Advanced oxidation protein products induce mesangial cell perturbation through PKC-dependent activation of NADPH oxidase. - Wikidata - wikimedia - TriplyDB

Advanced oxidation protein products induce mesangial cell perturbation through PKC-dependent activation of NADPH oxidase.

Advanced oxidation protein products induce mesangial cell perturbation through PKC-dependent activation of NADPH oxidase.

http://www.wikidata.org/entity/Q46237674

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AOPPs and the progression of kidney disease Rab34 and its effector munc13-2 constitute a new pathway modulating protein secretion in the cellular response to hyperglycemia Dopamine D1 receptor-mediated inhibition of NADPH oxidase activity in human kidney cells occurs via protein kinase A-protein kinase C cross talk.Advanced oxidation protein products sensitized the transient receptor potential vanilloid 1 via NADPH oxidase 1 and 4 to cause mechanical hyperalgesia.Huang Gan Formula Eliminates the Oxidative Stress Effects of Advanced Oxidation Protein Products on the Divergent Regulation of the Expression of AGEs Receptors via the JAK2/STAT3 Pathway.Manganese superoxide dismutase (SOD2) polymorphisms, plasma advanced oxidation protein products (AOPP) concentration and risk of kidney complications in subjects with type 1 diabetes.The protective role of Nrf2 in streptozotocin-induced diabetic nephropathy.PKC-alpha mediates flow-stimulated superoxide production in thick ascending limbs.Advanced Oxidative Protein Products Cause Pain Hypersensitivity in Rats by Inducing Dorsal Root Ganglion Neurons Apoptosis via NADPH Oxidase 4/c-Jun N-terminal Kinase Pathways.Sesquiterpene lactones inhibit advanced oxidation protein product-induced MCP-1 expression in podocytes via an IKK/NF-κB-dependent mechanism.Regulation of NADPH oxidase 5 by protein kinase C isoforms.Lipid raft redox signaling: molecular mechanisms in health and disease.Oxidative stress and galactose-deficient IgA1 as markers of progression in IgA nephropathy.Upstream regulators and downstream effectors of NADPH oxidases as novel therapeutic targets for diabetic kidney disease.Conditioned Medium from Early-Outgrowth Bone Marrow Cells Is Retinal Protective in Experimental Model of Diabetes Mechanisms of angiotensin II-enhanced connecting tubule glomerular feedback.Inflammatory cytokines as predictive markers for early detection and progression of diabetic nephropathy Cell signaling through protein kinase C oxidation and activation.Advanced oxidation protein products activate intrarenal renin-angiotensin system via a CD36-mediated, redox-dependent pathway.Oxidized plasma albumin promotes platelet-endothelial crosstalk and endothelial tissue factor expression.Advanced oxidation protein products induce cardiomyocyte death via Nox2/Rac1/superoxide-dependent TRAF3IP2/JNK signaling.Glucagon-like peptide-1 attenuates advanced oxidation protein product-mediated damage in islet microvascular endothelial cells partly through the RAGE pathway Diabetes and Kidney Disease: Role of Oxidative Stress.NOX Activation by Subunit Interaction and Underlying Mechanisms in Disease.Myofibroblast differentiation during fibrosis: role of NAD(P)H oxidases.IgA nephropathy and oxidative stress: news on clinically evaluated biomarkers hits the stage.Nox4 and diabetic nephropathy: with a friend like this, who needs enemies?Redox signaling as a therapeutic target to inhibit myofibroblast activation in degenerative fibrotic disease Advanced oxidation protein products induce hepatocyte epithelial-mesenchymal transition via a ROS-dependent, TGF-β/Smad signaling pathway.Advanced oxidation protein products decrease expression of nephrin and podocin in podocytes via ROS-dependent activation of p38 MAPK.S-allylcysteine prevents cisplatin-induced nephrotoxicity and oxidative stress.AOPPs (advanced oxidation protein products) promote apoptosis in trophoblastic cells through interference with NADPH oxidase signaling: implications for preeclampsia.Advanced oxidation protein products promote NADPH oxidase-dependent β-cell destruction and dysfunction through the Bcl-2/Bax apoptotic pathway.High-Density Lipoprotein in Chronic Kidney Diseases-The Devil Is in the Detail.Renal expression of advanced oxidative protein products predicts progression of renal fibrosis in patients with IgA nephropathy.Annexin A2 and FTH1 are potential biomarkers for lupus nephritis

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P2860

Advanced oxidation protein products induce mesangial cell perturbation through PKC-dependent activation of NADPH oxidase.

http://www.wikidata.org/entity/Q46237674

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年學術文章

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2008年學術文章

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Advanced oxidation protein pro ...... t activation of NADPH oxidase.

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Advanced oxidation protein pro ...... t activation of NADPH oxidase.

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American Journal of Physiology - Renal Physiology

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Advanced oxidation protein pro ...... t activation of NADPH oxidase.

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Bei Yi Liu

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Fan Fan Hou

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Min Liang

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Qiu Gen Zhou

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Xiao Fan Wei

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P2860

Biochemistry and molecular cell biology of diabetic complications

TGF-beta signal transduction and mesangial cell fibrogenesis

From hyperglycemia to diabetic kidney disease: the role of metabolic, hemodynamic, intracellular factors and growth factors/cytokines

Effects of long-term elevated glucose on collagen formation by mesangial cells

Inhibition of NADPH oxidase prevents advanced glycation end product-mediated damage in diabetic nephropathy through a protein kinase C-alpha-dependent pathway

Extracellular matrix metabolism in diabetic nephropathy.

Protein kinase C-dependent increase in reactive oxygen species (ROS) production in vascular tissues of diabetes: role of vascular NAD(P)H oxidase.

Point mutations in the proline-rich region of p22phox are dominant inhibitors of Nox1- and Nox2-dependent reactive oxygen generation.

Mesangial cell protein kinase C isozyme activation in the diabetic milieu.

Oxidative stress and stress-activated signaling pathways: a unifying hypothesis of type 2 diabetes.

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