Post-ischaemic silencing of p66Shc reduces ischaemia/reperfusion brain injury and its expression correlates to clinical outcome in stroke.
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Mitochondrial Oxidative Stress, Mitochondrial DNA Damage and Their Role in Age-Related Vascular DysfunctionMitochondrial Metabolism in Aging HeartMolecular mechanism of endothelial and vascular aging: implications for cardiovascular disease.New aspects of p66Shc in ischaemia reperfusion injury and other cardiovascular diseases.Ischemia/Reperfusion.Lack of Contribution of p66shc and Its Mitochondrial Translocation to Ischemia-Reperfusion Injury and Cardioprotection by Ischemic Preconditioning.Shooting vascular oxidative stress: new hopes for stroke patients?p66Shc gene expression in peripheral blood mononuclear cells and progression of diabetic complications.
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P2860
Post-ischaemic silencing of p66Shc reduces ischaemia/reperfusion brain injury and its expression correlates to clinical outcome in stroke.
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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name
Post-ischaemic silencing of p6 ...... to clinical outcome in stroke.
@en
Post-ischaemic silencing of p6 ...... to clinical outcome in stroke.
@nl
type
label
Post-ischaemic silencing of p6 ...... to clinical outcome in stroke.
@en
Post-ischaemic silencing of p6 ...... to clinical outcome in stroke.
@nl
prefLabel
Post-ischaemic silencing of p6 ...... to clinical outcome in stroke.
@en
Post-ischaemic silencing of p6 ...... to clinical outcome in stroke.
@nl
P2093
P2860
P50
P356
P1476
Post-ischaemic silencing of p6 ...... to clinical outcome in stroke.
@en
P2093
A Akhmedov
A Semerano
G Giacalone
M Glanzmann
M R Kaufmann
P2860
P304
P356
10.1093/EURHEARTJ/EHV140
P50
P577
2015-04-22T00:00:00Z