IL-6 induces an anti-inflammatory response in the absence of SOCS3 in macrophages. - Wikidata - wikimedia - TriplyDB

IL-6 induces an anti-inflammatory response in the absence of SOCS3 in macrophages.

IL-6 induces an anti-inflammatory response in the absence of SOCS3 in macrophages.

http://www.wikidata.org/entity/Q46376203

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The role of microtubule-associated protein 1S in SOCS3 regulation of IL-6 signaling Cross-talk between KLF4 and STAT3 regulates axon regeneration Selective regulatory function of Socs3 in the formation of IL-17-secreting T cells Induction of suppressor of cytokine signaling-3 by herpes simplex virus type 1 contributes to inhibition of the interferon signaling pathway Pathological versus protective functions of IL-22 in airway inflammation are regulated by IL-17A Cholinergic agonists regulate JAK2/STAT3 signaling to suppress endothelial cell activation Resistance to IL-10 inhibition of interferon gamma production and expression of suppressor of cytokine signaling 1 in CD4+ T cells from patients with rheumatoid arthritis Signaling by STATs Negative regulation of cytokine signaling and immune responses by SOCS proteins Hepatocellular carcinoma mouse models: Hepatitis B virus-associated hepatocarcinogenesis and haploinsufficient tumor suppressor genes Immunomodulatory functions of type I interferons SOCS3: An essential physiological inhibitor of signaling by interleukin-6 and G-CSF family cytokines The biology and mechanism of action of suppressor of cytokine signaling 3 Modulation of learning and memory by cytokines: signaling mechanisms and long term consequences Beneficial Effects of Anti-Interleukin-6 Antibodies on Impaired Gastrointestinal Motility, Inflammation and Increased Colonic Permeability in a Murine Model of Sepsis Are Most Pronounced When Administered in a Preventive Setup Critical and independent role for SOCS3 in either myeloid or T cells in resistance to Mycobacterium tuberculosis Alpha Interferon Induces Long-Lasting Refractoriness of JAK-STAT Signaling in the Mouse Liver through Induction of USP18/UBP43 SOCS3 binds specific receptor–JAK complexes to control cytokine signaling by direct kinase inhibition SOCS2 negatively regulates growth hormone action in vitro and in vivo MPT0B098, a Microtubule Inhibitor, Suppresses JAK2/STAT3 Signaling Pathway through Modulation of SOCS3 Stability in Oral Squamous Cell Carcinoma Suppressor of cytokine signaling (SOCS)-5 is a potential negative regulator of epidermal growth factor signaling FLN29, a novel interferon- and LPS-inducible gene acting as a negative regulator of toll-like receptor signaling Genetic reduction of embryonic leukemia-inhibitory factor production rescues placentation in SOCS3-null embryos but does not prevent inflammatory disease Dysregulation of suppressor of cytokine signaling 3 in keratinocytes causes skin inflammation mediated by interleukin-20 receptor-related cytokines Tyrosine-phosphorylated SOCS3 interacts with the Nck and Crk-L adapter proteins and regulates Nck activation.Exchange protein activated by cyclic AMP (Epac)-mediated induction of suppressor of cytokine signaling 3 (SOCS-3) in vascular endothelial cells.Interleukin-10 suppression of myeloid cell activation--a continuing puzzle.Influenza A virus inhibits type I IFN signaling via NF-kappaB-dependent induction of SOCS-3 expression.Activation of human monocytes by live Borrelia burgdorferi generates TLR2-dependent and -independent responses which include induction of IFN-beta.Induction of the interleukin 6/ signal transducer and activator of transcription pathway in the lungs of mice sub-chronically exposed to mainstream tobacco smoke.Cannabinoids Delta(9)-tetrahydrocannabinol and cannabidiol differentially inhibit the lipopolysaccharide-activated NF-kappaB and interferon-beta/STAT proinflammatory pathways in BV-2 microglial cells Expression and functional significance of SOCS-1 and SOCS-3 in astrocytes Regulation of adiponectin secretion by adipocytes in the polycystic ovary syndrome: role of tumor necrosis factor-{alpha}.Growth Hormone Resistance-Special Focus on Inflammatory Bowel Disease.Molecular markers of preterm labor in the choriodecidua.Endogenous interleukin-6 plays a crucial protective role in streptococcal toxic shock syndrome via suppression of tumor necrosis factor alpha production.IL-6 trans-signaling via STAT3 directs T cell infiltration in acute inflammation.Stat3: linking inflammation to epithelial cancer - more than a "gut" feeling?The molecular regulation of Janus kinase (JAK) activation SOCS3 regulates graft-versus-host disease.

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P2860

IL-6 induces an anti-inflammatory response in the absence of SOCS3 in macrophages.

http://www.wikidata.org/entity/Q46376203

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年學術文章

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2003年學術文章

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2003年學術文章

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name

IL-6 induces an anti-inflammatory response in the absence of SOCS3 in macrophages.

@en

IL-6 induces an anti-inflammatory response in the absence of SOCS3 in macrophages.

@nl

type

label

IL-6 induces an anti-inflammatory response in the absence of SOCS3 in macrophages.

@en

IL-6 induces an anti-inflammatory response in the absence of SOCS3 in macrophages.

@nl

prefLabel

IL-6 induces an anti-inflammatory response in the absence of SOCS3 in macrophages.

@en

IL-6 induces an anti-inflammatory response in the absence of SOCS3 in macrophages.

@nl

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Nature Immunology

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IL-6 induces an anti-inflammatory response in the absence of SOCS3 in macrophages.

@en

P2093

Akihiko Yoshimura

http://www.w3.org/2001/XMLSchema#string

Daisuke Aki

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Hideo Yasukawa

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Hiroyuki Mori

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Kenneth R Chien

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Kiyoshi Takeda

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Masaaki Murakami

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Masahiko Hoshijima

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Masanobu Ohishi

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Shizuo Akira

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551-556

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scholarly article

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10.1038/NI938

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6

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4

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12754507

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