rats by a novel free radical scavenger.

Iron and iron-handling proteins in the brain after intracerebral hemorrhage.

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715-725

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10.1038/JCBFM.2008.164

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29

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19142198

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