Reactive oxygen species contribute to Ca2+ signals produced by osmotic stress in mouse skeletal muscle fibres.
about
Hierarchical accumulation of RyR post-translational modifications drives disease progression in dystrophic cardiomyopathyInhibitory control over Ca(2+) sparks via mechanosensitive channels is disrupted in dystrophin deficient muscle but restored by mini-dystrophin expressionMinding the calcium store: Ryanodine receptor activation as a convergent mechanism of PCB toxicityDisrupted membrane structure and intracellular Ca²⁺ signaling in adult skeletal muscle with acute knockdown of Bin1.Hyperactive intracellular calcium signaling associated with localized mitochondrial defects in skeletal muscle of an animal model of amyotrophic lateral sclerosisMitochondrial redox potential during contraction in single intact muscle fibers.Assessment of calcium sparks in intact skeletal muscle fibersMitochondrial calcium uptake regulates rapid calcium transients in skeletal muscle during excitation-contraction (E-C) coupling.Differential impact of mitochondrial positioning on mitochondrial Ca(2+) uptake and Ca(2+) spark suppression in skeletal muscle.Reactive oxygen species in skeletal muscle signaling.NOX2-dependent ROS is required for HDAC5 nuclear efflux and contributes to HDAC4 nuclear efflux during intense repetitive activity of fast skeletal muscle fibersAbsence of Dystrophin Disrupts Skeletal Muscle Signaling: Roles of Ca2+, Reactive Oxygen Species, and Nitric Oxide in the Development of Muscular Dystrophy.Elevated extracellular glucose and uncontrolled type 1 diabetes enhance NFAT5 signaling and disrupt the transverse tubular network in mouse skeletal muscleAge-dependent uncoupling of mitochondria from Ca2⁺ release units in skeletal muscle.Cardiac phenotype of Duchenne Muscular Dystrophy: insights from cellular studiesDHPR activation underlies SR Ca2+ release induced by osmotic stress in isolated rat skeletal muscle fibersRole of Ca(2+) in injury-induced changes in sodium current in rat skeletal muscleMalformed mdx myofibers have normal cytoskeletal architecture yet altered EC coupling and stress-induced Ca2+ signaling.Sarcoplasmic reticulum-mitochondrial symbiosis: bidirectional signaling in skeletal muscle.Pathways of abnormal stress-induced Ca2+ influx into dystrophic mdx cardiomyocytesSarcoplasmic reticulum-mitochondrial through-space coupling in skeletal muscle.Dysferlin stabilizes stress-induced Ca2+ signaling in the transverse tubule membrane.X-ROS signaling in the heart and skeletal muscle: stretch-dependent local ROS regulates [Ca²⁺]i.Cardiac and respiratory dysfunction in Duchenne muscular dystrophy and the role of second messengers.Mechanical stretch-induced activation of ROS/RNS signaling in striated muscle.ROS and RNS signaling in skeletal muscle: critical signals and therapeutic targets.Hyperthermia, dehydration, and osmotic stress: unconventional sources of exercise-induced reactive oxygen species.Regulation of NADPH oxidases in skeletal muscle.Dystrophin Orchestrates the Epigenetic Profile of Muscle Cells Via miRNAsProof of concept: hypovolemic hyponatremia may precede and augment creatine kinase elevations during an ultramarathon.Detection of calcium sparks in intact and permeabilized skeletal muscle fibers.Redox modulation of global phosphatase activity and protein phosphorylation in intact skeletal muscle.Single KATP channel opening in response to stimulation of AMPA/kainate receptors