Angiogenic imbalance as a contributor to the pathophysiology of preeclampsia among black African women.

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Angiogenic imbalance as a contributor to the pathophysiology of preeclampsia among black African women.

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2016 nî lūn-bûn

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2016年の論文

@ja

2016年学术文章

@wuu

2016年学术文章

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2016年学术文章

@zh-cn

2016年学术文章

@zh-hans

2016年学术文章

@zh-my

2016年学术文章

@zh-sg

2016年學術文章

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2016年學術文章

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Angiogenic imbalance as a cont ...... sia among black African women.

@en

Angiogenic imbalance as a cont ...... sia among black African women.

@nl

type

Item

label

Angiogenic imbalance as a cont ...... sia among black African women.

@en

Angiogenic imbalance as a cont ...... sia among black African women.

@nl

prefLabel

Angiogenic imbalance as a cont ...... sia among black African women.

@en

Angiogenic imbalance as a cont ...... sia among black African women.

@nl

P1476

Angiogenic imbalance as a cont ...... sia among black African women.

@en

P2093

Allen Meeme

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Ambrose V Namugowa

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Geoffrey A Buga

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MaryKutty Mammen

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P2860

Inhibition of vascular endothelial cell growth factor activity by an endogenously encoded soluble receptor

Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia

Accuracy of mean arterial pressure and blood pressure measurements in predicting pre-eclampsia: systematic review and meta-analysis

Abnormalities in oxygen sensing define early and late onset preeclampsia as distinct pathologies.

Is fetal growth restriction associated with a more severe maternal phenotype in the setting of early onset pre-eclampsia? A retrospective study

Latest advances in understanding preeclampsia.

Soluble FLT1 sensitizes endothelial cells to inflammatory cytokines by antagonizing VEGF receptor-mediated signalling

Preeclampsia, a disease of the maternal endothelium: the role of antiangiogenic factors and implications for later cardiovascular disease.

The genetics of pre-eclampsia: a feto-placental or maternal problem?

Vascular endothelial growth factor ligands and receptors that regulate human cytotrophoblast survival are dysregulated in severe preeclampsia and hemolysis, elevated liver enzymes, and low platelets syndrome.

P304

1335-1341

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P31

scholarly article

P356

10.1080/14767058.2016.1212832

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P433

http://www.w3.org/2001/XMLSchema#string

P478

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P577

2016-08-08T00:00:00Z

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P698

27500318

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P921

pathophysiology

pre-eclampsia