Lung myofibroblasts as targets of salmeterol and fluticasone propionate: inhibition of alpha-SMA and NF-kappaB.
about
Regulation of myofibroblast differentiation by poly(ADP-ribose) polymerase 1β-agonists selectively modulate proinflammatory gene expression in skeletal muscle cells via non-canonical nuclear crosstalk mechanisms.Combined Beta-agonists and corticosteroids do not inhibit extracellular matrix protein production in vitroAnti-Inflammatory Effects of β2-Receptor Agonists Salbutamol and Terbutaline Are Mediated by MKP-1.Section 3. A discussion of flexible dosing and patient-centered therapy: highlights of the asthma summit 2009: beyond the guidelines.Tulobuterol inhibits rhinovirus infection in primary cultures of human tracheal epithelial cells.Can β2-adrenoceptor agonists, anticholinergic drugs, and theophylline contribute to the control of pulmonary inflammation and emphysema in COPD?The β2-subtype of adrenoceptors mediates inhibition of pro-fibrotic events in human lung fibroblasts.Enhancement of inflammatory mediator release by beta(2)-adrenoceptor agonists in airway epithelial cells is reversed by glucocorticoid action.Asthma drugs counter-regulate interleukin-8 release stimulated by sodium sulfite in an A549 cell line.Anti-proliferative and anti-remodelling effect of beclomethasone dipropionate, formoterol and salbutamol alone or in combination in primary human bronchial fibroblasts.Synergistic effects of fluticasone propionate and salmeterol on inhibiting rhinovirus-induced epithelial production of remodelling-associated growth factors.MiRNA-21 promotes fibrosis in orbital fibroblasts from thyroid-associated ophthalmopathyMechanisms for lung function impairment and airway hyperresponsiveness following chronic hypoxia in rats.Precapillary pulmonary hypertension leads to reversible bronchial hyperreactivity in rats.Reduced suppressive effect of β2-adrenoceptor agonist on fibrocyte function in severe asthma.Molecular changes during TGFβ-mediated lung fibroblast-myofibroblast differentiation: implication for glucocorticoid resistance.Stress-induced epinephrine levels compromise murine dermal fibroblast activity through β-adrenoceptors.
P2860
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P2860
Lung myofibroblasts as targets of salmeterol and fluticasone propionate: inhibition of alpha-SMA and NF-kappaB.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Lung myofibroblasts as targets ...... on of alpha-SMA and NF-kappaB.
@en
Lung myofibroblasts as targets ...... on of alpha-SMA and NF-kappaB.
@nl
type
label
Lung myofibroblasts as targets ...... on of alpha-SMA and NF-kappaB.
@en
Lung myofibroblasts as targets ...... on of alpha-SMA and NF-kappaB.
@nl
prefLabel
Lung myofibroblasts as targets ...... on of alpha-SMA and NF-kappaB.
@en
Lung myofibroblasts as targets ...... on of alpha-SMA and NF-kappaB.
@nl
P2093
P2860
P356
P1476
Lung myofibroblasts as targets ...... ion of alpha-SMA and NF-kappaB
@en
P2093
Bruno Azzarone
Francesca Cagnoni
Giulio Gabbiani
Massimo Giuliani
Renato Testi
Soria Baouz
Susanna Olsson
P2860
P304
P356
10.1093/INTIMM/DXH325
P577
2005-10-06T00:00:00Z