Ablation of LGR4 promotes energy expenditure by driving white-to-brown fat switch.
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LGR4 and Its Role in Intestinal Protection and Energy MetabolismLkb1 controls brown adipose tissue growth and thermogenesis by regulating the intracellular localization of CRTC3Redox regulation of endothelial cell fateMany obesity-associated SNPs strongly associate with DNA methylation changes at proximal promoters and enhancers.The association between the baseline bone resorption marker CTX and incident dysglycemia after 4 yearsOsteocyte-Secreted Wnt Signaling Inhibitor Sclerostin Contributes to Beige Adipogenesis in Peripheral Fat Depots.Brown and beige fat in humans: thermogenic adipocytes that control energy and glucose homeostasis.Immune regulation of metabolic homeostasis in health and disease.Inhibition of Sam68 triggers adipose tissue browning.Selection of Suitable Reference Genes for Quantitative Real-Time PCR Normalization in Three Types of Rat Adipose TissueBrown adipogenic potential of brown adipocytes and peri-renal adipocytes from human embryo.The R-spondin/Lgr5/Rnf43 module: regulator of Wnt signal strengthG protein-coupled receptors in energy homeostasis.The progress and challenges in metabolic research in China.Innate lymphoid cells as novel regulators of obesity and its-associated metabolic dysfunction.Taking control over intracellular fatty acid levels is essential for the analysis of thermogenic function in cultured primary brown and brite/beige adipocytes.Gut microbiome and serum metabolome alterations in obesity and after weight-loss intervention.Induction of thermogenic adipocytes: molecular targets and thermogenic small molecules.Association of a gain-of-function variant in LGR4 with central obesity.Adipocyte-specific deletion of mTOR inhibits adipose tissue development and causes insulin resistance in mice.A self-sustained loop of inflammation-driven inhibition of beige adipogenesis in obesity.Cell-Autonomous Brown-Like Adipogenesis of Preadipocytes From Retinoblastoma Haploinsufficient Mice.IRX3 Promotes the Browning of White Adipocytes and Its Rare Variants are Associated with Human Obesity Risk.Obesity: a gateway disease with a rising prevalence.Wnt, RSPO and Hippo Signalling in the Intestine and Intestinal Stem Cells.LGR4 modulates breast cancer initiation, metastasis, and cancer stem cells.Genetic interaction of DGAT2 and FAAH in the development of human obesity.AMP-Activated Protein Kinase (AMPK) Regulates Energy Metabolism through Modulating Thermogenesis in Adipose Tissue.Grape seed proanthocyanidin extract ameliorates inflammation and adiposity by modulating gut microbiota in high-fat diet mice.LGR4 is a receptor for RANKL and negatively regulates osteoclast differentiation and bone resorption.R-spondin3-LGR4 signaling protects hepatocytes against DMOG-induced hypoxia/reoxygenation injury through activating β-catenin.
P2860
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P2860
Ablation of LGR4 promotes energy expenditure by driving white-to-brown fat switch.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年学术文章
@wuu
2013年学术文章
@zh
2013年学术文章
@zh-cn
2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
@yue
2013年學術文章
@zh-hant
name
Ablation of LGR4 promotes energy expenditure by driving white-to-brown fat switch.
@en
Ablation of LGR4 promotes energy expenditure by driving white-to-brown fat switch.
@nl
type
label
Ablation of LGR4 promotes energy expenditure by driving white-to-brown fat switch.
@en
Ablation of LGR4 promotes energy expenditure by driving white-to-brown fat switch.
@nl
prefLabel
Ablation of LGR4 promotes energy expenditure by driving white-to-brown fat switch.
@en
Ablation of LGR4 promotes energy expenditure by driving white-to-brown fat switch.
@nl
P2093
P2860
P921
P356
P1433
P1476
Ablation of LGR4 promotes energy expenditure by driving white-to-brown fat switch.
@en
P2093
Guang Ning
Maopei Chen
Mingyao Liu
Ruixin Liu
P2860
P2888
P304
P356
10.1038/NCB2867
P50
P577
2013-11-10T00:00:00Z
P5875
P6179
1041812871