The anti-inflammatory compound curcumin inhibits Neisseria gonorrhoeae-induced NF-kappaB signaling, release of pro-inflammatory cytokines/chemokines and attenuates adhesion in late infection.

The anti-inflammatory compound curcumin inhibits Neisseria gonorrhoeae-induced NF-kappaB signaling, release of pro-inflammatory cytokines/chemokines and attenuates adhesion in late infection.

Item
http://www.wikidata.org/entity/Q46520617
Curcumin and its derivatives: their application in neuropharmacology and neuroscience in the 21st centuryThe anti-inflammatory activity of curcumin protects the genital mucosal epithelial barrier from disruption and blocks replication of HIV-1 and HSV-2Meningococcal porin PorB prevents cellular apoptosis in a toll-like receptor 2- and NF-kappaB-independent mannerEpithelial proinflammatory response and curcumin-mediated protection from staphylococcal toxic shock syndrome toxin-1Neisseria gonorrhoeae-mediated inhibition of apoptotic signalling in polymorphonuclear leukocytes.PPARalpha and PPARgamma effectively protect against HIV-induced inflammatory responses in brain endothelial cellsThe Okinawan diet: health implications of a low-calorie, nutrient-dense, antioxidant-rich dietary pattern low in glycemic load.The prevalence of Chlamydia trachomatis and Mycoplasma genitalium tubal infections and their effects on the expression of IL-6 and leukaemia inhibitory factor in Fallopian tubes with and without an ectopic pregnancy.Multifaceted roles of curcumin: two sides of a coin!Molecular mechanisms of curcumin action: gene expression.Expression of estrogen receptor alpha increases leptin-induced STAT3 activity in breast cancer cells.Curcumin and its analogues: a potential natural compound against HIV infection and AIDS.An overview of structure-activity relationship studies of curcumin analogs as antioxidant and anti-inflammatory agents.Topical curcumin can inhibit deleterious effects of upper respiratory tract bacteria on human oropharyngeal cells in vitro: potential role for patients with cancer therapy induced mucositis?IFN-gamma amplifies NFkappaB-dependent Neisseria meningitidis invasion of epithelial cells via specific upregulation of CEA-related cell adhesion molecule 1.Curcumin attenuates high glucose-induced inflammatory injury through the reactive oxygen species-phosphoinositide 3-kinase/protein kinase B-nuclear factor-κB signaling pathway in rat thoracic aorta endothelial cells.LIGHT enhances the bactericidal activity of human monocytes and neutrophils via HVEM.
P2860

The anti-inflammatory compound curcumin inhibits Neisseria gonorrhoeae-induced NF-kappaB signaling, release of pro-inflammatory cytokines/chemokines and attenuates adhesion in late infection.

Item
http://www.wikidata.org/entity/Q46520617
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
The anti-inflammatory compound ...... es adhesion in late infection.
@en
The anti-inflammatory compound ...... es adhesion in late infection.
@nl
type
label
The anti-inflammatory compound ...... es adhesion in late infection.
@en
The anti-inflammatory compound ...... es adhesion in late infection.
@nl
prefLabel
The anti-inflammatory compound ...... es adhesion in late infection.
@en
The anti-inflammatory compound ...... es adhesion in late infection.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P433
P478
P50
P577
P698
P921
P356
P1433
P1476
The anti-inflammatory compound ...... es adhesion in late infection.
@en
P2093
P2860
P304
P31
P356
P433
P478
P50
P577
P698
P921