Induction of GRP78 by ischemic preconditioning reduces endoplasmic reticulum stress and prevents delayed neuronal cell death.
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The divergent roles of autophagy in ischemia and preconditioningRegulatory proteins of eukaryotic initiation factor 2-alpha subunit (eIF2 alpha) phosphatase, under ischemic reperfusion and toleranceThe role of the unfolded protein response in the heart.Ischemic preconditioning and clinical scenarios.Adaptation to ER stress is mediated by differential stabilities of pro-survival and pro-apoptotic mRNAs and proteinsExpression of two endoplasmic reticulum stress markers, GRP78 and GADD153, in rat retinal detachment model and its implication.Preconditioning-induced ischemic tolerance: a window into endogenous gearing for cerebroprotectionAge and energy intake interact to modify cell stress pathways and stroke outcome.Down-regulation of GRP78 in human glaucomatous trabecular meshwork cells.Protein misfolding induces hypoxic preconditioning via a subset of the unfolded protein response machinery.Glutamine treatment attenuates endoplasmic reticulum stress and apoptosis in TNBS-induced colitisHypothermia protects the brain from transient global ischemia/reperfusion by attenuating endoplasmic reticulum response-induced apoptosis through CHOP.Rifampicin protects PC12 cells from rotenone-induced cytotoxicity by activating GRP78 via PERK-eIF2α-ATF4 pathway.Endoplasmic reticulum chaperone GRP78 is involved in autophagy activation induced by ischemic preconditioning in neural cells.Effects of hyperbaric oxygen on glucose-regulated protein 78 and c-Jun N-terminal kinase expression after spinal cord injury in rats.Ischaemic preconditioning: therapeutic implications for stroke?Key note lecture: toward a mechanistic taxonomy for cell death programs.Ischemic neuronal cell death and organellae damage.Attenuating the endoplasmic reticulum stress response improves functional recovery after spinal cord injury.Retinoic Acid Induced-Autophagic Flux Inhibits ER-Stress Dependent Apoptosis and Prevents Disruption of Blood-Spinal Cord Barrier after Spinal Cord Injury.Ischemic preconditioning prevents protein aggregation after transient cerebral ischemia.IRE1-RACK1 axis orchestrates ER stress preconditioning-elicited cytoprotection from ischemia/reperfusion injury in liverEndoplasmic reticulum stress.A point mutation in translation initiation factor 2B leads to a continuous hyper stress state in oligodendroglial-derived cells.Endoplasmic reticulum stress preconditioning attenuates methylmercury-induced cellular damage by inducing favorable stress responses.Sulfur mustard induces an endoplasmic reticulum stress response in the mouse ear vesicant model.Cellular and molecular neurobiology of brain preconditioning.Inositol 1,4,5-triphosphate receptors and NAD(P)H mediate Ca2+ signaling required for hypoxic preconditioning of hippocampal neurons.Ischemic tolerance as an active and intrinsic neuroprotective mechanism.Modulation of stress proteins and apoptotic regulators in the anoxia tolerant turtle brain.Advanced osteoarthritis in humans is associated with altered collagen VI expression and upregulation of ER-stress markers Grp78 and bag-1.Unfolded protein response signaling and metabolic diseases.Acidic pre-conditioning suppresses apoptosis and increases expression of Bcl-xL in coronary endothelial cells under simulated ischaemia.The endoplasmic reticulum stress inhibitor salubrinal inhibits the activation of autophagy and neuroprotection induced by brain ischemic preconditioning.Advances in research of the neuroprotective mechanisms of cerebral ischemic postconditioning.Oxygen-Glucose Deprivation (OGD) Modulates the Unfolded Protein Response (UPR) and Inflicts Autophagy in a PC12 Hypoxia Cell Line Model.Unfolded protein response in brain ischemia: A timely update.Novel anti-oxidative role of calreticulin in protecting A549 human type II alveolar epithelial cells against hypoxic injury.Comparative proteomic analysis of human donor tissues during orthotopic liver transplantation: ischemia versus reperfusion.Neuroprotective effects of atorvastatin against cerebral ischemia/reperfusion injury through the inhibition of endoplasmic reticulum stress.
P2860
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P2860
Induction of GRP78 by ischemic preconditioning reduces endoplasmic reticulum stress and prevents delayed neuronal cell death.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Induction of GRP78 by ischemic ...... s delayed neuronal cell death.
@en
Induction of GRP78 by ischemic ...... s delayed neuronal cell death.
@nl
type
label
Induction of GRP78 by ischemic ...... s delayed neuronal cell death.
@en
Induction of GRP78 by ischemic ...... s delayed neuronal cell death.
@nl
prefLabel
Induction of GRP78 by ischemic ...... s delayed neuronal cell death.
@en
Induction of GRP78 by ischemic ...... s delayed neuronal cell death.
@nl
P2093
P2860
P1476
Induction of GRP78 by ischemic ...... s delayed neuronal cell death.
@en
P2093
Atsushi Saito
Michel Ferrand-Drake
Pak H Chan
Shuzo Okuno
Takeshi Hayashi
P2860
P304
P356
10.1097/01.WCB.0000077641.41248.EA
P577
2003-08-01T00:00:00Z
P6179
1032400751