Motility allows S. Typhimurium to benefit from the mucosal defence. - Wikidata - wikimedia - TriplyDB

Motility allows S. Typhimurium to benefit from the mucosal defence.

Motility allows S. Typhimurium to benefit from the mucosal defence.

http://www.wikidata.org/entity/Q46778188

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Regulatory principles governing Salmonella and Yersinia virulence Non-genetic diversity shapes infectious capacity and host resistance The role of motility and chemotaxis in the bacterial colonization of protected surfaces.Salmonella transiently reside in luminal neutrophils in the inflamed gut An NK Cell Perforin Response Elicited via IL-18 Controls Mucosal Inflammation Kinetics during Salmonella Gut Infection A simple screen to identify promoters conferring high levels of phenotypic noise The microbiota mediates pathogen clearance from the gut lumen after non-typhoidal Salmonella diarrhea Granulocytes impose a tight bottleneck upon the gut luminal pathogen population during Salmonella typhimurium colitis Infiltration of Matrix-Non-producers Weakens the Salmonella Biofilm and Impairs Its Antimicrobial Tolerance and Pathogenicity Stromal IFN-γR-signaling modulates goblet cell function during Salmonella Typhimurium infection Salmonella uses energy taxis to benefit from intestinal inflammation Near surface swimming of Salmonella Typhimurium explains target-site selection and cooperative invasion The cost of virulence: retarded growth of Salmonella Typhimurium cells expressing type III secretion system 1 Salmonella-induced mucosal lectin RegIIIβ kills competing gut microbiota Review on the effects of potential prebiotics on controlling intestinal enteropathogens Salmonella and Escherichia coli in pig production.Molecular insights into farm animal and zoonotic Salmonella infections.Motility is required for the competitive fitness of entomopathogenic Photorhabdus luminescens during insect infection.Muc2 protects against lethal infectious colitis by disassociating pathogenic and commensal bacteria from the colonic mucosa.QseC mediates Salmonella enterica serovar typhimurium virulence in vitro and in vivo.Two atypical enteropathogenic Escherichia coli strains induce the production of secreted and membrane-bound mucins to benefit their own growth at the apical surface of human mucin-secreting intestinal HT29-MTX cells Th17 cytokines and the gut mucosal barrier Salmonella enterica serovar Enteritidis tatB and tatC mutants are impaired in Caco-2 cell invasion in vitro and show reduced systemic spread in chickens.The blessings and curses of intestinal inflammation.Complex c-di-GMP signaling networks mediate transition between virulence properties and biofilm formation in Salmonella enterica serovar Typhimurium.Differential phenotypic diversity among epidemic-spanning Salmonella enterica serovar enteritidis isolates from humans or animals.Salmonella enterica serovar typhimurium colonizing the lumen of the chicken intestine grows slowly and upregulates a unique set of virulence and metabolism genes.A complex network of small non-coding RNAs regulate motility in Escherichia coli.Salmonella enterica serovar Typhimurium skills to succeed in the host: virulence and regulation.Colonization resistance and microbial ecophysiology: using gnotobiotic mouse models and single-cell technology to explore the intestinal jungle.rpoS-Regulated core genes involved in the competitive fitness of Salmonella enterica Serovar Kentucky in the intestines of chickens.Innate immune detection of flagellin positively and negatively regulates salmonella infection.Interaction of Salmonella spp. with the Intestinal Microbiota The Enteric Two-Step: nutritional strategies of bacterial pathogens within the gut Anaerobic respiration of Escherichia coli in the mouse intestine.Naturally occurring motility-defective mutants of Salmonella enterica serovar Enteritidis isolated preferentially from nonhuman rather than human sources.The Intestinal Microbiota Influences Campylobacter jejuni Colonization and Extraintestinal Dissemination in Mice.Regulation of phenotypic heterogeneity permits Salmonella evasion of the host caspase-1 inflammatory response.Intestinal fucose as a mediator of host-microbe symbiosis.Commensal and Pathogenic Escherichia coli Metabolism in the Gut.Gut inflammation can boost horizontal gene transfer between pathogenic and commensal Enterobacteriaceae.

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P2860

Motility allows S. Typhimurium to benefit from the mucosal defence.

http://www.wikidata.org/entity/Q46778188

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年學術文章

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2008年學術文章

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name

Motility allows S. Typhimurium to benefit from the mucosal defence.

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Motility allows S. Typhimurium to benefit from the mucosal defence.

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type

label

Motility allows S. Typhimurium to benefit from the mucosal defence.

@en

Motility allows S. Typhimurium to benefit from the mucosal defence.

@nl

altLabel

Motility allows S. Typhimurium to benefit from the mucosal defence

@en

prefLabel

Motility allows S. Typhimurium to benefit from the mucosal defence.

@en

Motility allows S. Typhimurium to benefit from the mucosal defence.

@nl

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J.1462-5822.2008.01118.X

P1433

Cellular Microbiology

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Motility allows S. Typhimurium to benefit from the mucosal defence.

@en

P2093

Bärbel Stecher

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Lea Haberli

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Manja Barthel

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Marcus Kremer

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Markus C Schlumberger

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Wolfgang Rabsch

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P2860

Salmonella enterica serovar typhimurium exploits inflammation to compete with the intestinal microbiota

Genomic and metabolic studies of the impact of probiotics on a model gut symbiont and host

Disruption-induced mucus secretion: repair and protection

One-step inactivation of chromosomal genes in Escherichia coli K-12 using PCR products

Toll-like receptor 5 recognizes a conserved site on flagellin required for protofilament formation and bacterial motility

Honor thy symbionts

Evasion of Toll-like receptor 5 by flagellated bacteria.

Toll-like receptors in innate immunity

Making sense of it all: bacterial chemotaxis

Targeted disruption of the MyD88 gene results in loss of IL-1- and IL-18-mediated function

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1166-1180

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scholarly article

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10.1111/J.1462-5822.2008.01118.X

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5

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10

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Wolf-Dietrich Hardt

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5611345

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18241212

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