Vascular neointimal formation and signaling pathway activation in response to stent injury in insulin-resistant and diabetic animals.
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Coronary artery revascularization in patients with diabetes mellitusNeointimal hyperplasia persists at six months after sirolimus-eluting stent implantation in diabetic porcineHeparanase alters arterial structure, mechanics, and repair following endovascular stenting in mice.Sera from patients with diabetes do not alter the effect of mammalian target of rapamycin inhibition on smooth muscle cell proliferation.Effect of metabolic syndrome on the response to arterial injuryEverolimus-eluting stents improve vascular response in a diabetic animal model.Insulin enhances the effect of nitric oxide at inhibiting neointimal hyperplasia in a rat model of type 1 diabetesAkt pathway is hypoactivated by synergistic actions of diabetes mellitus and hypercholesterolemia resulting in advanced coronary artery disease.Loss of canonical insulin signaling accelerates vascular smooth muscle cell proliferation and migration through changes in p27Kip1 regulation.Revascularization for coronary artery disease in diabetes mellitus: angioplasty, stents and coronary artery bypass grafting.Leukotriene-C4 synthase, a critical enzyme in the activation of store-independent Orai1/Orai3 channels, is required for neointimal hyperplasia.Adventitial contributions of the extracellular signal-regulated kinase and Akt pathways to neointimal hyperplasia.Comparative vascular responses three months after paclitaxel and everolimus-eluting stent implantation in streptozotocin-induced diabetic porcine coronary arteries.Relative resistance to Mammalian target of rapamycin inhibition in vascular smooth muscle cells of diabetic donors.Alagebrium inhibits neointimal hyperplasia and restores distributions of wall shear stress by reducing downstream vascular resistance in obese and diabetic rats.Dysregulation of the Mammalian Target of Rapamycin and p27Kip1 Promotes Intimal Hyperplasia in Diabetes MellitusHistopathological Comparison among Biolimus, Zotarolimus and Everolimus-Eluting Stents in Porcine Coronary Restenosis Model.A novel mouse model of in situ stenting.Role of metabolic environment on nitric oxide mediated inhibition of neointimal hyperplasia in type 1 and type 2 diabetes.Contribution of insulin resistance to vascular dysfunction.Nitric oxide differentially affects ERK and Akt in type 1 and type 2 diabetic rats.Clinical science review article: understanding the implications of diabetes on the vascular system.Smooth muscle cells orchestrate the endothelial cell response to flow and injury.Insulin augments matrix metalloproteinase-9 expression in monocytes.Enhanced proliferation and migration of vascular smooth muscle cells in response to vascular injury under hyperglycemic conditions is controlled by beta3 integrin signaling.Modulation of TGF-β/BMP-6 expression and increased levels of circulating smooth muscle progenitor cells in a type I diabetes mouse model.C-Peptide in insulin resistance and vascular complications: teaching an old dog new tricks.Upregulation of miR-221 and -222 in response to increased extracellular signal-regulated kinases 1/2 activity exacerbates neointimal hyperplasia in diabetes mellitus.Carotid artery stenting in the Zucker rat: a novel, potentially 'diabetes-specific' model of in-stent restenosis.
P2860
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P2860
Vascular neointimal formation and signaling pathway activation in response to stent injury in insulin-resistant and diabetic animals.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Vascular neointimal formation ...... esistant and diabetic animals.
@en
Vascular neointimal formation ...... esistant and diabetic animals.
@nl
type
label
Vascular neointimal formation ...... esistant and diabetic animals.
@en
Vascular neointimal formation ...... esistant and diabetic animals.
@nl
prefLabel
Vascular neointimal formation ...... esistant and diabetic animals.
@en
Vascular neointimal formation ...... esistant and diabetic animals.
@nl
P2093
P1433
P1476
Vascular neointimal formation ...... esistant and diabetic animals.
@en
P2093
Aaron B Baker
Adam Groothuis
Campbell Rogers
Elazer R Edelman
Michael Jonas
Philip Seifert
P304
P356
10.1161/01.RES.0000183730.52908.C6
P577
2005-08-25T00:00:00Z