Postconditioning with sevoflurane protects against focal cerebral ischemia and reperfusion injury involving mitochondrial ATP-dependent potassium channel and mitochondrial permeability transition pore.
about
Sevoflurane Postconditioning-Induced Anti-Inflammation via Inhibition of the Toll-Like Receptor-4/Nuclear Factor Kappa B Pathway Contributes to Neuroprotection against Transient Global Cerebral Ischemia in Rats.Toxicity of inhaled agents after prolonged administration.Sevoflurane prevents stroke-induced depressive and anxiety behaviors by promoting cannabinoid receptor subtype I-dependent interaction between β-arrestin 2 and extracellular signal-regulated kinases 1/2 in the rat hippocampus.Mechanisms of the beneficial effect of sevoflurane in liver ischemia/reperfusion injury.Effects of sevoflurane on NF-кB and TNF-α expression in renal ischemia-reperfusion diabetic rats.
P2860
Postconditioning with sevoflurane protects against focal cerebral ischemia and reperfusion injury involving mitochondrial ATP-dependent potassium channel and mitochondrial permeability transition pore.
description
2014 nî lūn-bûn
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2014年の論文
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name
Postconditioning with sevoflur ...... permeability transition pore.
@en
Postconditioning with sevoflur ...... permeability transition pore.
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type
label
Postconditioning with sevoflur ...... permeability transition pore.
@en
Postconditioning with sevoflur ...... permeability transition pore.
@nl
prefLabel
Postconditioning with sevoflur ...... permeability transition pore.
@en
Postconditioning with sevoflur ...... permeability transition pore.
@nl
P2093
P2860
P1476
Postconditioning with sevoflur ...... permeability transition pore.
@en
P2093
Hui-Fang Wu
Jun-Kuan Wang
Xian-Zhi Liu
P2860
P356
10.1179/1743132814Y.0000000410
P577
2014-06-25T00:00:00Z