Contribution of TNF-alpha converting enzyme and proteinase-3 to TNF-alpha processing in human alveolar macrophages. - Wikidata - wikimedia - TriplyDB

Contribution of TNF-alpha converting enzyme and proteinase-3 to TNF-alpha processing in human alveolar macrophages.

Contribution of TNF-alpha converting enzyme and proteinase-3 to TNF-alpha processing in human alveolar macrophages.

http://www.wikidata.org/entity/Q46740264

about

Serglycin is the major secreted proteoglycan in macrophages and has a role in the regulation of macrophage tumor necrosis factor-alpha secretion in response to lipopolysaccharide Signaling pathways involved in LPS induced TNFalpha production in human adipocytes.Studies on the role of acid sphingomyelinase and ceramide in the regulation of tumor necrosis factor alpha (TNFalpha)-converting enzyme activity and TNFalpha secretion in macrophages.Investigation of the role of TNF-α converting enzyme (TACE) in the inhibition of cell surface and soluble TNF-α production by acute ethanol exposure Leukocyte cell surface proteinases: regulation of expression, functions, and mechanisms of surface localization.Neutrophil serine proteases fine-tune the inflammatory response Noncoding RNAs and LRRFIP1 regulate TNF expression.Importance of reverse signaling of the TNF superfamily in immune regulation.Reactive oxygen species and p38 mitogen-activated protein kinase mediate tumor necrosis factor α-converting enzyme (TACE/ADAM-17) activation in primary human monocytes.Membrane versus soluble isoforms of TNF-α exert opposing effects on tumor growth and survival of tumor-associated myeloid cells.Tumour necrosis factor-alpha processing in interstitial lung disease: a potential role for exogenous proteinase-3.Systems Biology Reveals Cigarette Smoke-Induced Concentration-Dependent Direct and Indirect Mechanisms That Promote Monocyte-Endothelial Cell Adhesion.Inflammatory cytokine production in tumor cells upon chemotherapy drug exposure or upon selection for drug resistance Lung endothelial ADAM17 regulates the acute inflammatory response to lipopolysaccharide Anti-inflammatory effect of palmitoylethanolamide on human adipocytes.

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Contribution of TNF-alpha converting enzyme and proteinase-3 to TNF-alpha processing in human alveolar macrophages.

http://www.wikidata.org/entity/Q46740264

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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2005年學術文章

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name

Contribution of TNF-alpha conv ...... in human alveolar macrophages.

@en

Contribution of TNF-alpha conv ...... in human alveolar macrophages.

@nl

type

label

Contribution of TNF-alpha conv ...... in human alveolar macrophages.

@en

Contribution of TNF-alpha conv ...... in human alveolar macrophages.

@nl

prefLabel

Contribution of TNF-alpha conv ...... in human alveolar macrophages.

@en

Contribution of TNF-alpha conv ...... in human alveolar macrophages.

@nl

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RCMB.2005-0087OC

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American Journal of Respiratory Cell and Molecular Biology

P1476

Contribution of TNF-alpha conv ...... in human alveolar macrophages.

@en

P2093

Ann B Millar

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Hayley A Whittington

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Kay M Uppington

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Lynne Armstrong

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Sofia I H Godinho

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P2860

Proteinase-3 as the major autoantigen of c-ANCA is strongly expressed in lung tissue of patients with Wegener's granulomatosis.

A metalloproteinase disintegrin that releases tumour-necrosis factor-alpha from cells

Evidence for a role of a tumor necrosis factor-alpha (TNF-alpha)-converting enzyme-like protease in shedding of TRANCE, a TNF family member involved in osteoclastogenesis and dendritic cell survival

The TNF and TNF receptor superfamilies: integrating mammalian biology

Proteinase 3. A distinct human polymorphonuclear leukocyte proteinase that produces emphysema in hamsters

Constitutive shedding of the amyloid precursor protein ectodomain is up-regulated by tumour necrosis factor-alpha converting enzyme

Expression of a tumor necrosis factor-alpha transgene in murine lung causes lymphocytic and fibrosing alveolitis. A mouse model of progressive pulmonary fibrosis

Tumor necrosis factor soluble receptors circulate during experimental and clinical inflammation and can protect against excessive tumor necrosis factor alpha in vitro and in vivo

Converting enzyme-independent release of tumor necrosis factor alpha and IL-1beta from a stimulated human monocytic cell line in the presence of activated neutrophils or purified proteinase 3

Antineutrophil cytoplasmic antibodies induce monocyte IL-8 release. Role of surface proteinase-3, alpha1-antitrypsin, and Fcgamma receptors.

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219-225

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scholarly article

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10.1165/RCMB.2005-0087OC

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2

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34

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2005-10-06T00:00:00Z

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16210695

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