Impact of combined C1 esterase inhibitor/coagulation factor XIII or N-acetylcysteine/tirilazad mesylate administration on leucocyte adherence and cytokine release in experimental endotoxaemia.

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Impact of combined C1 esterase inhibitor/coagulation factor XIII or N-acetylcysteine/tirilazad mesylate administration on leucocyte adherence and cytokine release in experimental endotoxaemia.

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http://www.wikidata.org/entity/Q46464286

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2008 nî lūn-bûn

@nan

2008年の論文

@ja

2008年学术文章

@wuu

2008年学术文章

@zh

2008年学术文章

@zh-cn

2008年学术文章

@zh-hans

2008年学术文章

@zh-my

2008年学术文章

@zh-sg

2008年學術文章

@yue

2008年學術文章

@zh-hant

name

Impact of combined C1 esterase ...... in experimental endotoxaemia.

@en

Impact of combined C1 esterase ...... in experimental endotoxaemia.

@nl

type

Item

label

Impact of combined C1 esterase ...... in experimental endotoxaemia.

@en

Impact of combined C1 esterase ...... in experimental endotoxaemia.

@nl

prefLabel

Impact of combined C1 esterase ...... in experimental endotoxaemia.

@en

Impact of combined C1 esterase ...... in experimental endotoxaemia.

@nl

P1476

Impact of combined C1 esterase ...... in experimental endotoxaemia.

@en

P2093

C D Spies

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C Lehmann

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E Klotz

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J Birnbaum

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J Feller

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J Mueller

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O Vargas Hein

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P2860

Human recombinant activated protein C for severe sepsis

N-acetylcysteine decreases lactate signal intensities in liver tissue and improves liver function in septic shock patients, as shown by magnetic resonance spectroscopy: extended case report

Is there a place for N-acetylcysteine in the treatment of septic shock?

C1 inhibitor prevents Gram-negative bacterial lipopolysaccharide-induced vascular permeability.

The role of oxidative stress in adult critical care.

Role of oxidative stress in experimental sepsis and multisystem organ dysfunction.

Effects of coagulation factor XIII on intestinal functional capillary density, leukocyte adherence and mesenteric plasma extravasation in experimental endotoxemia.

Effect of factor XIII on endothelial barrier function.

N-acetylcysteine attenuates TNF-alpha induced changes in secretion of interleukin-6, plasminogen activator inhibitor-1 and adiponectin from 3T3-L1 adipocytes.

Post-treatment with N-acetylcysteine ameliorates endotoxin shock-induced organ damage in conscious rats.

P304

748-759

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P31

scholarly article

P356

10.1177/147323000803600417

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2008-07-01T00:00:00Z

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P5875

51426207

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P698

18652771

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