Motility allows S. Typhimurium to benefit from the mucosal defence.
about
Regulatory principles governing Salmonella and Yersinia virulenceNon-genetic diversity shapes infectious capacity and host resistanceThe role of motility and chemotaxis in the bacterial colonization of protected surfaces.Salmonella transiently reside in luminal neutrophils in the inflamed gutAn NK Cell Perforin Response Elicited via IL-18 Controls Mucosal Inflammation Kinetics during Salmonella Gut InfectionA simple screen to identify promoters conferring high levels of phenotypic noiseThe microbiota mediates pathogen clearance from the gut lumen after non-typhoidal Salmonella diarrheaGranulocytes impose a tight bottleneck upon the gut luminal pathogen population during Salmonella typhimurium colitisInfiltration of Matrix-Non-producers Weakens the Salmonella Biofilm and Impairs Its Antimicrobial Tolerance and PathogenicityStromal IFN-γR-signaling modulates goblet cell function during Salmonella Typhimurium infectionSalmonella uses energy taxis to benefit from intestinal inflammationNear surface swimming of Salmonella Typhimurium explains target-site selection and cooperative invasionThe cost of virulence: retarded growth of Salmonella Typhimurium cells expressing type III secretion system 1Salmonella-induced mucosal lectin RegIIIβ kills competing gut microbiotaReview on the effects of potential prebiotics on controlling intestinal enteropathogens Salmonella and Escherichia coli in pig production.Molecular insights into farm animal and zoonotic Salmonella infections.Motility is required for the competitive fitness of entomopathogenic Photorhabdus luminescens during insect infection.Muc2 protects against lethal infectious colitis by disassociating pathogenic and commensal bacteria from the colonic mucosa.QseC mediates Salmonella enterica serovar typhimurium virulence in vitro and in vivo.Two atypical enteropathogenic Escherichia coli strains induce the production of secreted and membrane-bound mucins to benefit their own growth at the apical surface of human mucin-secreting intestinal HT29-MTX cellsTh17 cytokines and the gut mucosal barrierSalmonella enterica serovar Enteritidis tatB and tatC mutants are impaired in Caco-2 cell invasion in vitro and show reduced systemic spread in chickens.The blessings and curses of intestinal inflammation.Complex c-di-GMP signaling networks mediate transition between virulence properties and biofilm formation in Salmonella enterica serovar Typhimurium.Differential phenotypic diversity among epidemic-spanning Salmonella enterica serovar enteritidis isolates from humans or animals.Salmonella enterica serovar typhimurium colonizing the lumen of the chicken intestine grows slowly and upregulates a unique set of virulence and metabolism genes.A complex network of small non-coding RNAs regulate motility in Escherichia coli.Salmonella enterica serovar Typhimurium skills to succeed in the host: virulence and regulation.Colonization resistance and microbial ecophysiology: using gnotobiotic mouse models and single-cell technology to explore the intestinal jungle.rpoS-Regulated core genes involved in the competitive fitness of Salmonella enterica Serovar Kentucky in the intestines of chickens.Innate immune detection of flagellin positively and negatively regulates salmonella infection.Interaction of Salmonella spp. with the Intestinal MicrobiotaThe Enteric Two-Step: nutritional strategies of bacterial pathogens within the gutAnaerobic respiration of Escherichia coli in the mouse intestine.Naturally occurring motility-defective mutants of Salmonella enterica serovar Enteritidis isolated preferentially from nonhuman rather than human sources.The Intestinal Microbiota Influences Campylobacter jejuni Colonization and Extraintestinal Dissemination in Mice.Regulation of phenotypic heterogeneity permits Salmonella evasion of the host caspase-1 inflammatory response.Intestinal fucose as a mediator of host-microbe symbiosis.Commensal and Pathogenic Escherichia coli Metabolism in the Gut.Gut inflammation can boost horizontal gene transfer between pathogenic and commensal Enterobacteriaceae.
P2860
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P2860
Motility allows S. Typhimurium to benefit from the mucosal defence.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh-hant
name
Motility allows S. Typhimurium to benefit from the mucosal defence.
@en
Motility allows S. Typhimurium to benefit from the mucosal defence.
@nl
type
label
Motility allows S. Typhimurium to benefit from the mucosal defence.
@en
Motility allows S. Typhimurium to benefit from the mucosal defence.
@nl
altLabel
Motility allows S. Typhimurium to benefit from the mucosal defence
@en
prefLabel
Motility allows S. Typhimurium to benefit from the mucosal defence.
@en
Motility allows S. Typhimurium to benefit from the mucosal defence.
@nl
P2093
P2860
P1476
Motility allows S. Typhimurium to benefit from the mucosal defence.
@en
P2093
Bärbel Stecher
Lea Haberli
Manja Barthel
Marcus Kremer
Markus C Schlumberger
Wolfgang Rabsch
P2860
P304
P356
10.1111/J.1462-5822.2008.01118.X
P577
2008-01-30T00:00:00Z