Epidermal growth factor receptor (EGFR)-MAPK-nuclear factor(NF)-κB-IL8: A possible mechanism of particulate matter(PM) 2.5-induced lung toxicity.

Epidermal growth factor receptor (EGFR)-MAPK-nuclear factor(NF)-κB-IL8: A possible mechanism of particulate matter(PM) 2.5-induced lung toxicity.

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http://www.wikidata.org/entity/Q46430781
Metabolomics analysis reveals that benzo[a]pyrene, a component of PM2.5, promotes pulmonary injury by modifying lipid metabolism in a phospholipase A2-dependent manner in vivo and in vitro.Flavone protects HBE cells from DNA double-strand breaks caused by PM2.5.Urban particulate matter triggers lung inflammation via the ROS-MAPK-NF-κB signaling pathway.From the Cover: Lung-Specific Overexpression of Constitutively Active IKK2 Induces Pulmonary and Systemic Inflammations but Not Hypothalamic Inflammation and Glucose Intolerance.Concentrated Ambient PM2.5-Induced Inflammation and Endothelial Dysfunction in a Murine Model of Neural IKK2 Deficiency.Function of PM2.5 in the pathogenesis of lung cancer and chronic airway inflammatory diseases.Harmful impact of air pollution on severe acute exacerbation of chronic obstructive pulmonary disease: particulate matter is hazardous.
P2860

Epidermal growth factor receptor (EGFR)-MAPK-nuclear factor(NF)-κB-IL8: A possible mechanism of particulate matter(PM) 2.5-induced lung toxicity.

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http://www.wikidata.org/entity/Q46430781
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2017 nî lūn-bûn
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2017年の論文
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2017年学术文章
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2017年学术文章
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name
Epidermal growth factor recept ...... PM) 2.5-induced lung toxicity.
@en
type
label
Epidermal growth factor recept ...... PM) 2.5-induced lung toxicity.
@en
prefLabel
Epidermal growth factor recept ...... PM) 2.5-induced lung toxicity.
@en
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Epidermal growth factor recept ...... PM) 2.5-induced lung toxicity.
@en
P2093
P2860
P304
P31
P356
P433
P478
P577
P698
P921