Disrupting GluA2 phosphorylation potentiates reinstatement of cocaine seeking.

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Disrupting GluA2 phosphorylation potentiates reinstatement of cocaine seeking.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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Disrupting GluA2 phosphorylation potentiates reinstatement of cocaine seeking.

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Disrupting GluA2 phosphorylation potentiates reinstatement of cocaine seeking.

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Disrupting GluA2 phosphorylation potentiates reinstatement of cocaine seeking.

@en

Disrupting GluA2 phosphorylation potentiates reinstatement of cocaine seeking.

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Disrupting GluA2 phosphorylation potentiates reinstatement of cocaine seeking.

@en

Disrupting GluA2 phosphorylation potentiates reinstatement of cocaine seeking.

@nl

P1476

Disrupting GluA2 phosphorylation potentiates reinstatement of cocaine seeking.

@en

P2093

Andre U Deutschmann

http://www.w3.org/2001/XMLSchema#string

Anne Q Fosnocht

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P2860

NEURAL MECHANISMS OF ADDICTION: The Role of Reward-Related Learning and Memory

Phosphorylation of the AMPA receptor subunit GluR2 differentially regulates its interaction with PDZ domain-containing proteins

Phosphorylation of serine-880 in GluR2 by protein kinase C prevents its C terminus from binding with glutamate receptor-interacting protein

Cocaine-predictive stimulus induces drug-seeking behavior and neural activation in limbic brain regions after multiple months of abstinence: reversal by D(1) antagonists

Phosphorylation of the AMPA receptor GluR1 subunit is required for synaptic plasticity and retention of spatial memory

Cocaine-induced neuroadaptations in glutamate transmission: potential therapeutic targets for craving and addiction

Cell surface AMPA receptors in the rat nucleus accumbens increase during cocaine withdrawal but internalize after cocaine challenge in association with altered activation of mitogen-activated protein kinases

Detection of spontaneous synaptic events with an optimally scaled template.

AMPA receptor synaptic plasticity induced by psychostimulants: the past, present, and therapeutic future

Persistent inflammation-induced up-regulation of brain-derived neurotrophic factor (BDNF) promotes synaptic delivery of α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor GluA1 subunits in descending pain modulatory circuits.

P304

231-241

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P31

scholarly article

P356

10.1016/J.NEUROPHARM.2016.09.010

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P478

111

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P50

Lisa A Briand

Alexandra S Ellis

P577

2016-09-10T00:00:00Z

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P698

27622930

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P932

5074807

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