Histone deacetylase inhibition in colorectal cancer cells reveals competing roles for members of the oncogenic miR-17-92 cluster. - Wikidata - wikimedia - TriplyDB

Histone deacetylase inhibition in colorectal cancer cells reveals competing roles for members of the oncogenic miR-17-92 cluster.

Histone deacetylase inhibition in colorectal cancer cells reveals competing roles for members of the oncogenic miR-17-92 cluster.

http://www.wikidata.org/entity/Q46221899

about

MicroRNAs as growth regulators, their function and biomarker status in colorectal cancer Non-coding landscapes of colorectal cancer STAT3 and sphingosine-1-phosphate in inflammation-associated colorectal cancer The Role of Bioactive Dietary Components in Modulating miRNA Expression in Colorectal Cancer Diet, microorganisms and their metabolites, and colon cancer.Hsa-miR-19a is associated with lymph metastasis and mediates the TNF-α induced epithelial-to-mesenchymal transition in colorectal cancer Reciprocal regulation between microRNAs and epigenetic machinery in colorectal cancer.MicroRNAs in Cancer Biology and Therapy: Current Status and Perspectives.miR-18a inhibits CDC42 and plays a tumour suppressor role in colorectal cancer cells.Combination of the deacetylase inhibitor panobinostat and the multi-kinase inhibitor sorafenib for the treatment of metastatic hepatocellular carcinoma - review of the underlying molecular mechanisms and first case report.Serum miRNA expression profile as a prognostic biomarker of stage II/III colorectal adenocarcinoma.Butyrate inhibits pro-proliferative miR-92a by diminishing c-Myc-induced miR-17-92a cluster transcription in human colon cancer cells Mechanistic insights into the role of microRNAs in cancer: influence of nutrient crosstalk.Long non-coding RNAs in haematological malignancies.Regulation of microRNAs by epigenetics and their interplay involved in cancer.Effects of supplementation with nondigestible carbohydrates on fecal calprotectin and on epigenetic regulation of SFRP1 expression in the large-bowel mucosa of healthy individuals.Trichostatin A suppresses EGFR expression through induction of microRNA-7 in an HDAC-independent manner in lapatinib-treated cells.miR-18a counteracts AKT and ERK activation to inhibit the proliferation of pancreatic progenitor cells Epigenetic regulation of miRNA-cancer stem cells nexus by nutraceuticals.Linking Diet to Colorectal Cancer: The Emerging Role of MicroRNA in the Communication between Plant and Animal Kingdoms.MicroRNAs targeting EGFR signalling pathway in colorectal cancer.MicroRNAs: potential regulators of renal development genes that contribute to CAKUT.Epigenetically regulated microRNAs and their prospect in cancer diagnosis.Is resistant starch protective against colorectal cancer via modulation of the WNT signalling pathway?Histone deacetylation meets miRNA: epigenetics and post-transcriptional regulation in cancer and chronic diseases.Impact of Histone Deacetylase Inhibitors on microRNA Expression and Cancer Therapy: A Review.Chemopreventive potential of in vitro fermented nuts in LT97 colon adenoma and primary epithelial colon cells.How do tumor cells respond to HDAC inhibition?Involvement of Non-coding RNAs in the Signaling Pathways of Colorectal Cancer.Inhibition of microRNA miR-92a induces apoptosis and inhibits cell proliferation in human acute promyelocytic leukemia through modulation of p63 expression.Non-digestible carbohydrates supplementation increases miR-32 expression in the healthy human colorectal epithelium: A randomized controlled trial.Influence of miRNA-106b and miRNA-135a on butyrate-regulated expression of p21 and Cyclin D2 in human colon adenoma cells.Epigenetic Regulation of Gene Expression Induced by Butyrate in Colorectal Cancer: Involvement of MicroRNA.Green tea phenolics inhibit butyrate-induced differentiation of colon cancer cells by interacting with monocarboxylate transporter 1.Functional polymorphisms in the promoter region of miR-17-92 cluster are associated with a decreased risk of colorectal cancer.Epigenetics and MicroRNAs in Cancer.Genomic imbalances and microRNA transcriptional profiles in patients with mycosis fungoides.SOXC and MiR17-92 gene expression profiling defines two subgroups with different clinical outcome in mantle cell lymphoma.miR-106b-5p inhibits the invasion and metastasis of colorectal cancer by targeting CTSA.Effect of dietary components on miRNA and colorectal carcinogenesis

