Examining the interaction of apo E and neurotoxicity on a murine model of ALS-PDC.
about
Beyond Guam: the cyanobacteria/BMAA hypothesis of the cause of ALS and other neurodegenerative diseases.Neurodegenerative diseases: neurotoxins as sufficient etiologic agents?Exposure to environmental toxicants and pathogenesis of amyotrophic lateral sclerosis: state of the art and research perspectives.Full-length apolipoprotein E protects against the neurotoxicity of an apoE-related peptideChronic exposure to dietary sterol glucosides is neurotoxic to motor neurons and induces an ALS-PDC phenotype.The BSSG rat model of Parkinson's disease: progressing towards a valid, predictive model of disease.Magnesium deficiency over generations in rats with special references to the pathogenesis of the Parkinsonism-dementia complex and amyotrophic lateral sclerosis of Guam.
P2860
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P2860
Examining the interaction of apo E and neurotoxicity on a murine model of ALS-PDC.
description
2005 nî lūn-bûn
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2005年の論文
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年學術文章
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name
Examining the interaction of apo E and neurotoxicity on a murine model of ALS-PDC.
@en
Examining the interaction of apo E and neurotoxicity on a murine model of ALS-PDC.
@nl
type
label
Examining the interaction of apo E and neurotoxicity on a murine model of ALS-PDC.
@en
Examining the interaction of apo E and neurotoxicity on a murine model of ALS-PDC.
@nl
prefLabel
Examining the interaction of apo E and neurotoxicity on a murine model of ALS-PDC.
@en
Examining the interaction of apo E and neurotoxicity on a murine model of ALS-PDC.
@nl
P2093
P2860
P356
P1476
Examining the interaction of apo E and neurotoxicity on a murine model of ALS-PDC.
@en
P2093
Moghadasian MH
P2860
P304
P356
10.1139/Y04-140
P577
2005-02-01T00:00:00Z