Na+-K+-ATPase properties in rat heart and skeletal muscle 3 mo after coronary artery ligation.
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Complement Destabilizes Cardiomyocyte Function In Vivo after Polymicrobial Sepsis and In Vitro.Na+ dysregulation coupled with Ca2+ entry through NCX1 promotes muscular dystrophy in mice.Large fibre size in skeletal muscle is metabolically advantageous.Pivotal role of α2 Na+ pumps and their high affinity ouabain binding site in cardiovascular health and disease.Inhibition of Na/K-ATPase promotes myocardial tumor necrosis factor-alpha protein expression and cardiac dysfunction via calcium/mTOR signaling in endotoxemia.
P2860
Na+-K+-ATPase properties in rat heart and skeletal muscle 3 mo after coronary artery ligation.
description
2005 nî lūn-bûn
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2005年の論文
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年学术文章
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2005年學術文章
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2005年學術文章
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name
Na+-K+-ATPase properties in ra ...... fter coronary artery ligation.
@en
Na+-K+-ATPase properties in ra ...... fter coronary artery ligation.
@nl
type
label
Na+-K+-ATPase properties in ra ...... fter coronary artery ligation.
@en
Na+-K+-ATPase properties in ra ...... fter coronary artery ligation.
@nl
prefLabel
Na+-K+-ATPase properties in ra ...... fter coronary artery ligation.
@en
Na+-K+-ATPase properties in ra ...... fter coronary artery ligation.
@nl
P2093
P2860
P1476
Na+-K+-ATPase properties in ra ...... fter coronary artery ligation.
@en
P2093
P2860
P304
P356
10.1152/JAPPLPHYSIOL.00343.2004
P407
P577
2005-04-07T00:00:00Z