Kv7/KCNQ/M and HCN/h, but not KCa2/SK channels, contribute to the somatic medium after-hyperpolarization and excitability control in CA1 hippocampal pyramidal cells.
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Dissecting mechanisms of brain aging by studying the intrinsic excitability of neuronsPolarized axonal surface expression of neuronal KCNQ channels is mediated by multiple signals in the KCNQ2 and KCNQ3 C-terminal domainsSelective positive modulation of the SK3 and SK2 subtypes of small conductance Ca2+-activated K+ channelsNew molecular targets for antiepileptic drugs: alpha(2)delta, SV2A, and K(v)7/KCNQ/M potassium channelsMolecular targets for antiepileptic drug developmentDiverse impact of acute and long-term extracellular proteolytic activity on plasticity of neuronal excitabilityAcute alterations of somatodendritic action potential dynamics in hippocampal CA1 pyramidal cells after kainate-induced status epilepticus in micePacemaking Property of RVLM Presympathetic NeuronsMechanisms of Action of Antiseizure Drugs and the Ketogenic DietNeurotransmitter modulation of small-conductance Ca2+-activated K+ channels by regulation of Ca2+ gatingContribution of KCNQ2 and KCNQ3 to the medium and slow afterhyperpolarization currentsM-type potassium channels modulate the intrinsic excitability of infralimbic neurons and regulate fear expression and extinctionM-type potassium channels modulate Schaffer collateral-CA1 glutamatergic synaptic transmission.Homomeric and heteromeric assembly of KCNQ (Kv7) K+ channels assayed by total internal reflection fluorescence/fluorescence resonance energy transfer and patch clamp analysis.A tale of switched functions: from cyclooxygenase inhibition to M-channel modulation in new diphenylamine derivativesSelective interaction of syntaxin 1A with KCNQ2: possible implications for specific modulation of presynaptic activity.Molecular and cellular basis of small--and intermediate-conductance, calcium-activated potassium channel function in the brain.Identification of postsynaptic phosphatidylinositol-4,5-bisphosphate (PIP2) roles for synaptic plasticity using chemically induced dimerizationGain control in CA1 pyramidal cells using changes in somatic conductance.Learning and aging related changes in intrinsic neuronal excitabilityThe KCNQ5 potassium channel mediates a component of the afterhyperpolarization current in mouse hippocampus.Deletion of the L-type calcium channel Ca(V) 1.3 but not Ca(V) 1.2 results in a diminished sAHP in mouse CA1 pyramidal neurons.Polarized axonal surface expression of neuronal KCNQ potassium channels is regulated by calmodulin interaction with KCNQ2 subunit.Inhibition of post-synaptic Kv7/KCNQ/M channels facilitates long-term potentiation in the hippocampusThe slow afterhyperpolarization: a target of β1-adrenergic signaling in hippocampus-dependent memory retrieval.Voltage-gated potassium channels at the crossroads of neuronal function, ischemic tolerance, and neurodegeneration.HCN Channel Targets for Novel Antidepressant Treatment.Dynamic interplay of excitatory and inhibitory coupling modes of neuronal L-type calcium channels.N-methyl-D-aspartate receptors mediate activity-dependent down-regulation of potassium channel genes during the expression of homeostatic intrinsic plasticity.Oxidative modification of M-type K(+) channels as a mechanism of cytoprotective neuronal silencing.Contributions of Kv7-mediated potassium current to sub- and suprathreshold responses of rat layer II/III neocortical pyramidal neurons.Bidirectional pattern-specific plasticity of the slow afterhyperpolarization in rats: role for high-voltage activated Ca2+ channels and I h.Kv7.2 regulates the function of peripheral sensory neurons.Fasting and 17β-estradiol differentially modulate the M-current in neuropeptide Y neuronsMetaplastic effect of apamin on LTP and paired-pulse facilitationAdult mouse basal forebrain harbors two distinct cholinergic populations defined by their electrophysiology.Increasing SK2 channel activity impairs associative learningElectrophysiological Assessment of Serotonin and GABA Neuron Function in the Dorsal Raphe during the Third Trimester Equivalent Developmental Period in Mice.Maternal immune activation produces neonatal excitability defects in offspring hippocampal neurons from pregnant rats treated with poly I:CContribution of near-threshold currents to intrinsic oscillatory activity in rat medial entorhinal cortex layer II stellate cells.
P2860
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P2860
Kv7/KCNQ/M and HCN/h, but not KCa2/SK channels, contribute to the somatic medium after-hyperpolarization and excitability control in CA1 hippocampal pyramidal cells.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Kv7/KCNQ/M and HCN/h, but not ...... 1 hippocampal pyramidal cells.
@en
Kv7/KCNQ/M and HCN/h, but not ...... 1 hippocampal pyramidal cells.
@nl
type
label
Kv7/KCNQ/M and HCN/h, but not ...... 1 hippocampal pyramidal cells.
@en
Kv7/KCNQ/M and HCN/h, but not ...... 1 hippocampal pyramidal cells.
@nl
prefLabel
Kv7/KCNQ/M and HCN/h, but not ...... 1 hippocampal pyramidal cells.
@en
Kv7/KCNQ/M and HCN/h, but not ...... 1 hippocampal pyramidal cells.
@nl
P2860
P1476
Kv7/KCNQ/M and HCN/h, but not ...... A1 hippocampal pyramidal cells
@en
P2093
Johan F Storm
P2860
P304
P356
10.1113/JPHYSIOL.2005.086835
P407
P577
2005-05-12T00:00:00Z