Activation of nuclear factor-κB following muscle eccentric contractions in humans is localized primarily to skeletal muscle-residing pericytes.
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Eccentric exercise facilitates mesenchymal stem cell appearance in skeletal muscleDefining a role for non-satellite stem cells in the regulation of muscle repair following exerciseInsights into the molecular etiology of exercise-induced inflammation: opportunities for optimizing performanceSubstrate and strain alter the muscle-derived mesenchymal stem cell secretome to promote myogenesis.Pericytes: multitasking cells in the regeneration of injured, diseased, and aged skeletal muscle.Perivascular cells for regenerative medicine.Altered ROS production, NF-κB activation and interleukin-6 gene expression induced by electrical stimulation in dystrophic mdx skeletal muscle cellsMechanisms modulating skeletal muscle phenotype.Reductive stress after exercise: The issue of redox individuality.Contracting C2C12 myotubes release CCL2 in an NF-κB-dependent manner to induce monocyte chemoattraction.Resistance exercise increases intramuscular NF-κb signaling in untrained males.The acute response of pericytes to muscle-damaging eccentric contraction and protein supplementation in human skeletal muscle.Muscle damage and inflammation during recovery from exercise.Neuromuscular changes and the rapid adaptation following a bout of damaging eccentric exercise.Ibuprofen supplementation and its effects on NF-κB activation in skeletal muscle following resistance exercise.The muscle contraction mode determines lymphangiogenesis differentially in rat skeletal and cardiac muscles by modifying local lymphatic extracellular matrix microenvironments.Pericyte NF-κB activation enhances endothelial cell proliferation and proangiogenic cytokine secretion in vitro.NF-KB activity functions in primary pericytes in a cell- and non-cell-autonomous manner to affect myotube formation.The increase of pericyte population in human neuromuscular disorders supports their role in muscle regeneration in vivo.Polyphenol supplementation alters intramuscular apoptotic signaling following acute resistance exercise.Oral consumption of electrokinetically modified water attenuates muscle damage and improves postexercise recovery.Muscle Stem Cell and Physical Activity: What Point is the Debate at?
P2860
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P2860
Activation of nuclear factor-κB following muscle eccentric contractions in humans is localized primarily to skeletal muscle-residing pericytes.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年学术文章
@wuu
2011年学术文章
@zh-cn
2011年学术文章
@zh-hans
2011年学术文章
@zh-my
2011年学术文章
@zh-sg
2011年學術文章
@yue
2011年學術文章
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2011年學術文章
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name
Activation of nuclear factor-κ ...... tal muscle-residing pericytes.
@en
Activation of nuclear factor-κ ...... tal muscle-residing pericytes.
@nl
type
label
Activation of nuclear factor-κ ...... tal muscle-residing pericytes.
@en
Activation of nuclear factor-κ ...... tal muscle-residing pericytes.
@nl
prefLabel
Activation of nuclear factor-κ ...... tal muscle-residing pericytes.
@en
Activation of nuclear factor-κ ...... tal muscle-residing pericytes.
@nl
P2093
P356
P1433
P1476
Activation of nuclear factor-κ ...... etal muscle-residing pericytes
@en
P2093
Priscilla M Clarkson
Robert D Hyldahl
Stephanie Moeckel-Cole
P304
P356
10.1096/FJ.10-177105
P407
P577
2011-05-20T00:00:00Z