Heterogeneity of quadriceps muscle phenotype in chronic obstructive pulmonary disease (Copd); implications for stratified medicine?
about
A randomized controlled trial of angiotensin-converting enzyme inhibition for skeletal muscle dysfunction in COPD.Skeletal muscle adiposity is associated with physical activity, exercise capacity and fibre shift in COPDThe muscle oxidative regulatory response to acute exercise is not impaired in less advanced COPD despite a decreased oxidative phenotypeFHL1 activates myostatin signalling in skeletal muscle and promotes atrophy.Increased expression of H19/miR-675 is associated with a low fat-free mass index in patients with COPD.Growth differentiation factor-15 is associated with muscle mass in chronic obstructive pulmonary disease and promotes muscle wasting in vivo.Evaluation of body composition in COPD patients using multifrequency bioelectrical impedance analysis.Increased skeletal muscle-specific microRNA in the blood of patients with COPD.ACE and response to pulmonary rehabilitation in COPD: two observational studies.MuRF-1 and atrogin-1 protein expression and quadriceps fiber size and muscle mass in stable patients with COPD.Sarcopenia and frailty in chronic respiratory disease.Vastus lateralis fiber shift is an independent predictor of mortality in chronic obstructive pulmonary disease.Fiber atrophy, oxidative stress, and oxidative fiber reduction are the attributes of different phenotypes in chronic obstructive pulmonary disease patients.Evaluating the role of inflammation in chronic airways disease: the ERICA study.Reproducibility of NIRS assessment of muscle oxidative capacity in smokers with and without COPD.Reference values for vastus lateralis fiber type proportion and fiber size.miR-424-5p reduces ribosomal RNA and protein synthesis in muscle wasting.Quadriceps muscle fat infiltration is associated with cardiometabolic risk in COPD.miR-422a suppresses SMAD4 protein expression and promotes resistance to muscle loss.MicroRNA-542 Promotes Mitochondrial Dysfunction and SMAD Activity and Is Elevated in Intensive Care Unit-acquired Weakness.Using laser capture microdissection to study fiber specific signaling in locomotor muscle in COPD: A pilot study.Skeletal Muscle Dysfunction in Chronic Obstructive Pulmonary Disease (COPD): What We Know and Can Do for Our Patients.Can muscle protein metabolism be specifically targeted by exercise training in COPD?Molecular signalling towards mitochondrial breakdown is enhanced in skeletal muscle of patients with chronic obstructive pulmonary disease (COPD)Bioenergetics and intermuscular fat in chronic obstructive pulmonary disease-associated quadriceps weaknessCOPD is accompanied by co-ordinated transcriptional perturbation in the quadriceps affecting the mitochondria and extracellular matrix
P2860
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P2860
Heterogeneity of quadriceps muscle phenotype in chronic obstructive pulmonary disease (Copd); implications for stratified medicine?
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
@zh-my
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2013年學術文章
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name
Heterogeneity of quadriceps mu ...... tions for stratified medicine?
@en
Heterogeneity of quadriceps muscle phenotype in chronic obstructive pulmonary disease
@nl
type
label
Heterogeneity of quadriceps mu ...... tions for stratified medicine?
@en
Heterogeneity of quadriceps muscle phenotype in chronic obstructive pulmonary disease
@nl
prefLabel
Heterogeneity of quadriceps mu ...... tions for stratified medicine?
@en
Heterogeneity of quadriceps muscle phenotype in chronic obstructive pulmonary disease
@nl
P2093
P2860
P50
P356
P1433
P1476
Heterogeneity of quadriceps mu ...... tions for stratified medicine?
@en
P2093
Gemma S Marsh
Ilse G M Slot
John Moxham
Michael I Polkey
Samantha A Natanek
William D-C Man
P2860
P304
P356
10.1002/MUS.23784
P577
2013-08-27T00:00:00Z