Tryptophan depletion under conditions that imitate insulin resistance enhances fatty acid oxidation and induces endothelial dysfunction through reactive oxygen species-dependent and independent pathways.
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Indoleamine 2, 3-dioxygenase (IDO) increases during renal fibrogenesis and its inhibition potentiates TGF-β 1-induced epithelial to mesenchymal transition.Allopurinol protects human glomerular endothelial cells from high glucose-induced reactive oxygen species generation, p53 overexpression and endothelial dysfunction.What May Constrain the Success of Indoleamine 2,3-Dioxygenase 1 Inhibitors in Cancer Immunotherapy?
P2860
Tryptophan depletion under conditions that imitate insulin resistance enhances fatty acid oxidation and induces endothelial dysfunction through reactive oxygen species-dependent and independent pathways.
description
2017 nî lūn-bûn
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2017年の論文
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年學術文章
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2017年學術文章
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name
Tryptophan depletion under con ...... dent and independent pathways.
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Tryptophan depletion under con ...... dent and independent pathways.
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type
label
Tryptophan depletion under con ...... dent and independent pathways.
@en
Tryptophan depletion under con ...... dent and independent pathways.
@nl
prefLabel
Tryptophan depletion under con ...... dent and independent pathways.
@en
Tryptophan depletion under con ...... dent and independent pathways.
@nl
P2093
P2860
P1476
Tryptophan depletion under con ...... ndent and independent pathways
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P2093
Christos Rountas
Georgia Antoniadi
Loannis Stefanidis
Maria Sounidaki
Vassilios Liakopoulos
P2860
P2888
P356
10.1007/S11010-016-2915-7
P577
2017-02-04T00:00:00Z