Induction of premature senescence in cardiomyocytes by doxorubicin as a novel mechanism of myocardial damage.
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Juvenile exposure to anthracyclines impairs cardiac progenitor cell function and vascularization resulting in greater susceptibility to stress-induced myocardial injury in adult miceCellular senescence in aging and age-related disease: from mechanisms to therapyMitochondrial Metabolism in Aging HeartRegulation of signal transduction by reactive oxygen species in the cardiovascular systemInhibition of doxorubicin-induced senescence by PPARδ activation agonists in cardiac muscle cells: cooperation between PPARδ and Bcl6Doxorubicin induces senescence or apoptosis in rat neonatal cardiomyocytes by regulating the expression levels of the telomere binding factors 1 and 2.Doxorubicin as a molecular nanotheranostic agent: effect of doxorubicin encapsulation in micelles or nanoemulsions on the ultrasound-mediated intracellular delivery and nuclear trafficking.Cardio-oncology in targeting the HER receptor family: the puzzle of different cardiotoxicities of HER2 inhibitors.Cardiomyocyte death in doxorubicin-induced cardiotoxicityp38 MAPK and JNK antagonistically control senescence and cytoplasmic p16INK4A expression in doxorubicin-treated endothelial progenitor cellsActive Wnt signaling in response to cardiac injury.Protection from doxorubicin-induced cardiomyopathy using the modified anthracycline N-benzyladriamycin-14-valerate (AD 198).Adenylyl cyclase 6 improves calcium uptake and left ventricular function in aged heartsApoptosis in Anthracycline CardiomyopathyDownregulation of doxorubicin-induced myocardial apoptosis accompanies postnatal heart maturation.Ginkgo biloba extract EGB761 protects against aging-associated diastolic dysfunction in cardiomyocytes of D-galactose-induced aging rat.Tumor-targeted and pH-controlled delivery of doxorubicin using gold nanorods for lung cancer therapy.Pummelo protects Doxorubicin-induced cardiac cell death by reducing oxidative stress, modifying glutathione transferase expression, and preventing cellular senescenceMacrophage Migration Inhibitory Factor (MIF) Deficiency Exacerbates Aging-Induced Cardiac Remodeling and Dysfunction Despite Improved Inflammation: Role of Autophagy Regulation.Testosterone Antagonizes Doxorubicin-Induced Senescence of Cardiomyocytes.Evaluating Health Span in Preclinical Models of Aging and Disease: Guidelines, Challenges, and Opportunities for Geroscience.A comparison of replicative senescence and doxorubicin-induced premature senescence of vascular smooth muscle cells isolated from human aorta.Mechanisms and management of doxorubicin cardiotoxicity.Cardiac telomere length in heart development, function, and disease.Increasing role of the cancer chemotherapeutic doxorubicin in cellular metabolism.Modulation of induced cytotoxicity of doxorubicin by using apoferritin and liposomal cages.Oxidative DNA damage causes premature senescence in mouse embryonic fibroblasts deficient for Krüppel-like factor 4.Autophagy and mitophagy in the context of doxorubicin-induced cardiotoxicity.Senescence as a novel mechanism involved in β-adrenergic receptor mediated cardiac hypertrophy.Deficiency of senescence marker protein 30 exacerbates angiotensin II-induced cardiac remodelling.Oxidative Stress and Cellular Response to Doxorubicin: A Common Factor in the Complex Milieu of Anthracycline Cardiotoxicity.Non-canonical roles for caveolin in regulation of membrane repair and mitochondria: implications for stress adaptation with age.Therapeutic doses of doxorubicin induce premature senescence of human mesenchymal stem cells derived from menstrual blood, bone marrow and adipose tissue.Effect of valsartan on cardiac senescence and apoptosis in a rat model of cardiotoxicity.Short-Duration Swimming Exercise after Myocardial Infarction Attenuates Cardiac Dysfunction and Regulates Mitochondrial Quality Control in Aged Mice.Diethyl Blechnic, a Novel Natural Product Isolated from Bunge, Inhibits Doxorubicin-Induced Apoptosis by Inhibiting ROS and Activating JNK1/2
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P2860
Induction of premature senescence in cardiomyocytes by doxorubicin as a novel mechanism of myocardial damage.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
Induction of premature senesce ...... echanism of myocardial damage.
@en
Induction of premature senesce ...... echanism of myocardial damage.
@nl
type
label
Induction of premature senesce ...... echanism of myocardial damage.
@en
Induction of premature senesce ...... echanism of myocardial damage.
@nl
prefLabel
Induction of premature senesce ...... echanism of myocardial damage.
@en
Induction of premature senesce ...... echanism of myocardial damage.
@nl
P2093
P2860
P1433
P1476
Induction of premature senesce ...... echanism of myocardial damage.
@en
P2093
Hiroshi Ito
Kenzo Hirao
Mitsuaki Isobe
Susumu Adachi
Yasuhiro Maejima
P2860
P304
P356
10.1111/J.1474-9726.2007.00358.X
P577
2007-11-21T00:00:00Z