P2860

Q26775610-C353C984-2CE1-42AA-A4C8-B83D78747523 Q26778014-5EC0BD65-D7E5-4C33-A91A-81110E128B9D Q26998940-F2D8588E-D635-43F3-AD46-4271424348C8 Q28070764-D5F40C77-98CD-43A4-A6EB-F243C341FEF3 Q30241208-91FAEDB1-98E7-447B-9F82-2BC2135606C5 Q33552969-D286A28B-04BF-4EC6-8447-C70070B2A1DD Q33599015-58877054-3156-49E1-AD56-C46BC824B26C Q34599354-F31C52C9-A1F3-4156-A840-5E9AFCCFBBCD Q35402694-68E608FC-8793-463D-B3CE-786D3B7163E7 Q35895346-E38FE98C-9484-4908-9859-A77E3FC4B312 Q35926157-A72549E3-7226-4914-94C4-13B656161B43 Q36157436-7B6E1E39-3C59-400C-B15E-FB58605E709F Q36492992-59DF4E5F-E8C1-4615-8A0B-3C1C358B00FC Q37139037-ED397061-D4C1-42A7-96F4-EAF19D71B10D Q37418150-0AA1061D-A75F-4716-9AF8-00C8140005E9 Q37607632-E2EBABC7-236C-4290-893C-95FECB9B76F0 Q37632279-0E9F2D5F-BD2F-4953-A765-09AFE9EC67E5 Q37717418-CCF1DD0F-6B28-4A48-8B9B-D2D99D3548D5 Q37719054-9AA76B21-6378-4EA5-A4E8-E643B08A08FE Q37737640-DEB3AB51-FD92-4558-81B7-A400AC631D02 Q38118612-FA0E7C17-283F-438B-B77E-1A834F7A7AC5 Q38133419-1F98CAEC-E776-465A-9FEE-C9733AD89180 Q38219046-A10AFE09-ED74-4F07-9373-8BD163846A6F Q38361952-2A3887EA-6080-4BFA-B290-04BBE3357B02 Q38376048-9BCB745C-E0DD-41FE-BA9D-50C12E3E26AD Q38572855-949C0ED5-7813-4E0C-8631-0E66C85C79BA Q38725302-96B9050D-BDF9-40E7-84AE-A4B04A5C04E7 Q38817005-C8B20911-6816-42F5-93CC-B931450F6664 Q38940586-0514D9C0-5004-4C52-9E29-A769EAED9710 Q39028638-DA9847FA-04A3-415B-ACBB-3D3F295EF80F Q41540141-7E56CAE0-D253-4756-91FE-C8421D75B50B Q42094255-7AD580B0-72B3-4ABC-8E9C-90D6EECFA38A Q42365143-5D18C958-5638-411B-86C4-9EF823303EBF Q42746039-E009296E-2D74-4592-985D-95FAE581FA29 Q44216823-AEC40D70-B5E8-4066-A50F-4DEBFC6C5A7C Q51770872-14C70456-ED62-4AE1-AFB0-9A25199C2D4D Q52857590-06291B08-7BA1-4CBB-B038-728DFF0520DE Q53141841-FB0F3184-E60E-4251-98F4-A376C4057E9B Q55669243-6D1C697F-E485-40CB-8656-4D3A6D30C6EB Q58761772-9DD0BDC9-5C0F-4630-BCE0-49F9501D379B

P2860

Histone deacetylase inhibition in colorectal cancer cells reveals competing roles for members of the oncogenic miR-17-92 cluster.

http://www.wikidata.org/entity/Q46221899

description

2012 nî lūn-bûn

@nan

2012年の論文

@ja

2012年学术文章

@wuu

2012年学术文章

@zh-cn

2012年学术文章

@zh-hans

2012年学术文章

@zh-my

2012年学术文章

@zh-sg

2012年學術文章

@yue

2012年學術文章

@zh

2012年學術文章

@zh-hant

name

Histone deacetylase inhibition ...... e oncogenic miR-17-92 cluster.

@en

Histone deacetylase inhibition ...... e oncogenic miR-17-92 cluster.

