Exogenous hydrogen sulfide prevents cardiomyocyte apoptosis from cardiac hypertrophy induced by isoproterenol. - Wikidata - wikimedia - TriplyDB

Exogenous hydrogen sulfide prevents cardiomyocyte apoptosis from cardiac hypertrophy induced by isoproterenol.

Exogenous hydrogen sulfide prevents cardiomyocyte apoptosis from cardiac hypertrophy induced by isoproterenol.

http://www.wikidata.org/entity/Q46892353

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The Cardioprotective Effects of Hydrogen Sulfide in Heart Diseases: From Molecular Mechanisms to Therapeutic Potential Urotensin II Protects Cardiomyocytes from Apoptosis Induced by Oxidative Stress through the CSE/H2S Pathway Endogenous carbon monoxide downregulates hepatic cystathionine-γ-lyase in rats with liver cirrhosis Emerging role of hydrogen sulfide-microRNA crosstalk in cardiovascular diseases.Suppression of calcium‑sensing receptor ameliorates cardiac hypertrophy through inhibition of autophagy.Role of hydrogen sulfide in portal hypertension and esophagogastric junction vascular disease.Regulation of mitochondrial bioenergetic function by hydrogen sulfide. Part II. Pathophysiological and therapeutic aspects Hydrogen Sulfide Inhibits High-Salt Diet-Induced Myocardial Oxidative Stress and Myocardial Hypertrophy in Dahl Rats.Mitochondrial toxicity of cardiac drugs and its relevance to mitochondrial disorders.A sensitive and fast LC-MS/MS method for determination of β-receptor agonist JP-49b: application to a pharmacokinetic study in rats.Exogenous hydrogen sulfide alleviates high glucose-induced cardiotoxicity via inhibition of leptin signaling in H9c2 cells.Hydrosulfide attenuates acute myocardial ischemic injury through the glycogen synthase kinase-3β/β-catenin signaling pathway.Biochemical and histopathologic analysis of the effects of periodontitis on left ventricular heart tissues of rats.Boosting endogenous production of vasoprotective hydrogen sulfide via supplementation with taurine and N-acetylcysteine: a novel way to promote cardiovascular health.Canstatin inhibits isoproterenol-induced apoptosis through preserving mitochondrial morphology in differentiated H9c2 cardiomyoblasts.Hydrogen sulfide pretreatment improves mitochondrial function in myocardial hypertrophy via a SIRT3-dependent manner.Anti-Interleukin-22-Neutralizing Antibody Attenuates Angiotensin II-Induced Cardiac Hypertrophy in Mice.Protection of Luteolin-7-O-glucoside against apoptosis induced by hypoxia/reoxygenation through the MAPK pathways in H9c2 cells.Cyclovirobuxinum D alleviates cardiac hypertrophy in hyperthyroid rats by preventing apoptosis of cardiac cells and inhibiting the p38 mitogen-activated protein kinase signaling pathway.Hydrogen Sulfide (HS)-Releasing Compounds: Therapeutic Potential in Cardiovascular Diseases

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P2860

Exogenous hydrogen sulfide prevents cardiomyocyte apoptosis from cardiac hypertrophy induced by isoproterenol.

http://www.wikidata.org/entity/Q46892353

description

2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

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2013年學術文章

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name

Exogenous hydrogen sulfide pre ...... ophy induced by isoproterenol.

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Exogenous hydrogen sulfide pre ...... ophy induced by isoproterenol.

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Exogenous hydrogen sulfide pre ...... ophy induced by isoproterenol.

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Exogenous hydrogen sulfide pre ...... ophy induced by isoproterenol.

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Exogenous hydrogen sulfide pre ...... ophy induced by isoproterenol.

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Exogenous hydrogen sulfide pre ...... ophy induced by isoproterenol.

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S11010-013-1686-7

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Molecular and Cellular Biochemistry

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Exogenous hydrogen sulfide pre ...... ophy induced by isoproterenol.

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P2093

Changqing Xu

http://www.w3.org/2001/XMLSchema#string

Fan Yang

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Fanghao Lu

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Hulun Li

http://www.w3.org/2001/XMLSchema#string

Jun Xing

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Lina Wang

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Shiyun Dong

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Weihua Zhang

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Xinying Zhang

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Yajun Zhao

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P2860

Role of cystathionine γ-lyase/hydrogen sulfide pathway in cardiovascular disease: a novel therapeutic strategy?

The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology

Fluorescent Indicators for Imaging Nitric Oxide Production.

H2S ameliorates oxidative and proteolytic stresses and protects the heart against adverse remodeling in chronic heart failure

β-AR blockers suppresses ER stress in cardiac hypertrophy and heart failure.

NADPH oxidase 4 (Nox4) is a major source of oxidative stress in the failing heart.

Two's company, three's a crowd: can H2S be the third endogenous gaseous transmitter?

NAD(P)H oxidase 4 mediates transforming growth factor-beta1-induced differentiation of cardiac fibroblasts into myofibroblasts.

Hydrogen sulfide in biochemistry and medicine.

NADPH oxidase-derived reactive oxygen species in cardiac pathophysiology

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10.1007/S11010-013-1686-7

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1-2

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381

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