High-fat diet-induced obesity leads to increased NO sensitivity of rat coronary arterioles: role of soluble guanylate cyclase activation. - Wikidata - wikimedia - TriplyDB

High-fat diet-induced obesity leads to increased NO sensitivity of rat coronary arterioles: role of soluble guanylate cyclase activation.

High-fat diet-induced obesity leads to increased NO sensitivity of rat coronary arterioles: role of soluble guanylate cyclase activation.

http://www.wikidata.org/entity/Q46648152

about

The beneficial role of vitamin D in obesity: possible genetic and cell signaling mechanisms Diet-induced obesity impairs endothelium-derived hyperpolarization via altered potassium channel signaling mechanisms Dietary obesity increases NO and inhibits BKCa-mediated, endothelium-dependent dilation in rat cremaster muscle artery: association with caveolins and caveolae.Local oxidative and nitrosative stress increases in the microcirculation during leukocytes-endothelial cell interactions.Caveolin-1 limits the contribution of BK(Ca) channel to EDHF-mediated arteriolar dilation: implications in diet-induced obesity.Hypoxic relaxation of penile arteries: involvement of endothelial nitric oxide and modulation by reactive oxygen species.Arginase 1 contributes to diminished coronary arteriolar dilation in patients with diabetes Differential vulnerability of skeletal muscle feed arteries to dysfunction in insulin resistance: impact of fiber type and daily activity.Caveolin-1 prevents sustained angiotensin II-induced resistance artery constriction and obesity-induced high blood pressure.Influence of obesity and metabolic dysfunction on the endothelial control in the coronary circulation.Microvascular responsiveness in obesity: implications for therapeutic intervention.The Beta-1-Receptor Blocker Nebivolol Elicits Dilation of Cerebral Arteries by Reducing Smooth Muscle [Ca2+]i Increased risk for the development of preeclampsia in obese pregnancies: weighing in on the mechanisms.Hypertension in metabolic syndrome: vascular pathophysiology.Preserved endothelium-dependent dilatation of the coronary microvasculature at the early phase of diabetes mellitus despite the increased oxidative stress and depressed cardiac mechanical function ex vivo.Erectile dysfunction precedes coronary artery endothelial dysfunction in rats fed a high-fat, high-sucrose, Western pattern diet Endothelial regulation of coronary microcirculation in health and cardiometabolic diseases.Increased tissue angiotensin-converting enzyme activity impairs bradykinin-induced dilation of coronary arterioles in obesity Longitudinal analysis of short-term high-fat diet on endothelial senescence in baboons.Selective up-regulation of arginase-1 in coronary arteries of diabetic patients.Dietary supplementation with a specific melon concentrate reverses vascular dysfunction induced by cafeteria diet.Mechanisms of coronary microvascular adaptation to obesity.Microvascular dysfunction in the course of metabolic syndrome induced by high-fat diet.Nitrate decreases xanthine oxidoreductase-mediated nitrite reductase activity and attenuates vascular and blood pressure responses to nitrite.Body adiposity dictates different mechanisms of increased coronary reactivity related to improved in vivo cardiac function.Molecular imaging with optical coherence tomography using ligand-conjugated microparticles that detect activated endothelial cells: rational design through target quantification Early impairment of coronary microvascular perfusion capacity in rats on a high fat diet.Activation of hexosamine pathway impairs nitric oxide (NO)-dependent arteriolar dilations by increased protein O-GlcNAcylation.Cardiovascular, metabolic, and coronary dysfunction in high-fat-fed obesity-resistant/prone rats.Thioredoxin-interacting protein is required for endothelial NLRP3 inflammasome activation and cell death in a rat model of high-fat diet Progression and reversal of coronary and mesenteric vascular dysfunction associated with obesity.Coronary microvascular dysfunction after long-term diabetes and hypercholesterolemia.Role of Adipose Tissue Endothelial ADAM17 in Age-Related Coronary Microvascular Dysfunction.

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High-fat diet-induced obesity leads to increased NO sensitivity of rat coronary arterioles: role of soluble guanylate cyclase activation.

http://www.wikidata.org/entity/Q46648152

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2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年學術文章

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2008年學術文章

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name

High-fat diet-induced obesity ...... guanylate cyclase activation.

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High-fat diet-induced obesity ...... guanylate cyclase activation.

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High-fat diet-induced obesity ...... guanylate cyclase activation.

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High-fat diet-induced obesity ...... guanylate cyclase activation.

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High-fat diet-induced obesity ...... guanylate cyclase activation.

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AJPHEART.01198.2007

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American Journal of Physiology - Heart and Circulatory Physiology

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High-fat diet-induced obesity ...... guanylate cyclase activation.

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Akos Koller

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Attila Feher

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Csaba Kiraly

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Eniko T Pasztor

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Eva Jebelovszki

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Ibolya Rutkai

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Istvan Edes

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Nora Erdei

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Tamas Forster

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Zsolt Bagi

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P2860

Increased nitrovasodilator sensitivity in endothelial nitric oxide synthase knockout mice: role of soluble guanylyl cyclase.

Endothelial dysfunction in obesity and insulin resistance: a road to diabetes and heart disease.

Matching coronary blood flow to myocardial oxygen consumption.

Impact of obesity on the risk of heart failure and survival after the onset of heart failure.

Events at the host-microbial interface of the gastrointestinal tract. II. Role of the intestinal epithelium in pathogen-induced inflammation.

Vascular consequences of endothelial nitric oxide synthase uncoupling for the activity and expression of the soluble guanylyl cyclase and the cGMP-dependent protein kinase.

Cardiovascular adaptation to obesity and hypertension.

Diet-induced changes in endothelial-dependent relaxation of the rat aorta.

Impaired NO-dependent dilation of skeletal muscle arterioles in hypertensive diabetic obese Zucker rats.

Impaired endothelium-mediated relaxation in isolated cerebral arteries from insulin-resistant rats.

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10.1152/AJPHEART.01198.2007

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