Sex modulates the arrhythmogenic substrate in prepubertal rabbit hearts with Long QT 2. - Wikidata - wikimedia - TriplyDB

Sex modulates the arrhythmogenic substrate in prepubertal rabbit hearts with Long QT 2.

Sex modulates the arrhythmogenic substrate in prepubertal rabbit hearts with Long QT 2.

http://www.wikidata.org/entity/Q46475673

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Sex differences in the mechanisms underlying long QT syndrome Sex, age, and regional differences in L-type calcium current are important determinants of arrhythmia phenotype in rabbit hearts with drug-induced long QT type 2 A multiscale computational modelling approach predicts mechanisms of female sex risk in the setting of arousal-induced arrhythmias.Drug-induced long QT syndrome Sex differences in β-adrenergic responsiveness of action potentials and intracellular calcium handling in isolated rabbit hearts Autonomic nerve stimulation reverses ventricular repolarization sequence in rabbit hearts Deciphering Arrhythmia Mechanisms - Tools of the Trade WITHDRAWN: Sex-differences in sodium/calcium exchange expression is a determinant of the arrhythmia phenotype in pre-pubertal rabbit hearts with Long QT type 2.How do sex hormones modify arrhythmogenesis in long QT syndrome? Sex hormone effects on arrhythmogenic substrate and triggered activity.QT prolongation and proarrhythmia by moxifloxacin: concordance of preclinical models in relation to clinical outcome.Spatial heterogeneity of the restitution portrait in rabbit epicardium.The link between abnormal calcium handling and electrical instability in acquired long QT syndrome--Does calcium precipitate arrhythmic storms?Increased late sodium current contributes to long QT-related arrhythmia susceptibility in female mice.Role of abnormal repolarization in the mechanism of cardiac arrhythmia.Genomic upregulation of cardiac Cav1.2α and NCX1 by estrogen in women.Sex and regional differences in rabbit right ventricular L-type calcium current levels and mathematical modelling of arrhythmia vulnerability.Oestrogen upregulates L-type Ca²⁺ channels via oestrogen-receptor- by a regional genomic mechanism in female rabbit hearts.Regional genomic regulation of cardiac sodium-calcium exchanger by oestrogen.

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P2860

Sex modulates the arrhythmogenic substrate in prepubertal rabbit hearts with Long QT 2.

http://www.wikidata.org/entity/Q46475673

description

2005 nî lūn-bûn

@nan

2005年の論文

@ja

2005年学术文章

@wuu

2005年学术文章

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2005年学术文章

@zh-cn

2005年学术文章

@zh-hans

2005年学术文章

@zh-my

2005年学术文章

@zh-sg

2005年學術文章

@yue

2005年學術文章

@zh-hant

name

Sex modulates the arrhythmogenic substrate in prepubertal rabbit hearts with Long QT 2.

@en

Sex modulates the arrhythmogenic substrate in prepubertal rabbit hearts with Long QT 2.

@nl

type

label

Sex modulates the arrhythmogenic substrate in prepubertal rabbit hearts with Long QT 2.

@en

Sex modulates the arrhythmogenic substrate in prepubertal rabbit hearts with Long QT 2.

@nl

prefLabel

Sex modulates the arrhythmogenic substrate in prepubertal rabbit hearts with Long QT 2.

@en

Sex modulates the arrhythmogenic substrate in prepubertal rabbit hearts with Long QT 2.

@nl

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J.1540-8167.2005.40622.X

P1433

Journal of Cardiovascular Electrophysiology

P1476

Sex modulates the arrhythmogenic substrate in prepubertal rabbit hearts with Long QT 2.

@en

P2093

Bum-Rak Choi

http://www.w3.org/2001/XMLSchema#string

Guy Salama

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Milou-Daniel Drici

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Tong Liu

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P2860

Long QT syndromes and torsade de pointes.

Sex, hormones, and repolarization.

Modulating effects of age and gender on the clinical course of long QT syndrome by genotype.

The molecular genetics of the long QT syndrome: genes causing fainting and sudden death.

Age- and sex-related differences in clinical manifestations in patients with congenital long-QT syndrome: findings from the International LQTS Registry.

Differential effects of beta-adrenergic agonists and antagonists in LQT1, LQT2 and LQT3 models of the long QT syndrome.

Torsade de pointes due to noncardiac drugs: most patients have easily identifiable risk factors.

Two components of cardiac delayed rectifier K+ current. Differential sensitivity to block by class III antiarrhythmic agents.

Multiple mechanisms in the long-QT syndrome. Current knowledge, gaps, and future directions. The SADS Foundation Task Force on LQTS.

Early afterdepolarizations: mechanism of induction and block. A role for L-type Ca2+ current.

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516-524

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scholarly article

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10.1046/J.1540-8167.2005.40622.X

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5

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16

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15877623

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