Effects of maternal hypoxia or nutrient restriction during pregnancy on endothelial function in adult male rat offspring. - Wikidata - wikimedia - TriplyDB

Effects of maternal hypoxia or nutrient restriction during pregnancy on endothelial function in adult male rat offspring.

Effects of maternal hypoxia or nutrient restriction during pregnancy on endothelial function in adult male rat offspring.

http://www.wikidata.org/entity/Q46387179

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Fetal programming and early identification of newborns at high risk of free radical-mediated diseases Gender differences in developmental programming of cardiovascular diseases Intrauterine programming Antenatal hypoxia induces programming of reduced arterial blood pressure response in female rat offspring: role of ovarian function.Developmental programming of cardiovascular dysfunction by prenatal hypoxia and oxidative stress Effect of fetal anaemia on myocardial ischaemia-reperfusion injury and coronary vasoreactivity in adult sheep Hypoxia-induced intrauterine growth restriction increases the susceptibility of rats to high-fat diet-induced metabolic syndrome Maternal treatment with agonistic autoantibodies against type-1 angiotensin II receptor in late pregnancy increases apoptosis of myocardial cells and myocardial susceptibility to ischemia-reperfusion injury in offspring rats.Hypoxia stimulates the proliferation of neonatal rat vascular smooth muscle cells through activation of hypoxia-inducible factor-1α Chronic hypoxia in pregnancy affected vascular tone of renal interlobar arteries in the offspring.Antenatal maternal hypoxia: criterion for fetal growth restriction in rodents.Pre-eclampsia and offspring cardiovascular health: mechanistic insights from experimental studies.Fetal programming of hypertension.Intrauterine programming of physiological systems: causes and consequences.Intrauterine growth restriction: fetal programming of hypertension and kidney disease.Role of the hypothalamic-pituitary-adrenal axis in developmental programming of health and disease.Sex differences in the enhanced responsiveness to acute angiotensin II in growth-restricted rats: role of fasudil, a Rho kinase inhibitor.Melatonin rescues cardiovascular dysfunction during hypoxic development in the chick embryo Long-term effects of prenatal hypoxia on endothelium-dependent relaxation responses in pulmonary arteries of adult sheep Divergence of mechanistic pathways mediating cardiovascular aging and developmental programming of cardiovascular disease Role of fetal programming in the development of hypertension.Rate of weight gain in very-low birth weight Puerto Rican neonates.Developmental programming and hypertension.Developmental programming of cardiovascular disease by prenatal hypoxia.Impact of oxidative stress during pregnancy on fetal epigenetic patterns and early origin of vascular diseases.Interactions between local dilator and sympathetic vasoconstrictor influences in skeletal muscle in acute and chronic hypoxia.Prenatal hypoxia leads to hypertension, renal renin-angiotensin system activation and exacerbates salt-induced pathology in a sex-specific manner.In Utero Origins of Hypertension: Mechanisms and Targets for Therapy.A review of fundamental principles for animal models of DOHaD research: an Australian perspective.Maternal vascular responses to hypoxia in a rat model of intrauterine growth restriction.Prenatal hypoxia in rats increased blood pressure and sympathetic drive of the adult offspring.Gestational hypoxia disrupts the neonatal leptin surge and programs hyperphagia and obesity in male offspring in the Sprague-Dawley rat Effects of maternal hypoxia on muscle vasodilatation evoked by acute systemic hypoxia in adult rat offspring: changed roles of adenosine and A1 receptors.Perinatal L-arginine and antioxidant supplements reduce adult blood pressure but not ameliorate the altered vascular function in spontaneously hypertensive rats.Mechanisms of endothelium-dependent vasodilation in male and female, young and aged offspring born growth restricted.Maternal undernutrition leads to endothelial dysfunction in adult male rat offspring independent of postnatal diet.Muscarinic receptor M1 and phosphodiesterase 1 are key determinants in pulmonary vascular dysfunction following perinatal hypoxia in mice.Maternal undernutrition inhibits angiogenesis in the offspring: a potential mechanism of programmed hypertension.Prenatal Hypoxia Induced Dysfunction in Cerebral Arteries of Offspring Rats.A Low Cerebroplacental Ratio at 20-24 Weeks of Gestation Can Predict Reduced Fetal Size Later in Pregnancy or at Birth.

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Effects of maternal hypoxia or nutrient restriction during pregnancy on endothelial function in adult male rat offspring.

http://www.wikidata.org/entity/Q46387179

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JPHYSIOL.2005.084889

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The Journal of Physiology

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Effects of maternal hypoxia or ...... n in adult male rat offspring.

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Denise G Hemmings

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I Caroline McMillen

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Jana M Mitchell

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Sandra T Davidge

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Sarah J Williams

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P2860

Inhibition of smooth muscle cell growth by nitric oxide and activation of cAMP-dependent protein kinase by cGMP

Dietary restriction in pregnant rats causes gender-related hypertension and vascular dysfunction in offspring

Protection of vascular wall function in insulin-resistant rats from copper oxidative stress

Impaired acetylcholine-induced vascular relaxation in low birth weight infants: implications for adult hypertension?

Impaired endothelial function and increased carotid stiffness in 9-year-old children with low birthweight.

Preterm birth attenuates association between low birth weight and endothelial dysfunction.

Impact of low birth weight and cardiovascular risk factors on endothelial function in early adult life.

The role of size at birth and postnatal catch-up growth in determining systolic blood pressure: a systematic review of the literature.

The fetal origins hypothesis: placental insufficiency and inheritance versus maternal malnutrition in well-nourished populations.

Enhanced peroxynitrite formation is associated with vascular aging

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