IL-21R on T cells is critical for sustained functionality and control of chronic viral infection. - Wikidata - wikimedia - TriplyDB

IL-21R on T cells is critical for sustained functionality and control of chronic viral infection.

IL-21R on T cells is critical for sustained functionality and control of chronic viral infection.

http://www.wikidata.org/entity/Q46697142

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HIV-1-specific interleukin-21+ CD4+ T cell responses contribute to durable viral control through the modulation of HIV-specific CD8+ T cell function Interleukin-21-producing HIV-1-specific CD8 T cells are preferentially seen in elite controllers IL-21 Signaling in Immunity Molecular and cellular insights into T cell exhaustion Overcoming T cell exhaustion in infection and cancer Revealing the role of CD4(+) T cells in viral immunity Distinct roles of CD4+ T cell subpopulations in retroviral immunity: lessons from the Friend virus mouse model The role of interleukin-21 in HIV infection CD4 T-cell immunotherapy for chronic viral infections and cancer Early Loss of Splenic Tfh Cells in SIV-Infected Rhesus Macaques Maintenance of intestinal Th17 cells and reduced microbial translocation in SIV-infected rhesus macaques treated with interleukin (IL)-21 Crystal Structure of Interleukin-21 Receptor (IL-21R) Bound to IL-21 Reveals That Sugar Chain Interacting with WSXWS Motif Is Integral Part of IL-21R Innate and Adaptive Immune Regulation During Chronic Viral Infections Immunology. A chronic need for IL-21 IL-21: roles in immunopathology and cancer therapy Human TYK2 deficiency: Mycobacterial and viral infections without hyper-IgE syndrome HIV and Tuberculosis: a Deadly Human Syndemic IL-21R signaling suppresses IL-17+ gamma delta T cell responses and production of IL-17 related cytokines in the lung at steady state and after Influenza A virus infection.CD4+ T cells contain early extrapulmonary tuberculosis (TB) dissemination and rapid TB progression and sustain multieffector functions of CD8+ T and CD3- lymphocytes: mechanisms of CD4+ T cell immunity.Anatomy of viral persistence.Type I interferon suppresses de novo virus-specific CD4 Th1 immunity during an established persistent viral infection.CD40 signaling to the rescue: A CD8 exhaustion perspective in chronic infectious diseases IL-10 directly suppresses CD4 but not CD8 T cell effector and memory responses following acute viral infection.Induction of Gag-specific CD4 T cell responses during acute HIV infection is associated with improved viral control.Shaping successful and unsuccessful CD8 T cell responses following infection.IL-21-mediated reversal of NK cell exhaustion facilitates anti-tumour immunity in MHC class I-deficient tumours Polyfunctional CD4+ T cell responses to a set of pathogenic arenaviruses provide broad population coverage Inflammatory cytokines as a third signal for T cell activation Opposing positive and negative regulation of T cell activity during viral persistence.Chronic viral infection promotes sustained Th1-derived immunoregulatory IL-10 via BLIMP-1 A little 'help' from IL-21 during persistent viral infection Interleukin-21: a new mediator of inflammation in systemic lupus erythematosus.Immune activation is associated with CD8 T cell interleukin-21 production in HIV-1-infected individuals.Persistence of viral infection despite similar killing efficacy of antiviral CD8(+) T cells during acute and chronic phases of infection.Interplay between regulatory T cells and PD-1 in modulating T cell exhaustion and viral control during chronic LCMV infection.T-cell exhaustion: characteristics, causes and conversion.Pronounced virus-dependent activation drives exhaustion but sustains IFN-γ transcript levels.Enhancing T Cell Immune Responses by B Cell-based Therapeutic Vaccine Against Chronic Virus Infection.Ex vivo expansion of human CD8+ T cells using autologous CD4+ T cell help Minimal effect of CD103 expression on the control of a chronic antiviral immune response.

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IL-21R on T cells is critical for sustained functionality and control of chronic viral infection.

http://www.wikidata.org/entity/Q46697142

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

@zh-sg

2009年學術文章

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2009年學術文章

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2009年學術文章

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name

IL-21R on T cells is critical ...... ol of chronic viral infection.

@en

IL-21R on T cells is critical ...... ol of chronic viral infection.

@nl

type

label

IL-21R on T cells is critical ...... ol of chronic viral infection.

@en

IL-21R on T cells is critical ...... ol of chronic viral infection.

@nl

prefLabel

IL-21R on T cells is critical ...... ol of chronic viral infection.

@en

IL-21R on T cells is critical ...... ol of chronic viral infection.

@nl

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IL-21R on T cells is critical ...... ol of chronic viral infection.

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Abdijapar T Shamshiev

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Anja Fröhlich

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Benjamin J Marsland

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Iwana Schmitz

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Jacqueline Weber

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P2860

Interleukin-10 determines viral clearance or persistence in vivo

Restoring function in exhausted CD8 T cells during chronic viral infection

Interleukin-21: Basic Biology and Implications for Cancer and Autoimmunity

PD-1 expression on HIV-specific T cells is associated with T-cell exhaustion and disease progression

The role of CD4(+) T helper cells in the cytotoxic T lymphocyte response to HIV-1.

The CD4(+) T cell response to HIV-1.

Cytokine requirements for acute and Basal homeostatic proliferation of naive and memory CD8+ T cells.

Heterogeneity and cell-fate decisions in effector and memory CD8+ T cell differentiation during viral infection

Resolution of a chronic viral infection after interleukin-10 receptor blockade

Interleukin 15 is required for proliferative renewal of virus-specific memory CD8 T cells

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5934

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324

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Stefan Freigang

Annette Oxenius

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2009-05-28T00:00:00Z

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26249335

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19478140

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