Interrupting reperfusion as a stroke therapy: ischemic postconditioning reduces infarct size after focal ischemia in rats.
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Ionic homeostasis in brain conditioningFrom rapid to delayed and remote postconditioning: the evolving concept of ischemic postconditioning in brain ischemiaNon-pharmaceutical therapies for stroke: mechanisms and clinical implications.Protein Oxidative Modifications: Beneficial Roles in Disease and HealthIschemic Postconditioning as a Novel Avenue to Protect against Brain Injury after StrokeIsoflurane postconditioning reduces ischemia-induced nuclear factor-κB activation and interleukin 1β production to provide neuroprotection in rats and miceVolatile anesthetic post-treatment induces protection via inhibition of glycogen synthase kinase 3β in human neuron-like cells.Isoflurane induces a postconditioning effect on bovine pulmonary arterial endothelial cells exposed to oxygen-glucose deprivation.Postconditioning with isoflurane reduced ischemia-induced brain injury in rats.Hydroethidine- and MitoSOX-derived red fluorescence is not a reliable indicator of intracellular superoxide formation: another inconvenient truthRevisiting cerebral postischemic reperfusion injury: new insights in understanding reperfusion failure, hemorrhage, and edema.Delayed postconditioning protects against focal ischemic brain injury in rats.Neuroprotection provided by isoflurane pre-conditioning and post-conditioningProtein redox modification as a cellular defense mechanism against tissue ischemic injury.Neuroprotective effect of salvianolate lyophilized injection against cerebral ischemia in type 1 diabetic ratsEmulsified isoflurane postconditioning improves survival and neurological outcomes in a rat model of cardiac arrest.Differential effects of delta and epsilon protein kinase C in modulation of postischemic cerebral blood flow.Ischemic tolerance protects the rat retina from glaucomatous damageMultiple means to the same end: the genetic basis of acquired stress resistance in yeast.Non-invasive remote limb ischemic postconditioning protects rats against focal cerebral ischemia by upregulating STAT3 and reducing apoptosis.The chronic protective effects of limb remote preconditioning and the underlying mechanisms involved in inflammatory factors in rat strokeDerangements of post-ischemic cerebral blood flow by protein kinase C deltaInhibition of autophagy contributes to ischemic postconditioning-induced neuroprotection against focal cerebral ischemia in ratsNox2 knockout delays infarct progression and increases vascular recovery through angiogenesis in mice following ischaemic stroke with reperfusion.Ischemic postconditioning alleviates neuronal injury caused by relief of carotid stenosis in a rat model of cerebral hypoperfusion.Ischemic postconditioning protects neuronal death caused by cerebral ischemia and reperfusion via attenuating protein aggregation.Ischemic postconditioning fails to protect against neonatal cerebral stroke.Ischemic postconditioning decreases cerebral edema and brain blood barrier disruption caused by relief of carotid stenosis in a rat model of cerebral hypoperfusionThe challenge of translating ischemic conditioning from animal models to humans: the role of comorbidities.T cells contribute to stroke-induced lymphopenia in rats.Argon inhalation attenuates retinal apoptosis after ischemia/reperfusion injury in a time- and dose-dependent manner in ratsSignal transducers and activators of transcription: STATs-mediated mitochondrial neuroprotectionLower doses of isoflurane treatment has no beneficial effects in a rat model of intracerebral hemorrhage.Ischemic postconditioning enhances glycogen synthase kinase-3β expression and alleviates cerebral ischemia/reperfusion injury.Postconditioning in major vascular surgery: prevention of renal failure.Protection of ischemic postconditioning against neuronal apoptosis induced by transient focal ischemia is associated with attenuation of NF-κB/p65 activation.The protective effect of early hypothermia on PTEN phosphorylation correlates with free radical inhibition in rat strokeGinsenoside Rg1 suppressed inflammation and neuron apoptosis by activating PPARγ/HO-1 in hippocampus in rat model of cerebral ischemia-reperfusion injury.Rapid conditioning with oxygen oscillation: neuroprotection by intermittent normobaric hyperoxia after transient focal cerebral ischemia in rats.Phosphorylated mitogen-activated protein kinase/extracellular signal-regulated kinase 1/2 may not always represent its kinase activity in a rat model of focal cerebral ischemia with or without ischemic preconditioning.
P2860
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P2860
Interrupting reperfusion as a stroke therapy: ischemic postconditioning reduces infarct size after focal ischemia in rats.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
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2006年學術文章
@zh-hant
name
Interrupting reperfusion as a ...... after focal ischemia in rats.
@en
Interrupting reperfusion as a ...... after focal ischemia in rats.
@nl
type
label
Interrupting reperfusion as a ...... after focal ischemia in rats.
@en
Interrupting reperfusion as a ...... after focal ischemia in rats.
@nl
prefLabel
Interrupting reperfusion as a ...... after focal ischemia in rats.
@en
Interrupting reperfusion as a ...... after focal ischemia in rats.
@nl
P2093
P2860
P356
P1476
Interrupting reperfusion as a ...... after focal ischemia in rats.
@en
P2093
Gary K Steinberg
Robert M Sapolsky
P2860
P304
P356
10.1038/SJ.JCBFM.9600348
P577
2006-05-31T00:00:00Z