is mediated by Na+ accumulation and submembrane ATP and ADP changes.Effects of N-acetylcysteine on isolated mouse skeletal muscle: contractile properties, temperature dependence, and metabolism.N-Acetylcysteine ameliorates skeletal muscle pathophysiology in mdx mice.Osmosensation in TRPV2 dominant negative expressing skeletal muscle fibres.Reactive oxygen species are related to ionic fluxes and volume decrease in apoptotic cerebellar granule neurons: role of NOX enzymes.Calcium-induced release of calcium in muscle: 50 years of work and the emerging consensus.Mitochondrial production of reactive oxygen species contributes to the β-adrenergic stimulation of mouse cardiomycytes.Osmolality Selectively Offsets the Impact of Hyperthermia on Mouse Skeletal Muscle
P2860
Q24306846-6A3B6324-2C85-4953-BF7E-F447913166EFQ27438151-EB95EA3E-F93E-438C-B3DA-734C94759EC0Q28393648-16118379-1C07-4D5F-A45D-F0C91429906CQ30010226-99A94404-40C1-4A74-A127-FA409B9E2322Q33581470-A7B723DD-89EC-4C80-9184-15113EACD452Q34462921-31D8E9E5-4FB7-4728-8B37-2CB4C827094DQ35123222-2A75CD56-8495-4A1F-8DD4-0DD371DB377AQ35213316-2E6E31B0-8AB1-4787-8CFD-53DA3903C5A1Q35543276-A7BFEC74-37F7-459B-86F0-04CD64CBCB4EQ35607438-2B6BB8B8-B339-411D-ACD9-F5EE24E794F4Q36175713-870C576E-E3AE-4E79-A631-242D7BFD7C74Q36422465-7429BA94-C461-4D93-B98D-AD457F409A5EQ36464964-8FB9CD00-836A-41AA-B0DB-E1006B673DEBQ36546356-D24600D3-05D8-4C09-BAE7-B2FE9367959DQ36736081-D2BE3158-85C7-4EBE-ABA5-22742A75D629Q37267643-95E7F733-12E9-4435-A8BE-AEB7FFE33193Q37297998-1174F622-CBFD-47C0-B843-B814585FC6F0Q37338902-177DBD3A-B829-4CE3-9752-2E74BDA46139Q37339681-71F3D6AA-30A4-47BB-BD81-5B049DF9A817Q37349387-1F0D1D1B-A44F-427E-9033-EE8A4524DEFCQ37356683-6C554697-E5D4-439B-989E-177FF63ABB81Q37409431-5C626C1E-776E-4184-B46F-28E7138BC8B9Q37633116-0B7F964E-92EF-4410-AD50-C1B5F986155AQ38103213-6FB21B66-1EEB-4275-8955-EEF8CABCF7D8Q38131000-BCE6CFFF-83B9-4535-8DC7-955AE5900091Q38217375-9A9E745F-1A21-4933-8D4F-C2979D928D0EQ38631670-07C396E9-927B-4B31-A843-91981D620FBFQ38835704-8967FEF8-6B6F-4E12-9D81-5BD1A95994B1Q39656924-04D1607F-BF83-4EDD-B380-2FA41F2A2A27Q40127014-E762BC5D-B203-426B-BAAC-0BD4F3433001Q40841956-040AAC1D-34A4-415F-B28D-749CA54680F6Q43258663-FB4A2819-DEFA-475E-BA16-7EFB3F952468Q43973832-EDF29C0D-EED6-4180-9617-CDE9971559B6Q46573928-A9844CC4-C7E7-4F24-A687-951789C08C2EQ46765634-64DF9494-7585-4495-A564-418FD1360340Q50442022-5C5FBAFD-6C15-4454-95C1-C56B3DFA39F4Q53263574-FA697EE9-91B6-454F-885A-B205716F627CQ53839183-5E1B79FC-23D0-47C7-9187-DC0A749173A6Q54597053-2C0B8724-83BB-4166-96E4-6001188D688AQ59138074-B2EF9677-9D2B-4248-AE03-4DEAF2C6B241
P2860
Reactive oxygen species contribute to Ca2+ signals produced by osmotic stress in mouse skeletal muscle fibres.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
Reactive oxygen species contri ...... mouse skeletal muscle fibres.
@en
Reactive oxygen species contri ...... mouse skeletal muscle fibres.
@nl
type
label
Reactive oxygen species contri ...... mouse skeletal muscle fibres.
@en
Reactive oxygen species contri ...... mouse skeletal muscle fibres.
@nl
prefLabel
Reactive oxygen species contri ...... mouse skeletal muscle fibres.
@en
Reactive oxygen species contri ...... mouse skeletal muscle fibres.
@nl
P2093
P2860
P1476
Reactive oxygen species contri ...... n mouse skeletal muscle fibres
@en
P2093
Adriano S Martins
Martha C Nowycky
Vyacheslav M Shkryl
P2860
P304
P356
10.1113/JPHYSIOL.2007.146571
P407
P577
2007-11-01T00:00:00Z