Efficient execution of cell death in non-glycolytic cells requires the generation of ROS controlled by the activity of mitochondrial H+-ATP synthase. - Wikidata - wikimedia - TriplyDB

Efficient execution of cell death in non-glycolytic cells requires the generation of ROS controlled by the activity of mitochondrial H+-ATP synthase.

Efficient execution of cell death in non-glycolytic cells requires the generation of ROS controlled by the activity of mitochondrial H+-ATP synthase.

http://www.wikidata.org/entity/Q46856395

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Role of the c subunit of the FO ATP synthase in mitochondrial permeability transition The Dual Function of Reactive Oxygen/Nitrogen Species in Bioenergetics and Cell Death: The Role of ATP Synthase Biogenesis and dynamics of mitochondria during the cell cycle: significance of 3'UTRs.Reactive oxygen species, cellular redox systems, and apoptosis The mitochondrial protein Bak is pivotal for gliotoxin-induced apoptosis and a critical host factor of Aspergillus fumigatus virulence in mice Deregulation of mitochondrial ATPsyn-β in acute myeloid leukemia cells and with increased drug resistance Dihydroartemisinin (DHA) induces caspase-3-dependent apoptosis in human lung adenocarcinoma ASTC-a-1 cells Metabolic genes in cancer: their roles in tumor progression and clinical implications Regulation of the H+-ATP synthase by IF1: a role in mitohormesis.The bioenergetic signature of isogenic colon cancer cells predicts the cell death response to treatment with 3-bromopyruvate, iodoacetate or 5-fluorouracil A combined proteomics and metabolomics profiling of gastric cardia cancer reveals characteristic dysregulations in glucose metabolism Tyrosine kinase inhibition in leukemia induces an altered metabolic state sensitive to mitochondrial perturbations.Transient elevation of glycolysis confers radio-resistance by facilitating DNA repair in cells Fully automated software for quantitative measurements of mitochondrial morphology.Hypoxia-inducible factors: mediators of cancer progression and targets for cancer therapy.Expression, regulation and clinical relevance of the ATPase inhibitory factor 1 in human cancers.Mitochondria-mediated energy adaption in cancer: the H(+)-ATP synthase-geared switch of metabolism in human tumors.LDH-A inhibition, a therapeutic strategy for treatment of hereditary leiomyomatosis and renal cell cancer.Pleiotropic effects of the trichloroethylene-associated P81S VHL mutation on metabolism, apoptosis, and ATM-mediated DNA damage response.Regulation of mitochondrial respiratory chain biogenesis by estrogens/estrogen receptors and physiological, pathological and pharmacological implications.Bcl-2 family proteins as regulators of oxidative stress.Progesterone stimulates mitochondrial activity with subsequent inhibition of apoptosis in MCF-10A benign breast epithelial cells.Mitochondria and energetic depression in cell pathophysiology Metabolic reprogramming of the tumor.Hitting the Bull's-Eye in Metastatic Cancers-NSAIDs Elevate ROS in Mitochondria, Inducing Malignant Cell Death.In vivo inhibition of the mitochondrial H+-ATP synthase in neurons promotes metabolic preconditioning.Metformin rescues cell surface major histocompatibility complex class I (MHC-I) deficiency caused by oncogenic transformation.Apoptotic microtubule network organization and maintenance depend on high cellular ATP levels and energized mitochondria.Up-regulation of the ATPase inhibitory factor 1 (IF1) of the mitochondrial H+-ATP synthase in human tumors mediates the metabolic shift of cancer cells to a Warburg phenotype.Amplification of 8q21 in breast cancer is independent of MYC and associated with poor patient outcome.Increased mitochondrial DNA induces acquired docetaxel resistance in head and neck cancer cells.NQO-Induced DNA-Less Cell Formation Is Associated with Chromatin Protein Degradation and Dependent on A0A1-ATPase in Sulfolobus.Radiation response and regulation of apoptosis induced by a combination of TRAIL and CHX in cells lacking mitochondrial DNA: a role for NF-κB-STAT3-directed gene expression.Cancer abolishes the tissue type-specific differences in the phenotype of energetic metabolism.Selection of cancer cells with repressed mitochondria triggers colon cancer progression.A proteomic approach to elucidate the multiple targets of selenium-induced cell-growth inhibition in human lung cancer.Selective inhibition of beta-F1-ATPase mRNA translation in human tumours.Expression of oxidored nitro domain-containing protein 1(NOR1) impairs nasopharyngeal carcinoma cells adaptation to hypoxia and inhibits PDK1 expression.Characterization of mitochondrial bioenergetic functions between two forms of Leishmania donovani - a comparative analysis.ATP5b and β2-microglobulin are predictive markers for the prognosis of patients with gallbladder cancer.

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P2860

Efficient execution of cell death in non-glycolytic cells requires the generation of ROS controlled by the activity of mitochondrial H+-ATP synthase.

http://www.wikidata.org/entity/Q46856395

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2005 nî lūn-bûn

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Efficient execution of cell de ...... mitochondrial H+-ATP synthase.

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Efficient execution of cell de ...... mitochondrial H+-ATP synthase.

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Gema Santamaría

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José M Cuezva

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Marta Martínez-Diez

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P2860

Disruption of mitochondrial function during apoptosis is mediated by caspase cleavage of the p75 subunit of complex I of the electron transport chain

In self-defence: hexokinase promotes voltage-dependent anion channel closure and prevents mitochondria-mediated apoptotic cell death

BAD and glucokinase reside in a mitochondrial complex that integrates glycolysis and apoptosis

A systematic RNAi screen identifies a critical role for mitochondria in C. elegans longevity

Mitochondrial diseases in man and mouse

The role of dynamin-related protein 1, a mediator of mitochondrial fission, in apoptosis

The ATP synthase--a splendid molecular machine

Proteomic changes in renal cancer and co-ordinate demonstration of both the glycolytic and mitochondrial aspects of the Warburg effect.

Identification of tumor-associated proteins in oral tongue squamous cell carcinoma by proteomics.

Bafilomycins: a class of inhibitors of membrane ATPases from microorganisms, animal cells, and plant cells.

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Isabel Fabregat

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