Diverse properties of store-operated TRPC channels activated by protein kinase C in vascular myocytes.
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Orai1 calcium channels in the vasculatureRat hypocretin/orexin neurons are maintained in a depolarized state by TRPC channelsROS-dependent signaling mechanisms for hypoxic Ca(2+) responses in pulmonary artery myocytes.Reduction in TRPC4 expression specifically attenuates G-protein coupled receptor-stimulated increases in intracellular calcium in human myometrial cells.mGluR1-mediated excitation of cerebellar GABAergic interneurons requires both G protein-dependent and Src-ERK1/2-dependent signaling pathwaysOrai1 interacts with STIM1 and mediates capacitative Ca2+ entry in mouse pulmonary arterial smooth muscle cells.Protein kinases modulate store-operated channels in pulmonary artery smooth muscle cells.TRPC channels in smooth muscle cellsCholecystokinin facilitates neuronal excitability in the entorhinal cortex via activation of TRPC-like channels.TRPC1 proteins confer PKC and phosphoinositol activation on native heteromeric TRPC1/C5 channels in vascular smooth muscle: comparative study of wild-type and TRPC1-/- mice.Enhanced store-operated Ca²+ entry and TRPC channel expression in pulmonary arteries of monocrotaline-induced pulmonary hypertensive rats.Transient receptor potential channels in the vasculature.Inositol trisphosphate receptors in smooth muscle cells.Pharmacological profile of phosphatidylinositol 3-kinases and related phosphatidylinositols mediating endothelin(A) receptor-operated native TRPC channels in rabbit coronary artery myocytesActivation of transient receptor potential canonical 3 (TRPC3)-mediated Ca2+ entry by A1 adenosine receptor in cardiomyocytes disturbs atrioventricular conductionStore depletion induces Gαq-mediated PLCβ1 activity to stimulate TRPC1 channels in vascular smooth muscle cells.Transient receptor potential canonical 7: a diacylglycerol-activated non-selective cation channelType 1 inositol 1,4,5-trisphosphate receptors mediate UTP-induced cation currents, Ca2+ signals, and vasoconstriction in cerebral arteries.Smooth muscle cell calcium activation mechanisms.Increased rhythmicity in hypertensive arterial smooth muscle is linked to transient receptor potential canonical channels.Myristoylated alanine-rich C kinase substrate coordinates native TRPC1 channel activation by phosphatidylinositol 4,5-bisphosphate and protein kinase C in vascular smooth muscleOrai1, a critical component of store-operated Ca2+ entry, is functionally associated with Na+/Ca2+ exchanger and plasma membrane Ca2+ pump in proliferating human arterial myocytes.TRPC channel lipid specificity and mechanisms of lipid regulationPharmacology of transient receptor potential melastatin channels in the vasculatureTargeting TRPC6 channels in oesophageal carcinoma growth.Properties and therapeutic potential of transient receptor potential channels with putative roles in adversity: focus on TRPC5, TRPM2 and TRPA1.The closing and opening of TRPC channels by Homer1 and STIM1.Integration of transient receptor potential canonical channels with lipids.Effect of diindolylmethane on Ca2+ homeostasis and viability in PC3 human prostate cancer cells.Potential effect of resistin on the ET-1-increased reactions of blood pressure in rats and Ca2+ signaling in vascular smooth muscle cells.Effect of diindolylmethane on Ca(2+) movement and viability in HA59T human hepatoma cells.Effect of methoxychlor on Ca2+ handling and viability in OC2 human oral cancer cells.Chemotaxis of MDCK-F cells toward fibroblast growth factor-2 depends on transient receptor potential canonical channel 1.Evidence that Orai1 does not contribute to store-operated TRPC1 channels in vascular smooth muscle cells.Transient receptor potential canonical 5 channels plays an essential role in hepatic dyslipidemia associated with cholestasis.Activation of native TRPC1/C5/C6 channels by endothelin-1 is mediated by both PIP3 and PIP2 in rabbit coronary artery myocytesObligatory role for phosphatidylinositol 4,5-bisphosphate in activation of native TRPC1 store-operated channels in vascular myocytes.Store-operated interactions between plasmalemmal STIM1 and TRPC1 proteins stimulate PLCβ1 to induce TRPC1 channel activation in vascular smooth muscle cells.TRPC6 channels stimulated by angiotensin II are inhibited by TRPC1/C5 channel activity through a Ca2+- and PKC-dependent mechanism in native vascular myocytes.Ins(1,4,5)P3 interacts with PIP2 to regulate activation of TRPC6/C7 channels by diacylglycerol in native vascular myocytes.
P2860
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P2860
Diverse properties of store-operated TRPC channels activated by protein kinase C in vascular myocytes.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Diverse properties of store-op ...... kinase C in vascular myocytes.
@en
Diverse properties of store-op ...... kinase C in vascular myocytes.
@nl
type
label
Diverse properties of store-op ...... kinase C in vascular myocytes.
@en
Diverse properties of store-op ...... kinase C in vascular myocytes.
@nl
prefLabel
Diverse properties of store-op ...... kinase C in vascular myocytes.
@en
Diverse properties of store-op ...... kinase C in vascular myocytes.
@nl
P2860
P1476
Diverse properties of store-op ...... kinase C in vascular myocytes
@en
P2093
Sohag N Saleh
William A Large
P2860
P304
P356
10.1113/JPHYSIOL.2008.152157
P407
P577
2008-03-20T00:00:00Z