@nl

type

label

Histone deacetylase inhibition ...... e oncogenic miR-17-92 cluster.

@en

Histone deacetylase inhibition ...... e oncogenic miR-17-92 cluster.

@nl

prefLabel

Histone deacetylase inhibition ...... e oncogenic miR-17-92 cluster.

@en

Histone deacetylase inhibition ...... e oncogenic miR-17-92 cluster.

@nl

P1433

Q46221899-6B82AA01-8316-454F-AE2B-2FF679652342

P1476

Q46221899-FB8F3648-0E2E-4EF1-B548-7117F56502DC

P2093

Q46221899-32488883-3B2A-4DCF-AD3D-1B854B122D43

Q46221899-6640C599-9C98-48F1-B7C6-9815DA89FB22

Q46221899-8C33E383-7645-4987-91B8-3D4562673AB4

Q46221899-A1957B92-32EA-4AB4-8FAF-420FBC32F967

Q46221899-F484436A-C569-4617-A86F-9A807725E288

P2860

Q46221899-0084F10D-0D70-4B35-9B07-190A5926A010

Q46221899-19E5730A-7640-43EF-87AA-470176802C3A

Q46221899-1ED2B4D9-F9CE-40C7-8ECC-C0F8751C4DE9

Q46221899-23531734-3652-49CC-8879-3CB26F51CB69

Q46221899-24EB73BC-34B2-4DDB-844F-8BCB22858D7A

Q46221899-29CF079C-B5CB-47F8-BA7F-6B9C23AC95CD

Q46221899-2D8A0D39-5181-48C6-9F8D-0A9FA8812004

Q46221899-32EDEC22-71D8-4463-AC07-90A93D71119D

Q46221899-3E8D8ED9-7A3F-4D0B-BB07-8AE7D21B9F8D

Q46221899-3F0A50BC-EBA9-4AE7-A9F1-E0CCBC73B10B

P304

Q46221899-D259705F-3852-42FB-9980-0D54903ADD2D

P31

Q46221899-D77D5874-04A2-4A35-AA75-1E2A1267945B

P356

Q46221899-8F88DD69-848F-4E97-A767-B085502E9672

P433

Q46221899-C803DDD5-550C-436E-9658-AAF66613C145

P478

Q46221899-B743A557-A20A-47BC-82E5-6DEE1F1D3AF8

P577

Q46221899-C09C2645-31A2-4B49-94A1-C0F4B4EEDE50

P698

Q46221899-134D9114-DB55-422C-A660-99EC1F3350BB

P921

Q46221899-9E6821CB-BA2D-4061-ABEC-7F7515FE11DF

P356

P1433

Molecular Carcinogenesis

P1476

Histone deacetylase inhibition ...... e oncogenic miR-17-92 cluster.

@en

P2093

Karen J Humphreys

http://www.w3.org/2001/XMLSchema#string

Lynne Cobiac

http://www.w3.org/2001/XMLSchema#string

Mark B Van der Hoek

http://www.w3.org/2001/XMLSchema#string

Michael Z Michael

http://www.w3.org/2001/XMLSchema#string

Richard K Le Leu

http://www.w3.org/2001/XMLSchema#string

P2860

MiR-17-92 cluster is associated with 13q gain and c-myc expression during colorectal adenoma to adenocarcinoma progression

Targeted deletion reveals essential and overlapping functions of the miR-17 through 92 family of miRNA clusters

Repression of the miR-17-92 cluster by p53 has an important function in hypoxia-induced apoptosis

Genetic dissection of the miR-17~92 cluster of microRNAs in Myc-induced B-cell lymphomas

miR-19 is a key oncogenic component of mir-17-92

Mammalian microRNAs predominantly act to decrease target mRNA levels

A microRNA polycistron as a potential human oncogene

Mechanisms of post-transcriptional regulation by microRNAs: are the answers in sight?

c-Myc-regulated microRNAs modulate E2F1 expression

MicroRNA expression profiles classify human cancers

P304

459-474

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1002/MC.21879

http://www.w3.org/2001/XMLSchema#string

P433

6

http://www.w3.org/2001/XMLSchema#string

P478

52

http://www.w3.org/2001/XMLSchema#string

P577

2012-02-05T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

22308110

http://www.w3.org/2001/XMLSchema#string

P921

colorectal cancer