Voltage-sensitive calcium channels in spinal nociceptive processing: blockade of N- and P-type channels inhibits formalin-induced nociception.
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Ziconotide: a review of its pharmacology and use in the treatment of painIonotropic glutamate receptors and voltage-gated Ca²⁺ channels in long-term potentiation of spinal dorsal horn synapses and pain hypersensitivityAntidepressants as analgesics: an overview of central and peripheral mechanisms of action.Suppression of inflammatory and neuropathic pain symptoms in mice lacking the N-type Ca2+ channel.Targeting chronic and neuropathic pain: the N-type calcium channel comes of age.Insensitivity to pain induced by a potent selective closed-state Nav1.7 inhibitor.Systemic effects induced by intralesional injection of ω-conotoxin MVIIC after spinal cord injury in rats.The spinal biology in humans and animals of pain states generated by persistent small afferent inputN-type calcium current, Cav2.2, is enhanced in small-diameter sensory neurons isolated from Nf1+/- mice.Sigma-1 receptor antagonism restores injury-induced decrease of voltage-gated Ca2+ current in sensory neuronsSelective antinociceptive effects of a combination of the N-methyl-D-aspartate receptor peptide antagonist [Ser(1)]histogranin and morphine in rat models of painSuppression of inflammatory and neuropathic pain by uncoupling CRMP-2 from the presynaptic Ca²⁺ channel complexFurther insights into the antinociceptive potential of a peptide disrupting the N-type calcium channel-CRMP-2 signaling complex.The contribution of NMDA receptor activation to spinal c-Fos expression in a model of inflammatory pain.Regulation of spinal substance p release by intrathecal calcium channel blockade.Role of decreased sensory neuron membrane calcium currents in the genesis of neuropathic pain.Inhibition of transmitter release and attenuation of anti-retroviral-associated and tibial nerve injury-related painful peripheral neuropathy by novel synthetic Ca2+ channel peptides.Delivery of ziconotide to cerebrospinal fluid via intranasal pathway for the treatment of chronic pain.Release of glutamate and CGRP from trigeminal ganglion neurons: Role of calcium channels and 5-HT1 receptor signalingSuppression of pain-related behavior in two distinct rodent models of peripheral neuropathy by a homopolyarginine-conjugated CRMP2 peptide.Role of voltage gated Ca2+ channels in rat visceral hypersensitivity change induced by 2,4,6-trinitrobenzene sulfonic acidAcid-sensing ion channel 3 deficiency increases inflammation but decreases pain behavior in murine arthritis.Effects of general anesthetics on substance P release and c-Fos expression in the spinal dorsal hornEffects of intrathecal SNC80, a delta receptor ligand, on nociceptive threshold and dorsal horn substance p release.Pharmacological Inhibition of Voltage-gated Ca(2+) Channels for Chronic Pain Relief.Intrathecal P/Q- and R-type calcium channel blockade of spinal substance P release and c-Fos expression.Targeting voltage-gated calcium channels: developments in peptide and small-molecule inhibitors for the treatment of neuropathic pain.Omega-conotoxins as experimental tools and therapeutics in pain management.Gabapentin in acute postoperative pain managementCurrent and Future Issues in the Development of Spinal Agents for the Management of Pain.Sustained relief of ongoing experimental neuropathic pain by a CRMP2 peptide aptamer with low abuse potential.L-type calcium channel blockers, morphine and pain: Newer insightsEffect of Verapamil as an Adjuvant to Levobupivacaine in Supraclavicular Brachial Plexus Block.Antinociceptive effects of the marine snail peptides conantokin-G and conotoxin MVIIA alone and in combination in rat models of pain.Differential effects of voltage-gated calcium channel blockers on calcium channel alpha-2-delta-1 subunit protein-mediated nociceptionPharmacokinetic analysis of ziconotide (SNX-111), an intrathecal N-type calcium channel blocking analgesic, delivered by bolus and infusion in the dog.Algogen-specific pain processing in mouse spinal cord: differential involvement of voltage-dependent Ca(2+) channels in synaptic transmission.Painful peripheral nerve injury decreases calcium current in axotomized sensory neurons.ω-Conotoxins GVIA, MVIIA and CVID: SAR and Clinical Potential.Conotoxins: Therapeutic Potential and Application.
P2860
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P2860
Voltage-sensitive calcium channels in spinal nociceptive processing: blockade of N- and P-type channels inhibits formalin-induced nociception.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年学术文章
@wuu
1994年学术文章
@zh
1994年学术文章
@zh-cn
1994年学术文章
@zh-hans
1994年学术文章
@zh-my
1994年学术文章
@zh-sg
1994年學術文章
@yue
1994年學術文章
@zh-hant
name
Voltage-sensitive calcium chan ...... formalin-induced nociception.
@en
Voltage-sensitive calcium chan ...... formalin-induced nociception.
@nl
type
label
Voltage-sensitive calcium chan ...... formalin-induced nociception.
@en
Voltage-sensitive calcium chan ...... formalin-induced nociception.
@nl
prefLabel
Voltage-sensitive calcium chan ...... formalin-induced nociception.
@en
Voltage-sensitive calcium chan ...... formalin-induced nociception.
@nl
P1476
Voltage-sensitive calcium chan ...... s formalin-induced nociception
@en
P2093
A B Malmberg
P304
P356
10.1523/JNEUROSCI.14-08-04882.1994
P407
P577
1994-08-01T00:00:00Z