Retromer Endosome Exit Domains Serve Multiple Trafficking Destinations and Regulate Local G Protein Activation by GPCRs.
about
Updated Insight into the Physiological and Pathological Roles of the Retromer Complex.Internalized TSH receptors en route to the TGN induce local Gs-protein signaling and gene transcriptionTBC1D23 is a bridging factor for endosomal vesicle capture by golgins at the trans-Golgi.Integration of GPCR Signaling and Sorting from Very Early Endosomes via Opposing APPL1 Mechanisms.Cargo-selective SNX-BAR proteins mediate retromer trimer independent retrograde transport.The Dopamine Transporter Recycles via a Retromer-Dependent Postendocytic Mechanism: Tracking Studies Using a Novel Fluorophore-Coupling Approach.New approaches for solving old problems in neuronal protein trafficking.Retromer in Synaptic Function and PathologySNX3-retromer requires an evolutionary conserved MON2:DOPEY2:ATP9A complex to mediate Wntless sorting and Wnt secretion
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Retromer Endosome Exit Domains Serve Multiple Trafficking Destinations and Regulate Local G Protein Activation by GPCRs.
description
2016 nî lūn-bûn
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2016年の論文
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年學術文章
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2016年學術文章
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2016年學術文章
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name
Retromer Endosome Exit Domains ...... G Protein Activation by GPCRs.
@en
Retromer Endosome Exit Domains ...... G Protein Activation by GPCRs.
@nl
type
label
Retromer Endosome Exit Domains ...... G Protein Activation by GPCRs.
@en
Retromer Endosome Exit Domains ...... G Protein Activation by GPCRs.
@nl
prefLabel
Retromer Endosome Exit Domains ...... G Protein Activation by GPCRs.
@en
Retromer Endosome Exit Domains ...... G Protein Activation by GPCRs.
@nl
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Retromer Endosome Exit Domains ...... G Protein Activation by GPCRs.
@en
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Katherine C Varandas
Roshanak Irannejad
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10.1016/J.CUB.2016.09.052
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P577
2016-11-04T00:00:00Z