Antithrombin inhibits lipopolysaccharide-induced tumor necrosis factor-alpha production by monocytes in vitro through inhibition of Egr-1 expression.

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Antithrombin attenuates myocardial dysfunction and reverses systemic fluid accumulation following burn and smoke inhalation injury: a randomized, controlled, experimental study.Point Mutation of a Non-Elastase-Binding Site in Human α1-Antitrypsin Alters Its Anti-Inflammatory Properties.

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Antithrombin inhibits lipopolysaccharide-induced tumor necrosis factor-alpha production by monocytes in vitro through inhibition of Egr-1 expression.

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http://www.wikidata.org/entity/Q46844317

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2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

@zh-my

2007年学术文章

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2007年學術文章

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2007年學術文章

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2007年學術文章

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name

Antithrombin inhibits lipopoly ...... nhibition of Egr-1 expression.

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Antithrombin inhibits lipopoly ...... nhibition of Egr-1 expression.

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type

Item

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Antithrombin inhibits lipopoly ...... nhibition of Egr-1 expression.

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Antithrombin inhibits lipopoly ...... nhibition of Egr-1 expression.

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prefLabel

Antithrombin inhibits lipopoly ...... nhibition of Egr-1 expression.

@en

Antithrombin inhibits lipopoly ...... nhibition of Egr-1 expression.

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P1476

Antithrombin inhibits lipopoly ...... nhibition of Egr-1 expression.

@en

P2093

H Katsuya

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H Komura

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K Okajima

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M Arai

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M Uchiba

http://www.w3.org/2001/XMLSchema#string

N Harada

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Y Mizuochi

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P2860

Pathways of Egr-1-mediated gene transcription in vascular biology

Gabexate mesilate, a synthetic protease inhibitor, inhibits lipopolysaccharide-induced tumor necrosis factor-alpha production by inhibiting activation of both nuclear factor-kappaB and activator protein-1 in human monocytes

Lipopolysaccharide activation of the MEK-ERK1/2 pathway in human monocytic cells mediates tissue factor and tumor necrosis factor alpha expression by inducing Elk-1 phosphorylation and Egr-1 expression

LPS induction of gene expression in human monocytes.

FLI1 and EWS-FLI1 function as ternary complex factors and ELK1 and SAP1a function as ternary and quaternary complex factors on the Egr1 promoter serum response elements.

Activation of c-Jun N-terminal kinase in bacterial lipopolysaccharide-stimulated macrophages.

A ternary complex factor-dependent mechanism mediates induction of egr-1 through selective serum response elements following antigen receptor cross-linking in B lymphocytes.

Urea inducibility of egr-1 in murine inner medullary collecting duct cells is mediated by the serum response element and adjacent Ets motifs.

Biochemistry of heparin antithrombin interactions, and the physiologic role of this natural anticoagulant mechanism.

Cyclic AMP-dependent kinase regulates Raf-1 kinase mainly by phosphorylation of serine 259.

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499-507

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scholarly article

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10.1111/J.1538-7836.2007.02869.X

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245741255

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18088351

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P921

lipopolysaccharide

Antithrombin inhibits lipopolysaccharide-induced tumor necrosis factor-alpha production by monocytes in vitro through inhibition of Egr-1 expression.

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P2860

P2860

Antithrombin inhibits lipopolysaccharide-induced tumor necrosis factor-alpha production by monocytes in vitro through inhibition of Egr-1 expression.

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