Variants in the VCAM1 gene and risk for symptomatic stroke in sickle cell disease.
about
Genetic dissection and prognostic modeling of overt stroke in sickle cell anemiaGenomic approaches to identifying targets for treating β hemoglobinopathiesGenetic susceptibility to ischemic stroke.Sickle cell disease: a multigenic perspective of a single gene disorder.Sickle Cell Disease in the Post Genomic Era: A Monogenic Disease with a Polygenic Phenotype.Haplotype diversity across 100 candidate genes for inflammation, lipid metabolism, and blood pressure regulation in two populationsVariants associated with common disease are not unusually differentiated in frequency across populations.The effect of CYP2D6 polymorphisms on the response to pain treatment for pediatric sickle cell pain crisis.Genetic modifiers of sickle cell diseaseLevels of soluble endothelium-derived adhesion molecules in patients with sickle cell disease are associated with pulmonary hypertension, organ dysfunction, and mortality.Cerebrovascular disease associated with sickle cell pulmonary hypertension.Coagulation abnormalities of sickle cell disease: Relationship with clinical outcomes and the effect of disease modifying therapies.Effect of transfusion therapy on cerebral vasculopathy in children with sickle-cell anemia.The epidemiology, evaluation and treatment of stroke in adults with sickle cell disease.Advances in understanding the pathogenesis of cerebrovascular vasculopathy in sickle cell anaemia.Minireview: Genetic basis of heterogeneity and severity in sickle cell disease.Genetics of Sickle Cell-Associated Cardiovascular Disease: An Expert Review with Lessons Learned in Africa.Reticulocyte count is the most important predictor of acute cerebral ischemia and high-risk transcranial Doppler in a newborn cohort of 395 children with sickle cell anemia.Laboratory and Genetic Biomarkers Associated with Cerebral Blood Flow Velocity in Hemoglobin SC Disease.ICAM1 K469E and E-selectin S128R polymorphisms could predispose to increased autoantibody production and TSH suppression in Graves' disease.Genetic variation in CD36, HBA, NOS3 and VCAM1 is associated with chronic haemolysis level in sickle cell anaemia: a longitudinal study.
P2860
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P2860
Variants in the VCAM1 gene and risk for symptomatic stroke in sickle cell disease.
description
2002 nî lūn-bûn
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2002年の論文
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2002年学术文章
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2002年学术文章
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2002年学术文章
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2002年学术文章
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2002年学术文章
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2002年学术文章
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2002年學術文章
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2002年學術文章
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name
Variants in the VCAM1 gene and risk for symptomatic stroke in sickle cell disease.
@en
Variants in the VCAM1 gene and risk for symptomatic stroke in sickle cell disease.
@nl
type
label
Variants in the VCAM1 gene and risk for symptomatic stroke in sickle cell disease.
@en
Variants in the VCAM1 gene and risk for symptomatic stroke in sickle cell disease.
@nl
prefLabel
Variants in the VCAM1 gene and risk for symptomatic stroke in sickle cell disease.
@en
Variants in the VCAM1 gene and risk for symptomatic stroke in sickle cell disease.
@nl
P2093
P50
P1433
P1476
Variants in the VCAM1 gene and risk for symptomatic stroke in sickle cell disease.
@en
P2093
Delia C Tang
Graham R Serjeant
Griffin P Rodgers
Seth I Heller
Susan F Leitman
P304
P356
10.1182/BLOOD-2001-12-0306
P407
P577
2002-08-15T00:00:00Z