Acute traumatic coagulopathy: initiated by hypoperfusion: modulated through the protein C pathway? - Wikidata - wikimedia - TriplyDB

Acute traumatic coagulopathy: initiated by hypoperfusion: modulated through the protein C pathway?

Acute traumatic coagulopathy: initiated by hypoperfusion: modulated through the protein C pathway?

http://www.wikidata.org/entity/Q46166562

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Management of bleeding and coagulopathy following major trauma: an updated European guideline.CRASH-2 Study of Tranexamic Acid to Treat Bleeding in Trauma Patients: A Controversy Fueled by Science and Social Media Management of traumatic haemorrhage--the US perspective Coagulation complications following trauma Tranexamic acid: from trauma to routine perioperative use TACTIC: Trans-Agency Consortium for Trauma-Induced Coagulopathy The current understanding of trauma-induced coagulopathy (TIC): a focused review on pathophysiology Clinical presentation and blood gas analysis of multiple trauma patients for prediction of standard coagulation parameters at emergency department arrival.[Hemoglobin-oriented and coagulation factor-based algorithm : Effect on transfusion needs and standardized mortality rate in massively transfused trauma patients].[Trauma bay haemoglobin level. Predictor of coagulation disorder in major trauma].Evaluation of clotting factor activities early after severe multiple trauma and their correlation with coagulation tests and clinical data Hydrodynamic Isotonic Fluid Delivery Ameliorates Moderate-to-Severe Ischemia-Reperfusion Injury in Rat Kidneys.Criteria for empiric treatment of hyperfibrinolysis after trauma.The management of abnormal haemostasis in the ICU.Non-human primate model of poly-traumatic hemorrhagic shock recapitulates early platelet dysfunction observed following severe injury in humans.Taking the Blood Bank to the Field: The Design and Rationale of the Prehospital Air Medical Plasma (PAMPer) Trial Early release of high mobility group box nuclear protein 1 after severe trauma in humans: role of injury severity and tissue hypoperfusion.Diverse coagulopathies in a rabbit model with different abdominal injuries.Comparative response of platelet fV and plasma fV to activated protein C and relevance to a model of acute traumatic coagulopathy.Comparison of the predictive performance of the BIG, TRISS, and PS09 score in an adult trauma population derived from multiple international trauma registries.Traumatic brain injury causes platelet adenosine diphosphate and arachidonic acid receptor inhibition independent of hemorrhagic shock in humans and rats.Fibrinolysis greater than 3% is the critical value for initiation of antifibrinolytic therapy.Coagulopathy following major trauma hemorrhage: lytic, lethal and a lack of fibrinogen The acute management of trauma hemorrhage: a systematic review of randomized controlled trials Prehospital use of nonsteroidal anti-inflammatory drugs (NSAIDs) is associated with a reduced incidence of trauma-induced coagulopathy Management of bleeding following major trauma: an updated European guideline.Inflammatory response to trauma: implications for coagulation and resuscitation A principal component analysis of postinjury viscoelastic assays: clotting factor depletion versus fibrinolysis X chromosome-linked IRAK-1 polymorphism is a strong predictor of multiple organ failure and mortality postinjury.Clinical pathology of the shock syndromes.Differentiating disseminated intravascular coagulation (DIC) with the fibrinolytic phenotype from coagulopathy of trauma and acute coagulopathy of trauma-shock (COT/ACOTS).Revisiting early postinjury mortality: are they bleeding because they are dying or dying because they are bleeding?Systemic central venous oxygen saturation is associated with clot strength during traumatic hemorrhagic shock: A preclinical observational model.Trauma-Induced Coagulopathy: An Institution's 35 Year Perspective on Practice and Research.Blast injury research models Acute traumatic coagulopathy among major trauma patients in an urban tertiary hospital in sub Saharan Africa.Implementation of a military-derived damage-control resuscitation strategy in a civilian trauma center decreases acute hypoxia in massively transfused patients A non-lethal traumatic/hemorrhagic insult strongly modulates the compartment-specific PAI-1 response in the subsequent polymicrobial sepsis.Effectiveness of massive transfusion protocols on mortality in trauma: a systematic review and meta-analysis.Advances in the understanding of trauma-induced coagulopathy

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Acute traumatic coagulopathy: initiated by hypoperfusion: modulated through the protein C pathway?

http://www.wikidata.org/entity/Q46166562

description

2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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Acute traumatic coagulopathy: ...... through the protein C pathway?

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Acute traumatic coagulopathy: ...... through the protein C pathway?

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Acute traumatic coagulopathy: ...... through the protein C pathway?

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Annals of Surgery

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Acute traumatic coagulopathy: ...... through the protein C pathway?

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P2093

Jean-François Pittet

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Karim Brohi

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Michael T Ganter

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Mitchell J Cohen

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Robert C Mackersie

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P2860

Acute renal failure - definition, outcome measures, animal models, fluid therapy and information technology needs: the Second International Consensus Conference of the Acute Dialysis Quality Initiative (ADQI) Group

The injury severity score: a method for describing patients with multiple injuries and evaluating emergency care

Macromolecular specificity determinants on thrombin for fibrinogen and thrombomodulin

The American-European Consensus Conference on ARDS. Definitions, mechanisms, relevant outcomes, and clinical trial coordination

Transection of the oesophagus for bleeding oesophageal varices.

Protein C pathway in sepsis.

Treating coagulopathy in trauma patients.

Base deficit stratifies mortality and determines therapy.

The lectin-like domain of thrombomodulin confers protection from neutrophil-mediated tissue damage by suppressing adhesion molecule expression via nuclear factor kappaB and mitogen-activated protein kinase pathways

Initial severity of metabolic acidosis predicts the development of acute lung injury in severely traumatized patients.

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Michael Matthay

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1877079

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