Involvement of hypoxia-triggered endoplasmic reticulum stress in outlet obstruction-induced apoptosis in the urinary bladder.
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Reciprocal regulation between proinflammatory cytokine-induced inducible NO synthase (iNOS) and connexin43 in bladder smooth muscle cellsEndoplasmic reticulum stress and unfolded protein response in renal pathophysiology: Janus facesCaspase-12 ablation preserves muscle function in the mdx mouse.Activation of the unfolded protein response is associated with pulmonary hypertension.AMPK Suppresses Connexin43 Expression in the Bladder and Ameliorates Voiding Dysfunction in Cyclophosphamide-induced Mouse Cystitis.Benefits of mesenchymal stem cells after partial bladder outlet obstruction.miR-663 overexpression induced by endoplasmic reticulum stress modulates hepatocellular carcinoma cell apoptosis via transforming growth factor beta 1.Eviprostat activates cAMP signaling pathway and suppresses bladder smooth muscle cell proliferation.The chemical chaperone 4-phenylbutyrate inhibits adipogenesis by modulating the unfolded protein response.Impact of partial urethral obstruction on bladder function: time-dependent changes and functional correlates of altered expression of Ca²⁺ signaling regulators.A molecular web: endoplasmic reticulum stress, inflammation, and oxidative stress.Inhibition of HIF Reduces Bladder Hypertrophy and Improves Bladder Function in Murine Model of Partial Bladder Outlet Obstruction.A Chinese Medicine Formula "Xian-Jia-Tang" for Treating Bladder Outlet Obstruction by Improving Urodynamics and Inhibiting Oxidative Stress through Potassium Channels.The sphingosine-1-phosphate pathway is upregulated in response to partial urethral obstruction in male rats and activates RhoA/Rho-kinase signalling.Urodynamic improvements following oral medical therapy for partial bladder outlet obstruction in an animal model.4-phenylbutyric acid attenuates endoplasmic reticulum stress-mediated pancreatic β-cell apoptosis in rats with streptozotocin-induced diabetes.Benefits and limitations of animal models in partial bladder outlet obstruction for translational research.Effects of varying degrees of partial bladder outlet obstruction on urinary bladder function of rats: A novel link to inflammation, oxidative stress and hypoxia.The inflammatory cytokine IL-1β is involved in bladder remodeling after bladder outlet obstruction in mice.Discrepancy of uterine leiomyoma and myometrium to hypoxia-induced endoplasmic reticulum stress after uterine occlusion therapy accounts for therapeutic effect.
P2860
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P2860
Involvement of hypoxia-triggered endoplasmic reticulum stress in outlet obstruction-induced apoptosis in the urinary bladder.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Involvement of hypoxia-trigger ...... ptosis in the urinary bladder.
@en
Involvement of hypoxia-trigger ...... ptosis in the urinary bladder.
@nl
type
label
Involvement of hypoxia-trigger ...... ptosis in the urinary bladder.
@en
Involvement of hypoxia-trigger ...... ptosis in the urinary bladder.
@nl
prefLabel
Involvement of hypoxia-trigger ...... ptosis in the urinary bladder.
@en
Involvement of hypoxia-trigger ...... ptosis in the urinary bladder.
@nl
P2093
P2860
P1476
Involvement of hypoxia-trigger ...... optosis in the urinary bladder
@en
P2093
Isao Araki
Masanori Kitamura
Masayuki Takeda
Nobuhiko Hiramatsu
Norifumi Sawada
P2860
P2888
P304
P356
10.1038/LABINVEST.2008.21
P577
2008-03-17T00:00:00Z